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Subconjunctival Haemorrhage — Assessment & ManagementRuptured globe exclusion · VA mandatory · RAPD swinging torch · BP check · anticoagulation management · rhinorrhoea CSF · NAI safeguarding · lubricant drops
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The full reasoning pathway โ€” a subconjunctival haemorrhage is benign and self-limiting; the job is to confirm normal vision and check for an underlying bleeding tendency or trauma. Reassure and safety-net.StartDecisionInvestigateActionReferStop / Admit
PresentationSubconjunctival haemorrhage
Painless, well-demarcated red patch, normal vision. Ask about trauma, anticoagulation, Valsalva, recurrence. Check BP.
Step 1 ยท Safety โ€” trauma / red flagsTrauma / red flags?
Significant trauma (?globe injury), no visible posterior border (orbital/head injury), reduced vision/pain, or recurrent bleeds suggesting bleeding disorder.
YES
Stop ยท EscalateUrgent / investigate
Trauma with possible globe injury โ†’ ophthalmology. Recurrent โ†’ clotting screen / review anticoagulation.
NO
AssessBy pattern
History + examination localise the cause.
Step 3 ยท common causes
Spontaneous
Commonest
Valsalva (cough/sneeze/straining), idiopathic; reassure โ€” resolves in 1โ€“2 weeks.
Anticoagulation / bleeding
Check
Warfarin/DOAC, hypertension, bleeding disorder; check INR/BP if recurrent.
Traumatic
Assess
Exclude globe injury if trauma; otherwise reassure.
Step 6 ยท ReferEscalation
Ophthalmology traumatic with possible globe injury or abnormal vision; investigate recurrent bleeds (clotting, BP, anticoagulation review).
Step 8 ยท reassurance & modifiable factors
Step 8 ยท Reassurance & modifiable factorsBenign โ€” treat the underlying contributors
Reassure โ€” resolves over 1โ€“2 weeks (may change colour like a bruise); lubricants if mild irritation, no specific treatment needed. Check and manage blood pressure; review anticoagulation/antiplatelet control (INR if on warfarin). Avoid eye rubbing and address recurrent Valsalva triggers (cough, constipation/straining). Contact-lens hygiene if relevant.
Step 9 ยท review & safety-net
Step 9 ยท Review & safety-netReassure, but check the basics
Should clear within ~2 weeks โ€” review if it doesn't, spreads, or vision becomes affected. Ophthalmology for any trauma with possible globe injury (no visible posterior border, pain, reduced vision). Investigate recurrent bleeds โ€” check BP, clotting and anticoagulation; consider a bleeding tendency. Otherwise simple reassurance.
โš ๏ธ Reassure but check the basics: an isolated subconjunctival haemorrhage with normal vision is benign โ€” but recurrent bleeds warrant a look at blood pressure, anticoagulation and clotting.
1
Safety

Red Flags โ€” Traumatic Rupture, Bleeding Diathesis & Cavernous Sinus

Subconjunctival haemorrhage + significant blunt eye trauma + decreased visual acuity + afferent pupillary defect Ruptured globe or posterior segment injury. Do NOT apply pressure. Eye shield (not pad). โ†’ 999 ophthalmology emergency. CT orbits if ruptured globe suspected. Nil by mouth (surgical repair).
Bilateral subconjunctival haemorrhage post-trauma (no direct eye injury) or after head/neck compression Traumatic asphyxia (Perthes sign) or base of skull fracture. โ†’ 999. CT head urgently. Bilateral haemorrhage without direct ocular trauma = intracranial pressure transmission.
Subconjunctival haemorrhage + fever + photophobia + neck stiffness + non-blanching petechiae Meningococcal septicaemia โ€” petechial haemorrhages including conjunctiva. โ†’ 999. IV benzylpenicillin 1.2g IM/IV immediately. Time-critical.
Recurrent bilateral or very large subconjunctival haemorrhage + easy bruising + gum bleeding Bleeding diathesis โ€” platelets (ITP, anticoagulant toxicity), coagulation defect (haemophilia, liver failure), or leukaemia. โ†’ FBC + coagulation screen + LFTs urgently. Haematology.
Subconjunctival haemorrhage extending posterior to the equator (360 degrees, very posterior, unable to see the border) Possible occult ruptured globe. โ†’ 999 ophthalmology. Do not attempt examination of the eye.
Proptosis + periorbital oedema + pulsatile tinnitus + chemosis (conjunctival oedema) + arterialized veins Carotid-cavernous sinus fistula (post-traumatic or spontaneous). โ†’ 999 neurosurgery. MRA/CTA head urgently.
A subconjunctival haemorrhage (SCH) is almost always a benign, self-limiting condition โ€” the haemorrhage represents bleeding into the potential space between the conjunctiva and the underlying sclera, caused by rupture of a small conjunctival blood vessel. The blood is bright red initially, may spread and darken over days, and resolves completely within 2-4 weeks without treatment. The critical clinical task for GPs is not to manage the SCH itself, but to identify the rare cases where it is a sign of serious underlying pathology. The most important distinguishing feature: in simple SCH, visual acuity is completely normal. Any reduction in visual acuity in the presence of a red eye is not a simple SCH โ€” it requires urgent ophthalmology assessment. The posterior limit of a benign SCH should be visible (the blood does not extend beyond the equator of the globe) โ€” if you cannot see the posterior border, a ruptured globe or posterior injury must be excluded.
2
Diagnose

Causes of Subconjunctival Haemorrhage

Spontaneous (most common)
Usually no identifiable cause. Risk factors: hypertension (check BP at every SCH presentation), anticoagulants (warfarin, DOACs โ€” check INR/dose), antiplatelet agents (aspirin, clopidogrel โ€” reduce platelet function), aspirin + NSAID combination, blood thinning supplements (fish oil, ginkgo, garlic). Age: elderly (fragile conjunctival vessels). Rarely: vigorous Valsalva (coughing, straining at stool, heavy lifting, sneezing, vomiting, childbirth).
Traumatic
Direct ocular trauma: assess carefully for ruptured globe (see Step 1). Contact lens trauma (microtrauma from lens edge โ€” check fitting). Foreign body (retained FB under upper eyelid). Iatrogenic: post-cataract surgery, post-eye injection (intravitreal), post-eyelid surgery.
Valsalva manoeuvre
Forceful coughing (pertussis, asthma exacerbation), vomiting (hyperemesis, bulimia), straining (constipation, heavy lifting, labour), vigorous exercise. The manoeuvre transiently increases intrathoracic pressure โ†’ increased venous pressure โ†’ rupture of conjunctival capillaries. Bilateral SCH from Valsalva: possible but requires exclusion of intracranial pathology.
Secondary to systemic disease
Anticoagulation (warfarin โ€” supratherapeutic INR; DOAC at standard dose can also cause). Bleeding diathesis (ITP, haemophilia, liver disease). Hypertension (well-established association โ€” check BP). Diabetes (fragile microvasculature). Leptospirosis, viral haemorrhagic fevers (travel history). Allergic conjunctivitis (associated conjunctival hyperaemia).
The hypertension-subconjunctival haemorrhage association deserves special attention โ€” hypertension is a well-established risk factor for SCH, and a patient who presents with a first SCH without clear precipitating cause should have their blood pressure checked at the same consultation. The mechanism: chronic hypertension causes hyaline degeneration of small vessel walls (arteriosclerosis), making the conjunctival capillaries more fragile and prone to spontaneous rupture even with minor physiological triggers (rising from sleep, morning coughing). The practical approach: any patient with a first SCH and no clear cause โ†’ check BP. If BP is elevated: this is an opportunity to diagnose and treat hypertension. If hypertension is already known and treated: check medication adherence and BP control. This does not mean that SCH itself requires hypertension treatment urgently โ€” the SCH will resolve regardless โ€” but it is a diagnostic opportunity not to be wasted.
3
Diagnose

Assessment โ€” Examination & Key Differentials

Clinical assessment
Visual acuity (mandatory โ€” Snellen chart or asking patient to read text): any reduction = not a simple SCH. Pupil reactions: afferent pupillary defect (APD/RAPD = relative afferent pupillary defect) = posterior segment/optic nerve injury. Anterior chamber: assess depth and clarity (flat chamber = ruptured globe; hyphaema = layered blood in AC = blunt trauma). Intraocular pressure: not needed in primary care. Extent of haemorrhage: can the posterior border be seen? If not visible = 999.
Differentiating from other causes of red eye
Anterior uveitis โ€” ciliary injection (ring around cornea, not diffuse), photophobia, small irregular pupil. Acute angle-closure glaucoma โ€” rock-hard globe, mid-dilated fixed pupil, halos around lights, severe pain, nausea. Bacterial conjunctivitis โ€” diffuse conjunctival injection, mucopurulent discharge, no haemorrhage. Episcleritis โ€” sectoral redness, movable redness (moves with conjunctiva on pressing eyelid), mild ache. SCH โ€” bright red flat haemorrhage, clearly demarcated, white sclera visible at periphery, no pain, normal vision.
History
Onset (sudden = typical SCH). Pain (SCH = painless; painful red eye = uveitis, glaucoma, keratitis). Vision change (none in SCH). Trauma. Contact lens use. Anticoagulant medications. Previous SCH episodes. Systemic conditions (hypertension, diabetes, liver disease, haematological).
The afferent pupillary defect (APD) is the single most important examination finding that distinguishes serious posterior segment pathology from a benign superficial haemorrhage. The swinging flashlight test: in a dim room, swing a bright pen torch from one eye to the other. Normal: both pupils constrict when light is directed at them. RAPD (relative afferent pupillary defect): when the torch swings from the normal eye to the affected eye, the pupils paradoxically dilate rather than constrict โ€” this indicates that the optic nerve input from the affected eye is weaker than from the normal eye (the brain compares the two signals). An RAPD in the context of a red eye or SCH after trauma indicates: retinal detachment, vitreous haemorrhage, traumatic optic neuropathy, or posterior rupture of the globe โ€” all requiring emergency ophthalmology assessment. The swinging flashlight test takes approximately 30 seconds and should be performed for every patient presenting with a red eye โ€” it is a key component of the red eye assessment that GPs should be confident performing.
4
Diagnose

Investigations โ€” When to Go Beyond Clinical Examination

When clinical examination is sufficient
First simple SCH in a young-to-middle-aged adult with identifiable trigger (coughing, sneezing, straining) or no trigger + normal BP + no anticoagulants + normal vision + full anterior border visible: reassurance + self-limiting. No investigations required. Check BP if not recorded recently.
When to investigate
First SCH without identifiable cause in a patient over 60: BP + FBC (platelet count) + coagulation screen (INR if on warfarin โ€” check level). Recurrent SCH (>2 episodes in 6 months without explanation): FBC + clotting + LFTs + HbA1c. Patient on anticoagulant: check INR (warfarin) or confirm DOAC adherence and renal function. Travel history + fever + SCH: blood film (malaria) + leptospirosis serology + viral haemorrhagic fever screen (if endemic area โ€” Public Health England).
Anticoagulation-related SCH
Check INR if on warfarin. If INR supratherapeutic: adjust warfarin dose. If INR therapeutic: anticoagulation-related SCH is a known minor bleeding event โ€” continue anticoagulation (the clinical benefit outweighs the cosmetic nuisance of SCH). Document as a minor bleeding event. Do NOT stop anticoagulation for SCH.
The management of anticoagulation in patients with SCH is an important prescribing decision โ€” stopping anticoagulation for an SCH exposes the patient to the very thromboembolic risks that the anticoagulation was prescribed to prevent (stroke, DVT/PE, valve thrombosis). An SCH is a minor, self-limiting, cosmetically unpleasant but clinically inconsequential event that does not warrant suspension of anticoagulation. The correct approach: check the INR (if on warfarin) or assess DOAC adherence; if INR is supratherapeutic, adjust the warfarin dose; document the SCH as a minor bleeding event in the anticoagulation record; continue anticoagulation. The only bleeding events that should prompt consideration of anticoagulation interruption are: intracranial haemorrhage, massive GI bleeding, life-threatening haemorrhage from any site. An SCH does not meet these criteria.
5
Refer

Referral Pathways

999
Reduced visual acuity + SCH after trauma ยท RAPD present ยท Posterior border of SCH not visible ยท Bilateral SCH without direct ocular trauma ยท Ruptured globe features (flat AC, abnormal pupil)
Ophthalmology (urgent within 24-48h)
Traumatic SCH + normal vision but contact lens wearer (FB exclusion) ยท SCH + chemosis (conjunctival oedema) + proptosis (orbital process)
Ophthalmology (routine)
Recurrent SCH (>2 episodes without identified cause) after primary care workup normal ยท Anticoagulated patient with multiple SCH episodes โ€” confirm no conjunctival pathology
Haematology
Recurrent SCH + low platelet count or coagulation abnormality ยท Suspected bleeding diathesis
GP management
Single uncomplicated SCH: reassurance + lubricant drops for comfort + self-limiting (2-3 weeks) + BP check. Second episode: investigate and treat underlying cause (hypertension, anticoagulation review, constipation causing straining).
The GP has a valuable reassurance opportunity at every SCH consultation โ€” the haemorrhage looks alarming to patients (bright red, spreading, covering the white of the eye) and causes significant anxiety. Effective reassurance: 'A subconjunctival haemorrhage is like a bruise on the skin โ€” it looks worse than it is. It is caused by a tiny blood vessel on the surface of the eye breaking. It will gradually change colour from red to orange to yellow, and completely disappear over the next 2-3 weeks without any treatment. It does not affect your vision and will not damage your eye.' Managing expectations: the colour change and spreading over the first few days can be alarming โ€” tell the patient in advance that the haemorrhage may look larger or darker over days 2-5 before it begins to resolve. Cold compresses in the first 24 hours reduce spreading; artificial tear lubricant drops (if the eye is dry and uncomfortable) can provide symptomatic relief. No eye drops, tablets, or other specific treatment is needed.
6
Treat

Management of Simple Subconjunctival Haemorrhage

Self-limiting management
No specific treatment required. Cold compress in first 24 hours may reduce spreading. Artificial tear lubricant eye drops (hypromellose 0.3% or carbomer gel) if eye feels gritty or uncomfortable โ€” 2-4 times daily. Avoid rubbing the eye (worsens spreading). Avoid aspirin and NSAIDs for the first 48 hours if not essential (mild platelet inhibition). Continue regular anticoagulation. No restrictions on activities (including driving) โ€” vision is unaffected.
Treat the precipitating cause
Hypertension: treat per NICE NG136 guidelines (amlodipine first-line in non-Black patients over 55). Constipation (straining): lactulose 15 ml BD or macrogol + dietary fibre + fluid intake. Chronic cough (causing recurrent SCH from Valsalva): investigate + treat cause (see chronic cough algorithm). Anticoagulation supratherapeutic: dose adjustment. Contact lens fit: optician review. Dry eyes (fragile surface vessels): lubricant drops long-term.
Recurrent SCH management
Investigate: FBC + clotting + LFTs + HbA1c + BP. If all normal: review medications (anticoagulants, antiplatelets, fish oil โ€” combined), treat any Valsalva cause. If platelet abnormality: haematology. If liver disease: hepatology. Address cardiovascular risk factors comprehensively.
Post-intravitreal injection SCH
Iatrogenic SCH after intravitreal injection (anti-VEGF agents โ€” ranibizumab, bevacizumab, aflibercept): extremely common (up to 40% of injections), always at the injection site, resolves within 2 weeks, requires no treatment. Reassure the patient this is normal and expected. Does not affect efficacy of treatment.
The post-intravitreal injection SCH management is worth specific inclusion because anti-VEGF therapy for wet AMD, diabetic macular oedema, and retinal vein occlusion is now extremely common โ€” the UK administers approximately 1 million intravitreal injections per year. After each injection, a small SCH at the injection site is expected and normal (the needle punctures the conjunctiva). Patients who are treated monthly or bimonthly may contact their GP about the red eye post-injection, and GPs must know to reassure rather than alarm. However: any SCH that develops in a different location from the injection site, or is associated with reduced vision or pain, should be referred back to the ophthalmology team. The key information GPs need to provide: 'This red patch near where you had your injection is completely normal โ€” it is just a tiny bruise on the surface of the eye from the needle. It will disappear in 1-2 weeks and does not affect how well your eye injection is working.'
7
Treat

When to Escalate โ€” Medicolegal Red Flags

Always measure visual acuity
Missing a reduction in visual acuity in a "simple SCH" is the most common medicolegal error in GP ophthalmic assessment. A patient with traumatic SCH + ruptured globe or vitreous haemorrhage who has their vision incorrectly assessed as normal (due to absent visual acuity testing) will have a delayed diagnosis of a sight-threatening injury. VA measurement takes 30 seconds with a near-vision card or Snellen chart. Document the VA result in every red eye consultation.
Document the posterior border
A simple SCH has a clearly visible posterior border โ€” the blood does not extend behind the equator of the globe. If the entire conjunctiva is haemorrhagic with no visible posterior limit: treat as potential ruptured globe. Do not apply eye pad (pressure). Apply rigid eye shield. 999.
Contact lens wearers + SCH
An SCH in a contact lens wearer (particularly daily disposable or extended wear) can be caused by a retained lens fragment under the upper eyelid. The upper eyelid must be everted to exclude a retained foreign body. Failure to evert the upper eyelid and finding a retained lens fragment that then scratches the cornea repeatedly causes a persistent corneal abrasion โ€” preventable with a 30-second examination.
Post-traumatic SCH without clear mechanism
An SCH in a child or elderly person following unexplained trauma must prompt consideration of non-accidental injury (NAI โ€” child) or elder abuse. Document the mechanism carefully. Is the mechanism consistent with the injury? Safeguarding assessment if inconsistency. NICE safeguarding guidance applies.
Non-accidental injury should be considered in the differential when a child presents with a subconjunctival haemorrhage without a clearly consistent mechanism โ€” retinal haemorrhages (not visible to the GP without ophthalmoscopy) are a classical feature of abusive head trauma ('shaken baby syndrome'), and conjunctival haemorrhages can co-exist. Any child presenting with an SCH where: the mechanism is unclear or inconsistent with the injury, there are other unexplained bruises or injuries, the parent's account is inconsistent, or the child's behaviour is abnormal โ€” should trigger a safeguarding assessment. Document the exact mechanism as described by the parent/guardian. Refer to paediatrics for full assessment if non-accidental injury is suspected. The medicolegal documentation standard: 'Mechanism of injury as described by parent: [exact statement]. Injury consistent/inconsistent with mechanism. Safeguarding concern: [yes/no and reasoning]. Action taken: [specify].'
8
Lifestyle

Preventing Recurrence & Eye Health

Blood pressure control and eye health Hypertension damages conjunctival vessels through the same hyaline arteriosclerosis mechanism that causes retinal haemorrhages and stroke. SCH is an opportunity to motivate BP treatment: "This red eye is a reminder that your blood pressure needs to be controlled โ€” high blood pressure makes your small blood vessels more fragile." Lifestyle: reduce salt to <6g/day, increase aerobic exercise (150 min/week), DASH diet (fruit, vegetables, low dairy, reduced red meat), reduce alcohol (<14 units/week), weight loss (each 1kg lost reduces SBP approximately 1 mmHg).
Anticoagulation awareness Patients on anticoagulants should know that minor bleeds (SCH, easy bruising, minor nosebleeds) are expected side effects and do not require emergency action. They should contact GP/pharmacy/anticoagulant clinic for: black/tarry stools, vomiting blood, severe headache, weakness/slurred speech (stroke), heavy menstrual bleeding, prolonged nosebleed (>20 minutes). SCH is minor โ€” reassurance is appropriate.
Avoiding Valsalva triggers Constipation: fibre (25-35g/day), 8 glasses water daily, macrogol if needed. Chronic cough: investigate and treat cause. Heavy weightlifting: breath out during exertion (exhale on the exertion phase โ€” reduces Valsalva-related venous pressure). Singers and wind instrument musicians: awareness of repeated Valsalva stress on conjunctival vessels.
Contact lens hygiene Replace lenses as prescribed (do not extend daily disposable to 2+ days). Remove lenses before sleep (even extended-wear lenses should be removed weekly for corneal oxygenation). Clean lens case daily with fresh solution (not tap water). Discard solution after each use. Annual optician contact lens check โ€” corneal health assessment. Any persistent eye redness with contact lens wear: remove lenses and see GP.
Dry eye and conjunctival fragility Chronic dry eye causes conjunctival surface fragility โ€” desiccated conjunctival epithelium tears more easily, predisposing to SCH. Symptoms of dry eye: grittiness, burning, foreign body sensation, paradoxical excessive tearing, worse in air-conditioned environments. Treatment: preservative-free lubricant drops (hypromellose PF, sodium hyaluronate) 4-6x daily + omega-3 supplements (reduces eyelid margin inflammation) + humidifier in dry environments. Referral to optician or ophthalmology for severe dry eye (punctal plugs, topical ciclosporin eye drops).
Screen time and conjunctival health Prolonged screen use reduces blink rate by approximately 60% (from 15-20 blinks/min to 5-7 blinks/min), causing surface dryness and conjunctival stress. The 20-20-20 rule: every 20 minutes, look at something 20 feet away for 20 seconds. Ensure screen is at or below eye level (reduces ocular surface exposure). Regular breaks. Lubricant drops if symptomatic.
Smoking and eye health Smoking is the strongest modifiable risk factor for age-related macular degeneration (AMD) โ€” 4x increased risk in current smokers. Smoking also increases conjunctival vascular fragility, dry eye, cataract risk, and uveitis risk. Every eye consultation is an opportunity to ask about smoking and offer NHS Stop Smoking referral.
Post-delivery SCH education (obstetric) SCH during childbirth from pushing (Valsalva) is extremely common โ€” occurs in approximately 5% of deliveries. Mothers should be reassured proactively: "You may notice a red area in the whites of your eyes after delivery โ€” this is very common after the pushing during labour and will disappear completely in 2-3 weeks." This prevents unnecessary post-natal GP attendances for reassurance about a normal finding.
The obstetric Valsalva SCH is worth specific mention because of its high frequency and because it causes significant maternal anxiety if not proactively addressed โ€” studies report SCH in approximately 4-8% of vaginal deliveries, and the rate is higher after prolonged second-stage labour. Midwives and obstetricians should ideally mention the possibility of SCH in antenatal education so that mothers are not alarmed when they look in the mirror post-delivery. GPs who see post-natal women for the 6-week check should ask about any eye symptoms and reassure about SCH if present. The condition is benign and self-limiting, but a new mother who notices 'a lot of blood in her eye' without prior warning may experience significant anxiety and seek emergency care unnecessarily.
9
Safety

Follow-Up & Key Safety Points

First uncomplicated SCH
No specific follow-up required. Review in 3-4 weeks if not resolved (confirm normal resolution). Safety-net: return immediately if vision changes, pain develops, or haemorrhage recurs.
Recurrent SCH (2+ episodes)
Review at 4-6 weeks after investigation results. Treat identified cause. If no cause found: annual eye health review (BP, medications, FBC). Ophthalmology referral if concerned about structural conjunctival disease.
Anticoagulated patient with SCH
Document in anticoagulation record as minor bleeding event. Continue anticoagulation. If warfarin: check INR. Review anticoagulation risk:benefit if multiple minor bleeds. Haematology input if complex anticoagulation decision.
Child with SCH
Document mechanism and consistency. Safeguarding assessment if any doubt. Paediatric referral if NAI concern or if child has multiple haemorrhages.
Return / refer same day
Any vision change with SCH โ†’ ophthalmology emergency ยท Posterior border of SCH not visible โ†’ 999 ยท RAPD on swinging torch test โ†’ 999 ยท Bilateral SCH post-trauma โ†’ 999
Within 48h
SCH in contact lens wearer โ†’ confirm FB excluded by lid eversion ยท SCH in anticoagulated patient โ†’ check INR (warfarin) + review DOAC dose
The documented visual acuity measurement is medicolegally essential for every red eye consultation in primary care โ€” the GMC Good Medical Practice standards require that clinical assessments are thorough and appropriate to the presenting condition. In a patient with a red eye, VA measurement is a basic and mandatory component of the assessment. A GP who documents 'vision normal' without measuring VA, and whose patient subsequently has a missed retinal detachment or posterior segment injury, faces a defensible medico-legal challenge only if they can demonstrate that VA was formally measured and recorded. The documentation standard: 'VA: Right 6/6 (or 'reads news print at 30 cm'). Left 6/6. Posterior border of haemorrhage visible. Pupils equal and reactive. No RAPD. Diagnosis: simple SCH. Management and safety-net provided.'
Educational use only. Based on RCOphth Red Eye Guidelines 2019, NICE NG136 Hypertension, NICE Safeguarding Children, GMC Good Medical Practice documentation standards, BNF lubricant eye drop prescribing.