Protocol Β· Already diagnosedConfirmed Hypertension β ongoing management
Everything to be done in primary care for a patient with a confirmed diagnosis β non-drug measures, the stepwise drug ladder, monitoring, follow-up and safety-netting β up to the point of referral to secondary care. Re-enter at the ladder on any follow-up where BP is not at target. NICE NG136 Β· CKS 2026.
Diagnosis is already confirmed (ABPM/HBPM β₯135/85, or clinic β₯140/90 with end-organ damage). Before anything else, agree the number you are treating to β it drives every step-up decision below.
Patient group
Clinic target
ABPM / HBPM
Age <80 years
<140/90
<135/85
Age β₯80 years
<150/90
<145/85
Type 2 diabetes
<130/80
<125/75
CKD + ACR β₯70
<120β129 / <80
<115β124 / <75
Postural drop / frailty / multimorbidity
Check lyingβstanding BP if β₯80y, T2DM or postural symptoms β significant drop = systolic β₯20 or diastolic β₯10 mmHg; then aim for the target on the standing BP. Clinical judgement over a strict number
A target makes the plan testable: at each review you either are or are not there, which removes ambiguity about whether to step up. Tighter targets in diabetes/CKD reflect their higher absolute cardiovascular and renal risk. In the very elderly or frail, over-treatment causes falls β the standing BP and the patient's function matter more than the headline figure.
2Non-drug Β· every patient, continuously
Lifestyle prescription β a real treatment, not a delay tactic
Offer to every patient at every stage β before, alongside and after drugs. Each measure has a mechanism, a measurable target and a quantified BP effect. Agree one specific change today rather than reciting five.
π§ Salt <6 g/dayβ plasma volume β β cardiac output. Works in everyone (renin-independent). Avoid processed food; caution KβΊ salt-substitutes if on ACEi/ARB or CKD.<6 g/dayβ4β5 mmHg
π Exerciseβ peripheral resistance via endothelial NO; β sympathetic tone. Even 10-min daily walks help β start where they are.150 min/wkβ4β9 mmHg
βοΈ Weightβ insulin resistance β β RAAS. Frame: "losing 5 kg is like one BP tablet."BMI <25~1 mmHg / kg
π΄ Sleep / OSAScreen for obstructive sleep apnoea if snoring + daytime somnolence β a treatable secondary contributor.STOP-BANG
Lifestyle changes alone can drop your blood pressure by as much as a tablet would. Let's pick just one to start with β which feels most doable for you?
Lifestyle is treatment, not a holding pattern: the DASH diet alone lowers systolic BP 8β14 mmHg β equivalent to a first-line drug. One specific, achievable, measurable target massively outperforms a generic list the patient will ignore. It continues after drugs start because it reduces the number of tablets ultimately needed.
Does this patient need drug treatment now?
Stage & risk decide. Lifestyle continues either way.
NO β lifestyle trial
Stage 1, <80y, QRISK <10%, no end-organ damage
NICE permits deferring drugs. 3-month structured lifestyle trial, then repeat BP + QRISK. If still above target or new indication appears β cross into the drug ladder.
YES β start the ladder
Stage 2, OR Stage 1 with any indication
Treat now if: Stage 2 (any age), or Stage 1 plus end-organ damage, established CVD, renal disease, diabetes, or QRISK β₯10%. Begin at Step 1 below.
3Medication Β· stepwise
The A β C β D drug ladder
Start at Step 1 and step up only when BP stays above target. Before every step-up: confirm adherence, re-check HBPM, and reinforce lifestyle β do not add a drug to compensate for one that isn't being taken. Recheck U&Es 1β2 weeks after any ACEi/ARB or spironolactone change.
Step 1Monotherapy β choice depends on profiletitrate to max tolerated
<55y Β· not African / Afro-Caribbean
ACEi A
Ramipril 1.25β2.5 β 10 mg OD (or perindopril erbumine 4 β 8 mg, or lisinopril 10 β 20 mg). Switch to ARB β candesartan 8 β 32 mg (more effective & safer than losartan) β if dry cough.
Renoprotective regardless of age/ethnicity. ARB first-line in Black patients.
BP β₯ target after 4 wk β check adherence, then β
Step 2Dual therapy β A + Cadd the class not yet used
Add whichever of A or C is not already on board.
Combining two classes lowers BP more than doubling the dose of one.
ACEi + CCB has the strongest outcome evidence (ACCOMPLISH).
Consider a fixed-dose combination tablet (e.g. perindopril + amlodipine) to aid adherence.
BP β₯ target after 4 wk β check adherence, then β
Step 3Triple therapy β A + C + Dthiazide-like diuretic
Add D β indapamide 1.5 mg MR OD (preferred over bendroflumethiazide; better CV outcomes).
Check U&Es + eGFR before starting and at 4β6 weeks β can drop KβΊ/NaβΊ (stop if NaβΊ <130) and precipitate gout. Bendroflumethiazide 2.5 mg is an alternative.
If already on a thiazide for another reason, that counts as D β don't double up diuretics.
Still β₯ target on optimal A+C+D = resistant HTN β β
Step 4Resistant hypertension β add a 4th agentconfirm true resistance first
First confirm it is real: adherence (consider urine drug screen), exclude white-coat (ABPM), exclude secondary cause. Then choose by serum potassium:
KβΊ β€ 4.5 mmol/L
Spironolactone 25 mg OD
Most effective 4th agent (PATHWAY-2). Recheck U&Es/KβΊ at 1 month β esp. with ACEi/ARB.
KβΊ > 4.5 mmol/L
Ξ±-blocker (doxazosin) or Ξ²-blocker (bisoprolol)
Doxazosin IR 2β4 mg OD, or bisoprolol 2.5 mg OD (Ξ²-blocker if other indication β AF, IHD, HF). Doxazosin: ask about PDE5 inhibitors first.
β Hyperkalaemia: ACEi/ARB + spironolactone needs close KβΊ monitoring β KβΊ >5.5 = stop + recheck. Still uncontrolled on 4 drugs β refer (see step 7).
The age/ethnicity split reflects renin physiology: younger non-Black patients have high-renin HTN that responds to RAAS blockade (A); older and Black patients have low-renin, volume-dependent HTN best controlled by a CCB or diuretic. Ξ²-blockers are not first-line for uncomplicated HTN β they re-enter only at Step 4 or when separately indicated. Spironolactone is the highest-yield 4th agent because resistant HTN is frequently aldosterone-driven.
4Monitoring
Bloods after each change β the "1-2-1" rule
ACEi / ARB / spironolactone β U&Es within 1 week. Thiazide-like β within 2 weeks. Spironolactone at Step 4 β recheck at 1 month. Everyone β structured review once a year.
Most adverse events from a new antihypertensive happen in the first 1β4 weeks β a rising creatinine on an ACEi can signal undiagnosed renal artery stenosis, and hyperkalaemia is silent until dangerous. The blood test isn't a formality; it's how you catch harm before the patient feels it.
5Follow-up
The review schedule
1
2β4 weeks after any change
Response & tolerability check
BP response + side effects. U&Es per the 1-2-1 rule. Titrate dose or step up if not at target and adherence is good.
2
Every 4 weeks while titrating
Step-up reviews
Continue 4-weekly until at target. Each visit: adherence β HBPM β lifestyle β then escalate. Use HBPM diaries between visits.
3
3 months
Lifestyle-only Stage 1 patients
Repeat BP, reassess QRISK, reinforce lifestyle. Target not met β start the drug ladder.
4 Β· Lifestyle driftSalt creep, weight gain, alcohol, reduced activity. Re-set one measurable target.
5 Β· Secondary causeYoung, abrupt, or truly resistant? Screen for Conn's, renal disease, phaeo, OSA (see refer).
6 Β· Optimise dosesAre existing drugs at maximum tolerated dose before a new one is added?
All excluded and adherent β return to the ladder and step up to the next rung
6Safety-net
Safety-netting β say these out loud, document them
π΄ 999 β all patients"Sudden severe headache unlike any before, weakness/numbness on one side, sudden vision change, chest pain, or confusion β call 999 immediately. Don't drive yourself; don't wait to ring us."
π Starting any drug"You may feel dizzy when standing, especially in week one β move slowly. If you feel faint, call us β but don't stop the tablet without speaking to us first."
π On an ACEi"A dry tickly cough isn't dangerous β don't stop, ring us and we'll switch. But swelling of lips/tongue/throat is different β call 999."
βοΈ On a diuretic / spiro"If you get a vomiting-and-diarrhoea illness, pause these for 48h ('sick-day rules') and call us β they can affect your kidneys when you're dehydrated."
Naming the exact symptom and the exact action ("call 999", not "seek help") produces faster patient-activated escalation and is medico-legally protective β documented specific advice is a defence if the patient later deteriorates. Pre-warning about the ACEi cough prevents needless off-class switching; angioedema counselling is potentially life-saving.
when primary-care management is exhausted or a red flag appears
7Refer to secondary care
When to step beyond primary care
Manage everything above in primary care first. Refer when you hit a ceiling or a red flag β keep treating in the interim unless instructed otherwise, and tell the patient the reason and what to expect.
Same-day / emergencyAccelerated HTN (papilloedema / retinal haemorrhage); BP β₯180/120 with new chest pain, breathlessness, neuro signs or AKI; suspected phaeo crisis; pregnancy β₯20 wk with BP β₯140/90.
Resistant HTNAbove target on optimal A + C + D + 4th agent, once adherence and white-coat are excluded β specialist hypertension clinic.
Suspected secondary causeHypokalaemia + HTN (Conn's); abdominal bruit / rapid eGFR fall (renal artery stenosis); paroxysmal sweating/palpitations (phaeo); resistant OSA.
Young patientAge <40 with confirmed hypertension β secondary screen and specialist assessment.
Specialist input neededIntolerance to multiple classes; eGFR <30 needing RAAS drugs; pregnancy / pre-conception planning; HTN with significant end-organ disease.
I've taken your treatment as far as we safely can here, so I'm referring you to a specialist who can run some extra tests and fine-tune things. We'll keep your current tablets going while you wait.
Educational use only. Management pathway anchored to NICE NG136 and NICE CKS (2026). This is a static reference for an already-confirmed diagnosis β it does not replace clinical judgement, the patient's individual context, or your local prescribing formulary. Always verify drugs and doses against the current BNF.