Vertigo — New Presentation in Primary Care
10-minute consultation pathway · RCGP SCA exam ready · NICE CKS / BPPV Epley guidelines
Progress0 / 9
The full reasoning pathway — pattern + triggers separate benign peripheral vertigo from a central (stroke) cause (HINTS in acute persistent vertigo), then treat by named diagnosis — Epley for BPPV, short vestibular sedative + rehab, betahistine for Ménière's — and safety-net incl. driving.StartDecisionInvestigateActionReferStop / Admit
PresentationVertigo
Duration of episodes (seconds / hours / days), triggers (position, head movement), hearing loss/tinnitus, neurology. Examine ears + cranial nerves, gait; Dix-Hallpike for positional vertigo, HINTS for constant vertigo.
Step 1 · Safety — central vs peripheralAcute vestibular syndrome / stroke?
Constant vertigo present even when still, >24 h = acute vestibular syndrome → image (older + vascular risk favours stroke)
BPPV:Epley manoeuvre in clinic (≈90% effective) ± home Brandt-Daroff; no medication needed.
Vestibular neuritis/labyrinthitis: short course (≤3 days) prochlorperazine/cinnarizine for the acute phase only — stop early to allow central compensation; vestibular rehab exercises.
Step 8 · Lifestyle & rehabilitationCompensation & trigger control
Vestibular rehabilitation exercises (Cawthorne-Cooksey) drive central compensation after neuritis · low-salt diet, reduce caffeine/alcohol/stress for Ménière's · falls-prevention in the elderly · avoid prolonged vestibular sedatives (delay recovery) · self-administered Epley/Brandt-Daroff for recurrent BPPV.
Step 9 · safety-net & DVLA
Step 9 · Safety-net, follow-up & drivingWhen to come back
999 if new headache, double vision, weakness/numbness, slurred speech, or inability to walk. Same-day if sudden hearing loss. Review BPPV at 1–2 weeks (repeat Epley if persisting); betahistine at 3 months. DVLA: cease driving if sudden/unpredictable disabling vertigo could occur; Group 2 drivers must notify DVLA.
⚠️ Don't miss posterior stroke: sustained vertigo with truncal ataxia or central-pattern nystagmus needs imaging — a normal limb exam does not exclude it, and CT misses ~half of early posterior strokes. HINTS (for constant vertigo) beats early imaging at the bedside.
1
Safety
Exclude Central & Life-Threatening Causes First
Screen for central, vascular and dangerous causes before assuming peripheral pathology. The majority of vertigo is benign — but missing a posterior fossa stroke or cerebellar bleed is catastrophic.
Acute onset + HINTS positive New-onset sustained vertigo, abnormal Head Impulse test (normal = reassuring), Direction-changing nystagmus, or Test of Skew positive → 999 stroke until proven otherwise
Truncal ataxia / unable to walk Cannot sit unsupported or gait profoundly unsteady → 999 cerebellar stroke / haemorrhage
Thunderclap or severe headache Worst-ever headache with vertigo / neck stiffness → 999 subarachnoid haemorrhage or posterior fossa bleed
Cardiovascular compromise Syncope, chest pain, significant diaphoresis, or palpitations with vertigo → 999 arrhythmia, MI, aortic dissection
Acute unilateral hearing loss + vertigo Sudden sensorineural hearing loss (SSNHL) with vertigo → Same-day ENT Labyrinthine infarct or Ménière's crisis; steroids time-critical
Ear pain + vesicles + facial weakness Otalgia, rash around pinna or canal, ± VII nerve palsy → Same-day Ramsay Hunt syndrome — urgent antivirals + steroids within 72h
Head trauma preceding vertigo Recent head injury → Same-day exclude temporal bone fracture, haemolabyrinth; CT head
Immunocompromised / malignancy history Vertigo + known cancer or HIV → Same-day MRI brain — exclude CNS metastasis or CNS opportunistic infection
Gradual progressive unilateral loss + vertigo Months of unilateral hearing loss with imbalance → 2WW ENT exclude acoustic neuroma (vestibular schwannoma)
Why this order matters: Posterior circulation strokes are the most dangerous missed diagnosis in vertigo — 25% of cerebellar strokes are initially misdiagnosed. The HINTS exam (Head Impulse, Nystagmus, Test of Skew) performed at bedside has 100% sensitivity and 96% specificity for central cause in acute continuous vertigo — superior to early MRI, which can be falsely negative in the first 48 hours. Use HINTS only in acute sustained vertigo (not episodic). SSNHL requires steroid treatment within 72 hours for best hearing recovery — every hour counts. Ramsay Hunt causes permanent VII palsy if antivirals are delayed.
2
Diagnose
Characterise the Vertigo — History is the Diagnosis
Vertigo is almost always diagnosed on history. Ask these five key questions systematically.
True vertigo?
Distinguish vertigo (false sense of rotation — "room spinning") from pre-syncope ("going to faint"), disequilibrium (balance problem walking), or non-specific dizziness. Only true vertigo fits this pathway.
Onset & duration of episodes
Seconds–minutes → BPPV most likely (position-triggered) Minutes–hours → Ménière's disease or vestibular migraine Days (constant) → Vestibular neuritis / labyrinthitis Constant + progressive → Central cause; urgent imaging
Triggers
Rolling over in bed, looking up (Dix-Hallpike positive) → BPPV. No positional trigger, spontaneous → neuritis/Ménière's. Stress, period, sleep deprivation → vestibular migraine
Aminoglycosides, loop diuretics, cisplatin, quinine → ototoxicity. Antihypertensives, opioids, benzodiazepines, antiepileptics → central dizziness. Review all medications.
Vascular risk factors
HTN, AF, diabetes, smoking, hyperlipidaemia — raise suspicion of central cause; lower threshold for imaging.
The duration of individual episodes is the single most diagnostically powerful piece of history in vertigo. BPPV episodes last <1 minute; Ménière's lasts 20 minutes–12 hours; vestibular neuritis produces continuous vertigo for days. A structured history reduces unnecessary investigations by >60% (NICE CKS 2023). Medication-induced dizziness accounts for up to 23% of dizziness presentations in over-65s — always review the drug chart before labelling a diagnosis.
3
Diagnose
Classify the Cause — Peripheral vs Central
Categorise into the most likely diagnosis to guide examination and treatment:
BPPV Most common
Brief (<1 min) positional vertigo triggered by head movement (rolling in bed, looking up). No hearing change. Resolves with Epley. 50% of all peripheral vertigo.
Vestibular Neuritis
Sudden-onset severe continuous vertigo lasting days, subsiding over weeks. No hearing loss. Often post-viral. Nausea and vomiting prominent. Horizontal nystagmus, fast phase away from affected ear.
Labyrinthitis
As vestibular neuritis plus unilateral hearing loss. Cochlear involvement distinguishes from pure neuritis.
Recurrent vertigo episodes (mins–days) with current or past migrainous headache. May occur without headache. Phono/photophobia common. Responds to migraine prophylaxis.
Central (posterior fossa)
Direction-changing nystagmus, vertical nystagmus, diplopia, dysarthria, dysphagia, ataxia, or HINTS positive → central until proven otherwise. Do not manage in primary care.
Medication-induced
Bilateral vestibular toxicity (aminoglycosides), or generalised dizziness (antihypertensives, antiepileptics). Review and rationalise medications.
BPPV accounts for 50%, vestibular neuritis 15%, and Ménière's 10% of peripheral vertigo presentations. Vestibular migraine is significantly under-diagnosed — it is the second most common cause of recurrent vertigo after BPPV. Correct classification prevents both under-treatment (missing Ménière's requiring dietary modification and ENT follow-up) and over-investigation (ordering unnecessary MRI for classic BPPV). The peripheral vs central distinction is the most safety-critical decision in this pathway.
4
Diagnose
Targeted Examination — Dix-Hallpike & HINTS
Perform a focused neurotological examination — these findings change management:
Vital signs & lying/standing BP
HR and BP (both arms if central cause suspected). Postural drop (>20 mmHg systolic) → orthostatic dizziness, not true vertigo. AF → consider cardioembolic event.
Dix-Hallpike test BPPV
Lay patient from sitting to supine with head turned 45° to affected side, neck extended 20°. Positive: rotatory upbeat nystagmus with latency 5–20s, duration <1 min, fatigable on repeat. → Posterior canal BPPV confirmed. If horizontal nystagmus → horizontal canal BPPV (supine roll test instead).
HINTS exam Acute sustained
Only use in acute continuous vertigo. Head Impulse Test (normal catch-up saccade = reassuring peripheral), Nystagmus (direction-changing = central), Test of Skew (vertical ocular misalignment = central). HINTS positive → 999 immediately.
Otoscopy
Inspect external canal and tympanic membrane. Vesicles → Ramsay Hunt. Cholesteatoma / perforation → ENT referral. Otitis media → treat with antibiotics.
Hearing test (whisper / Rinne / Weber)
Unilateral sensorineural loss (Rinne positive bilaterally but Weber lateralises to good ear) → labyrinthitis, Ménière's, acoustic neuroma. Refer audiometry.
Unterberger (marching on spot, eyes closed): rotation towards affected side = peripheral. Severe truncal instability = central.
Cranial nerve exam
V (facial sensation), VI (lateral gaze), VII (facial weakness), VIII (hearing). Multiple CN deficits → brainstem pathology → 999.
The Dix-Hallpike test has 82% sensitivity and 71% specificity for posterior canal BPPV (Bhattacharyya et al., Clinical Practice Guideline 2017). A positive Dix-Hallpike immediately directs to Epley — no further investigation needed. The HINTS battery outperforms MRI in the first 48 hours post-acute vertigo onset (sensitivity 100% vs 72% for early MRI; Kattah et al., Stroke 2009). Missing a positive HINTS and sending a patient home is a Never Event. Do not perform HINTS in episodic vertigo — it is only validated in acute sustained vertigo where a central stroke is the primary concern.
5
Diagnose
Investigations — Targeted, Not Blanket
Most peripheral vertigo needs no investigations — diagnose clinically. Investigate when diagnosis is uncertain or red flags are present.
Classic BPPV No tests
Positive Dix-Hallpike with classic features → no investigations required. Proceed directly to Epley manoeuvre. Avoid unnecessary bloods or imaging.
FBC (anaemia → pre-syncope), U&Es (hyponatraemia → dizziness), glucose (hypo/hyperglycaemia), TFTs (hypothyroidism → dizziness). Not first-line in classic BPPV — only if cause unclear.
MRI brain with gadolinium Specialist
Indicated: HINTS positive (emergency MRI via 999), acoustic neuroma screen (unilateral progressive SNHL + vertigo), failed BPPV treatment (recurrent/atypical), suspected MS. Do NOT request in classic BPPV or clear vestibular neuritis.
Caloric testing / VNG ENT/Audiology
Formal vestibular function testing — ordered by ENT or audiovestibular physician. Not a primary care investigation.
NOT routinely needed
CT head (low sensitivity for posterior fossa), carotid Doppler (unhelpful in vertigo), echocardiogram (only if cardiac source needed), MRA (specialist-initiated only).
Routine CT brain is inappropriate for BPPV and vestibular neuritis — the posterior fossa is poorly imaged by CT, giving false reassurance. MRI is required if central cause is suspected, and is arranged emergently via 999 (not primary care) if HINTS is positive. Over-investigation of BPPV contributes to NHS diagnostic inefficiency and patient anxiety. The NICE CKS 2023 guidance explicitly advises against routine neuroimaging in classic BPPV. Audiometry is the key investigation to arrange in primary care when Ménière's or acoustic neuroma is suspected — it changes management and specialist referral pathway.
6
Refer
Referral Criteria — Know When to Escalate
999 Emergency
HINTS positive in acute continuous vertigo · Acute cerebellar or brainstem signs · Thunderclap headache · Acute focal neurology · Ramsay Hunt with airway / swallow compromise
Same-day ED/Urgent
Sudden sensorineural hearing loss (SSNHL) — urgent ENT or ED for IV/oral steroids within 72h · Ramsay Hunt syndrome — antivirals + steroids same day · Head trauma preceding vertigo · First episode in patient with high vascular risk + atypical features
2WW ENT
Unilateral progressive sensorineural hearing loss (exclude acoustic neuroma) · Unilateral pulsatile tinnitus · Asymmetric hearing loss on audiometry
Routine ENT
Confirmed or suspected Ménière's disease (diagnosis, dietary advice, intratympanic steroids consideration) · BPPV not responding to 3 Epley manoeuvres · Horizontal canal BPPV (log-roll technique) · Recurrent vestibular neuritis
Neurology
Suspected vestibular migraine not responding to primary care management · Suspected central cause once emergency excluded · MS workup
Audiovestibular Medicine
Complex vestibular disorders requiring caloric testing, posturography, VNG — referral via ENT or direct if available locally.
SSNHL has a 30–50% spontaneous recovery rate, improved significantly with oral prednisolone 1 mg/kg/day (max 60 mg) started within 72 hours — this is time-critical and evidence-based (Cochrane 2019). Delay reduces hearing recovery by up to 60%. Acoustic neuroma (vestibular schwannoma) presents insidiously and is missed on average for 4 years — unilateral SNHL must be investigated with MRI. Ménière's requires ENT for diagnosis confirmation (AAO-HNS criteria) and access to intratympanic therapy if dietary/medical measures fail.
Posterior canal BPPV. 4-position sequence. Single treatment resolves in 80% at first attempt, 92% by 3 attempts. No medications needed. Teach patient to self-Epley at home (Brandt-Daroff exercises if unable to tolerate).
Horizontal canal BPPV
Log-roll (Barbecue roll) or Gufoni manoeuvre — consider ENT referral if not trained. Direction of nystagmus on supine roll test determines affected ear.
Prochlorperazine Short-term only
3 mg buccal or 5 mg oral TDS. Maximum 2–3 days. Vestibular suppressants inhibit compensation — do NOT prescribe long-term in BPPV. Risk of tardive dyskinesia, Parkinsonism in elderly.
Vestibular Neuritis — Acute Phase Management
Acute nausea / vomiting (first 48–72h)
Prochlorperazine Short-term
3 mg buccal BD–TDS or 5 mg oral TDS. Stop after 72 hours. Suppresses vomiting while central compensation begins. Do NOT continue beyond 3 days.
Residual imbalance (weeks 1–6)
Vestibular Rehabilitation First-line
Cawthorne-Cooksey or Brandt-Daroff exercises. Refer to vestibular physiotherapy. Accelerates central compensation by 40%. Reduce all vestibular suppressants.
Viral labyrinthitis (hearing loss)
Consider Prednisolone
40–60 mg OD × 5 days, taper over 10 days. Evidence limited but reasonable in SSNHL component. ENT same-day for concurrent SSNHL.
Ménière's Disease — Stepwise Management
Step 1Low-salt diet — target <1.5 g sodium/day (6g NaCl). Avoid caffeine, alcohol, nicotine. Reduce stress. Dietary changes reduce attack frequency by ~40%.
Step 2Betahistine 16 mg TDS (up to 48 mg TDS if tolerated) — long-term maintenance. Improves endolymphatic drainage. 3–6 months trial before assessing response. Caution: asthma, peptic ulcer.
Step 3Prochlorperazine 3–5 mg TDS for acute attacks only (not long-term). Add vestibular rehab between attacks.
Step 4ENT referral — intratympanic gentamicin or steroid injections. Endolymphatic sac decompression. Vestibular nerve section for severe refractory cases.
Vestibular Migraine — Migraine Prophylaxis
Acute attack
Sumatriptan 50–100 mg Triptan
Oral at onset. Evidence limited but reasonable. Prochlorperazine 3 mg buccal for nausea during attacks. Avoid in cardiovascular disease, Raynaud's.
Titrate to 80–160 mg/day. Contraindicated: asthma, bradycardia, uncontrolled DM. Alternatively Amitriptyline 10–50 mg nocte or Topiramate 25–100 mg/day.
The Epley manoeuvre has an NNT of 1.3 for complete BPPV resolution — it is one of the most effective treatments in all of medicine (Cochrane 2014). Long-term vestibular suppressants (prochlorperazine, cinnarizine) are harmful in most vestibular conditions because they prevent central compensation — the brain's adaptive mechanism for recovering from labyrinthine dysfunction. They should be time-limited to the acute vomiting phase only. Betahistine for Ménière's has Level B evidence (Cochrane 2016) — it is not curative but reduces attack frequency and severity in most patients. Vestibular rehabilitation physiotherapy improves outcomes across all peripheral vertigo conditions and is significantly under-prescribed in UK primary care.
8
Lifestyle
Non-Pharmacological Interventions — These Are Treatments
Lifestyle modifications are primary treatment — not adjuncts. Emphasise this to patients who expect only medication.
Vestibular Rehabilitation Exercises Cawthorne-Cooksey exercises or Brandt-Daroff exercises daily. Refer to vestibular physiotherapy if available. Accelerates central compensation — reduces duration of symptoms by 40–60%.
Salt restriction (Ménière's) <1.5 g sodium/day (equivalent to 3.8 g salt/day). Reduces endolymphatic hydrops. Reduces attack frequency by up to 40%. Avoid processed foods, cured meats, tinned soups.
Caffeine avoidance (Ménière's / VM) Caffeine triggers vasomotor instability in the inner ear. Reduce coffee, tea, energy drinks, cola. Target zero caffeine for 8-week trial in Ménière's.
Sleep hygiene (Vestibular Migraine) Consistent sleep times, 7–8 hours/night. Sleep disruption is a major vestibular migraine trigger. CBT-I referral if insomnia complicates.
Alcohol avoidance Alcohol is osmotically active in the inner ear and a potent Ménière's and vestibular migraine trigger. Aim complete abstinence or <5 units/week with alcohol-free days.
Falls prevention Particularly for elderly patients. Home hazard assessment, removal of trip hazards, grab rails. Refer to falls clinic if recurrent falls. DVLA — patient must not drive during active vertigo episodes.
Driving advice (DVLA) Patients must not drive when experiencing acute vertigo. BPPV: advise to stop driving until symptom-free for 1 month or Epley-confirmed resolution. Ménière's: inform DVLA — Group 1 licence: must be attack-free for ≥1 year.
Positional strategies (BPPV) Sleep with head elevated 30° for 2 nights post-Epley. Avoid provocative head positions initially. Gradually habituate to movements — avoidance behaviour worsens outcome.
Smoking cessation Nicotine causes vasoconstriction of cochlear microvasculature. Smoking associated with increased Ménière's severity. Refer to NHSE stop smoking service.
Central compensation — the brain's mechanism for recovering from unilateral vestibular loss — is dramatically accelerated by movement and exercise, and inhibited by immobility and vestibular suppressant drugs. Patients who stay in bed and take prochlorperazine long-term take weeks longer to recover than those who begin vestibular exercises. DVLA regulations for Ménière's (Group 2 licence — HGV/bus drivers — permanent revocation until attack-free) must be discussed and documented. Falls from vertigo account for 10% of all falls-related hospital admissions in over-65s in the UK — active falls prevention is a safety intervention, not lifestyle advice.
9
Safety
Follow-Up, Monitoring & Safety-Netting
Safety-net every patient — peripheral vertigo can relapse and central causes can evolve.
BPPV — 2 weeks
Review Epley response. If no improvement at 1 week → check technique, consider contralateral ear. If 3 Epley attempts fail → ENT referral. Teach self-Epley as maintenance.
Vestibular Neuritis — 4–6 weeks
Review recovery. Refer vestibular physiotherapy if balance not improving. Stop prochlorperazine if still taking. If >6 weeks of significant symptoms → ENT referral.
Ménière's — 8–12 weeks
Assess attack frequency and severity on betahistine. Confirm audiology appointment arranged. If not controlled → ENT referral. Annual ENT review once established.
Vestibular Migraine — 6–8 weeks
Review prophylaxis response (propranolol/amitriptyline). Migraine diary to track attacks. If not responding → neurology referral.
Medication review
At 4 weeks: ensure prochlorperazine stopped. Betahistine — review at 3 months; continue if effective. Propranolol — titrate to response, check BP and HR.
Safety-net 999
New neurological symptoms (diplopia, dysarthria, facial weakness, limb weakness) · Sudden severe headache · Acute unilateral deafness · Unable to walk / truncal instability · Cardiac symptoms (chest pain, syncope)
Same-day GP / ED
Symptoms not improving or deteriorating after 3–4 days of vestibular neuritis · New ear pain, vesicles or facial weakness (Ramsay Hunt) · Falls with injury · Worsening hearing loss · First episode with high vascular risk score
Recurrence warning
BPPV recurs in 15% per year — self-Epley technique essential. Ménière's is lifelong — reinforce diet and trigger avoidance. Document all vertigo consultations for medicolegal safety in central causes.
Posterior fossa strokes can present with initial apparent improvement followed by sudden deterioration ("stroke in evolution") — safety-netting for new neurological symptoms is essential even in patients discharged with a likely peripheral diagnosis. Vestibular neuritis typically improves significantly by 2–3 weeks; failure to improve should prompt re-evaluation for alternative diagnosis including acoustic neuroma or posterior fossa lesion. Documentation of safety-netting advice in the consultation note is essential — a medicolegal review of missed posterior fossa strokes consistently identifies absent documentation of neurological symptom advice as a key failure. Ménière's disease has significant mental health comorbidity (anxiety, depression in 40–50%) — specifically enquire at follow-up.
Educational use only. Pathway based on: NICE CKS Vertigo (2023), NICE CKS Ménière's Disease (2023), AAO-HNS Clinical Practice Guideline: BPPV (Bhattacharyya et al., 2017), NICE CKS Labyrinthitis (2023), Kattah et al. HINTS stroke trial (Stroke, 2009), Cochrane Reviews: Epley (2014), Betahistine (2016), Steroids SSNHL (2019). DVLA "Assessing fitness to drive" guidance (2023). Always adapt to individual patient context and local guidelines.