ENT · Full case

Vertigo

NICE NG128 Bárány 2022 (international)
VT
Vertigo · Clinical Reasoning Framework v2
GP & SCA · NICE NG127 / BPPV Clinical Guidelines (international)
82%Reported Dix-Hallpike sensitivity for posterior canal BPPV (AAO-HNS, international)
≥30 secHold each Epley position until vertigo and nystagmus settle
1+Most people improve after one or more Epley manoeuvres
1 weekVestibular neuritis acute phase duration
20 min–12 hrsMenière's attack duration (episodic)
HINTSHead Impulse·Nystagmus·Test of Skew
DVLANo driving while liable to sudden disabling vertigo; notify DVLA
3 canalsPosterior canal BPPV = most cases
📋 Clinical Stem — Acute Vertigo in a Middle-Aged Adult
A 57-year-old woman presents with sudden-onset vertigo and vomiting — differentiate BPPV, vestibular neuritis, and posterior circulation stroke
"Mrs Helen Okafor, 57, a secondary school teacher. She presents with sudden-onset severe rotational vertigo that started this morning when she rolled over in bed. The room is spinning. She has vomited twice. She finds it very difficult to stand or walk. No headache. No hearing loss. No diplopia or dysphagia. No weakness or numbness. Her vascular risk factors: hypertension (amlodipine 10 mg OD, ramipril 5 mg OD), BMI 32, non-smoker, drinks 12 units alcohol per week. The vertigo is there at rest but dramatically worse when she moves her head."
The sudden onset on head movement (rolling over in bed) with positional worsening strongly suggests BPPV. However, the severity of her presentation and her vascular risk factors (hypertension, obesity) mean a posterior circulation stroke must be actively excluded — not assumed absent. The HINTS exam (Head Impulse test, Nystagmus type, Test of Skew) is the primary clinical tool for distinguishing peripheral from central vertigo at the bedside, and is more sensitive than MRI in the first 48 hours of posterior fossa stroke.
Scenario A — BPPV (Classic) Vertigo with position change (rolling over, looking up), brief (<1 min), Dix-Hallpike positive (torsional upbeat nystagmus, fatigues, latency of a few seconds). Normal HINTS. Epley manoeuvre in clinic.
Scenario B — Vestibular Neuritis Sustained vertigo for days following a viral illness, worse with movement, no hearing loss, HINTS: abnormal head impulse test (catch-up saccade present = peripheral), direction-fixed nystagmus, no skew. Short-course prochlorperazine (2–3 days) + vestibular rehabilitation; steroids not started routinely.
Scenario C — Posterior Circulation Stroke (AICA/PICA) Acute vertigo + new headache, diplopia, dysarthria, facial numbness, ipsilateral limb ataxia, or contralateral limb weakness. HINTS: normal head impulse (no catch-up saccade), direction-changing nystagmus, skew deviation present. 999 immediately — stroke pathway.
Scenario D — Menière's Disease Episodic vertigo lasting 20 minutes to 12 hours, associated ipsilateral SNHL (fluctuating), tinnitus, aural fullness. Attacks spontaneous, not positional. Audiometry and ENT referral; acute-attack suppressant only; betahistine trial (benefit uncertain) and diuretics specialist-led.
Scenario E — Drug-Induced Vertigo New medication (aminoglycosides, loop diuretics, antiepileptics, antihypertensives causing postural hypotension) temporally correlated with onset. Medication review first; stop culprit; reassess.
Key variables Was onset positional or spontaneous? Duration of each episode (seconds = BPPV; days = vestibular neuritis; 20 min–12 hrs = Menière's). HINTS exam result. Associated neurological symptoms (headache, diplopia, dysarthria, weakness). Hearing change. Vascular risk factor profile.
Steps:
1
Step 1
History Taking — Open Question First · Targeted Questions · ICE · Psychosocial Context
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The history in vertigo is dominated by two questions: What is the character of the dizziness? (true rotational vertigo vs presyncope vs disequilibrium — these have completely different differentials) and What is the temporal pattern? (episodic brief = BPPV; episodic moderate = Menière's; sustained days = vestibular neuritis; acute constant = posterior fossa stroke). The neurological red flag screen must be completed before any positional testing is performed.
🎓 Consultation opener
"That sounds really frightening and I can imagine it's quite distressing — before I ask you anything specific, can you describe exactly what this dizziness is like? Is the room spinning, or is it more of a lightheaded or off-balance feeling?"
Distinguishing true rotational vertigo from presyncope or disequilibrium at the opening is the most important single question in any dizziness presentation — the diagnosis flows directly from the answer.
1A — Characterise the vertigo: type, temporal pattern, triggers
Question to askWhy it matters clinicallyChanges what?
🟢 OPEN QUESTION"Can you describe what the dizziness is like — is the room spinning around you, or is it more of a lightheaded or unsteady feeling?" The type of dizziness is the single most important discriminating factor. True rotational vertigo (room or self spinning) = vestibular cause (BPPV, vestibular neuritis, Menière's, central). Presyncope (lightheaded, grey-out) = cardiovascular cause. Disequilibrium (unsteady without spinning) = cerebellar, proprioceptive, or medication-related. Multisensory dizziness in older adults is a separate category.Never conflate "dizziness" across these categories. The examiner expects you to establish the type of dizziness before making any diagnosis or performing any positional testing. DiagnosesRisk
Is it related to head position?"Does the spinning happen when you change position — rolling over in bed, looking up, bending down?" Positional triggering is the hallmark of BPPV — otoliths displaced into the semicircular canals respond to gravity with each position change. Vertigo that is purely positional, brief (<1 minute), and reproducible is highly likely to be BPPV. Vertigo present at rest regardless of position suggests a different mechanism (vestibular neuritis, central pathology).The specific movement that triggers BPPV: rolling over in bed is the most common trigger (especially rolling toward the affected ear). Looking up (top-shelf vertigo) = posterior canal BPPV affecting both ears on upward gaze. BPPV vs other
How long does each episode last?"When the vertigo comes on, how long does it last — seconds, minutes, hours, or does it stay all day?" Duration is one of the most diagnostically useful single features: Seconds (<1 minute) = BPPV (otolith settling in canal). 20 minutes–12 hours = Menière's disease (endolymphatic hydrops). Days (continuous, improving over 1–3 weeks) = vestibular neuritis. Constant from onset = central pathology (posterior circulation stroke) or acute labyrinthitis.A patient who says "the room has been spinning continuously for 3 days" has vestibular neuritis or a central cause — not BPPV. This duration question alone narrows the differential dramatically. DDx
Neurological red flags — screen first"Any headache when the vertigo started? Any double vision, slurred speech, difficulty swallowing? Any weakness or numbness in your face, arm, or leg?" This is the critical safety question — the most dangerous diagnosis in vertigo is posterior circulation stroke/TIA. Key red flags: new headache at onset (SAH/posterior fossa haemorrhage), diplopia, dysarthria, dysphagia, facial numbness, limb ataxia or weakness, or sudden onset vertigo in a patient with high vascular risk. Any of these = 999 immediately.The HINTS exam (bedside examination) is more sensitive for posterior fossa stroke than MRI in the first 48 hours — a critical caveat that every GP should know. A normal HINTS in the right hands is more reassuring than a normal MRI in acute vertigo. Emergency if positiveCentral vs peripheral
Associated hearing change or tinnitus?"Have you noticed any change in your hearing, or any ringing in your ears, since the vertigo started?" Vertigo + ipsilateral SNHL + tinnitus = Menière's disease (or AICA territory stroke — the anterior inferior cerebellar artery supplies both the labyrinth and the cerebellum). Vertigo without hearing change = vestibular neuritis (8th nerve dysfunction without cochlear involvement) or BPPV. Acute vestibular syndrome with sudden hearing loss = AICA infarct until excluded.Sudden hearing loss + severe vertigo = AICA stroke until proven otherwise by MRI. Do not diagnose labyrinthitis without excluding a posterior fossa infarct in this presentation. Menière's vs VN vs AICAIf acute hearing loss + vertigo
Recent viral illness?"Have you had a cold, flu, or ear infection in the 1–3 weeks before this started?" Vestibular neuritis classically follows a viral upper respiratory tract infection by 1–3 weeks — the virus (most commonly herpes simplex type 1 reactivation) infects the vestibular nerve causing acute unilateral vestibular deafferentation. In the context of post-viral onset, sustained vertigo lasting days is reassuring as vestibular neuritis (peripheral) rather than a stroke.Vestibular neuritis produces the most severe acute vertigo of any peripheral cause — the severity of the vertigo alone does not distinguish it from a central lesion. The HINTS exam is still needed. Vestibular neuritisVestibular rehab
Vascular risk factors?"Do you have high blood pressure, diabetes, or have you had a stroke or heart attack before?" High vascular risk (hypertension, diabetes, AF, IHD, previous stroke) raises the pre-test probability of a posterior circulation stroke as the cause of acute vertigo — even if the history sounds peripheral. HINTS exam becomes even more important in high-risk patients. A "normal" positional vertigo history in a 70-year-old with AF still requires a thorough central exclusion.In this stem: Mrs Okafor has hypertension, obesity, and is 57 — her vascular risk is not trivial. The HINTS exam is mandatory before reassuring her. Stroke riskRisk stratification
Medication review"Have any new medications been started recently? Do you take any tablets that can affect balance or blood pressure?" Many medications cause dizziness or vertigo: antihypertensives (postural hypotension), antiepileptics (phenytoin, carbamazepine — cerebellar toxicity), aminoglycosides (vestibulotoxic), loop diuretics at high doses, benzodiazepines (impair vestibular compensation), antipsychotics. Review the drug list before attributing vertigo to a primary vestibular cause.In this patient: amlodipine at 10 mg can cause orthostatic hypotension — check lying/standing BP before diagnosing BPPV. Drug-inducedStop culprit
1B — Red flags
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Red Flags — posterior circulation stroke must be excluded first

Red flagWhy dangerousAction
New headache at onset of vertigoSudden onset severe headache with vertigo = posterior fossa haemorrhage (cerebellar bleed) or SAH. Even a mild new headache at the onset of acute vertigo in a hypertensive patient requires CT head urgently. "The worst headache of my life" = SAH pathway immediately.CT head + 999
Diplopia, dysarthria, or dysphagiaBrainstem involvement — diplopia (ocular nerve nuclei), dysarthria (corticobulbar tracts), dysphagia (vagal and hypoglossal nuclei). Any of these alongside vertigo = posterior circulation stroke until proven by DWI-MRI. 999 immediately — stroke pathway regardless of how mild the symptoms appear.999 — stroke pathway
Limb weakness or ataxiaIpsilateral limb ataxia = cerebellar involvement (PICA territory infarct). Contralateral limb weakness = brainstem or corticospinal tract involvement. Either alongside vertigo = posterior circulation stroke. A patient who cannot walk and attributes this to vertigo — perform HINTS and activate stroke pathway.999 — stroke pathway
HINTS exam suggesting central pathologyHINTS (Head Impulse test, Nystagmus type, Test of Skew) is the bedside examination that differentiates peripheral from central vertigo in acute vestibular syndrome. Central HINTS = normal head impulse (no catch-up saccade) + direction-changing nystagmus + vertical skew deviation → posterior fossa lesion until disproven by MRI.999 — immediate stroke pathway
Facial numbness or weaknessFacial nerve or trigeminal involvement alongside vertigo localises the lesion to the posterior fossa. Lateral medullary syndrome (Wallenberg): vertigo + ipsilateral facial numbness + contralateral body pain/temperature loss + ipsilateral Horner's syndrome — classic posterior circulation stroke syndrome.999 — stroke pathway
Sudden hearing loss + severe vertigoSudden combined cochlear and vestibular loss = AICA territory infarct (anterior inferior cerebellar artery supplies both the labyrinth and cerebellum). This is a stroke and not labyrinthitis until MRI proves otherwise. 999 immediately.999 — AICA stroke pathway
1D — ICE
💭 Ideas
"What do you think might be causing this — do you have any thoughts about what's happening?"
Many patients present convinced they have had a stroke — particularly those with vascular risk factors. Others are certain it is "inner ear." Understanding their model allows the GP to position the examination and explanation appropriately — confirming or addressing the model before explaining the actual diagnosis.
😟 Concerns
"I can imagine this is quite frightening — is there something specific you've been worried this might be?"
The dominant fear in acute severe vertigo is stroke — particularly in middle-aged and older adults with known cardiovascular risk factors. Naming this concern directly ("Are you worried this might be a stroke?") and then explaining the examination and its findings gives the patient the specific reassurance they need rather than generic "everything is fine."
🎯 Expectations
"What were you hoping we might be able to do about this today?"
Many patients with acute vertigo expect an MRI brain scan. Understanding this expectation allows the GP to explain why the HINTS bedside examination is more sensitive than MRI in the first 48 hours of acute vestibular syndrome — a counterintuitive but evidence-based and confidence-building explanation.
1E — Psychosocial context
😰 Fear and Acute Distress

Acute severe rotational vertigo is one of the most distressing symptoms a patient can experience — the inability to stand, persistent vomiting, and the sensation of the world spinning are terrifying, particularly when they have occurred suddenly without warning. Acknowledging this experience empathetically before beginning the clinical assessment significantly improves patient cooperation and comfort.

"I can see this is really frightening — vertigo can be incredibly disabling. I'm going to examine you carefully to understand what's causing it and what we can do."
🚗 Driving — Acute Safety Restriction

Acute disabling vertigo requires a driving restriction until fully resolved — DVLA Assessing fitness to drive: a driver liable to sudden and disabling vertigo must not drive and must notify the DVLA; driving may resume once symptoms are satisfactorily controlled (there is no fixed minimum period). This must be addressed at the initial consultation, particularly for a school teacher who may drive to work. The restriction is safety-based and time-limited, not permanent.

"I need to mention that it wouldn't be safe to drive while you're experiencing this level of vertigo — and the DVLA rules say you mustn't drive while you're liable to sudden disabling attacks. Let's review when it's safe to drive again once this is under control."
💼 Work and Daily Function

Vestibular neuritis and BPPV can severely impact a teacher's ability to stand at the front of a classroom, write on a board, or move around the school environment. A fit note may be needed for the acute phase. Understanding what the patient does at work allows appropriate advice — the Epley manoeuvre can be performed the same day, potentially resolving BPPV and enabling rapid return to function.

"Given your job, I understand how disabling this must feel. The good news is that if this is the most common type of inner-ear vertigo, there's a very effective treatment we can do right here today that works quickly for most people."
😔 Chronic Vertigo and Anxiety

Patients who have had recurrent vertigo attacks (as in Menière's disease or BPPV) develop anticipatory anxiety about future episodes, often restricting their lives — avoiding situations where vertigo might occur, stopping exercise, and withdrawing from social activities. Vestibular rehabilitation explicitly targets this avoidance behaviour alongside physical reconditioning of the vestibular system.

"Some people find that after vertigo episodes, they start avoiding movement because they're frightened it will bring it back — but actually, gentle movement and rehabilitation is the best way to get better faster. Have you been finding yourself doing that?"
🤢 Nausea and Wellbeing

The nausea and vomiting of acute vertigo can prevent oral medication, adequate hydration, and engagement with vestibular rehabilitation. Antiemetic medication (prochlorperazine buccal) is both symptom-relieving and necessary for the patient to be able to cooperate with examination and treatment. Its use should be time-limited — prolonged antiemetics impair vestibular compensation.

"The nausea is a really distressing part of this, and I'm going to give you something to help with that first — so we can make you comfortable enough to do the examination and treatment."
🏠 Falls Risk and Safety

Acute severe vertigo creates a significant falls risk — particularly in older adults. The inability to stand without support, combined with the tendency to veer toward the affected side (past-pointing, Romberg), means the patient should not be left alone, and their home environment should be assessed for fall hazards. Handrails, shower seats, and carer support may be needed during the acute phase of vestibular neuritis.

"Given how severe this is, I don't want you to be at home alone during the acute phase — is there someone who can be with you? And please make sure you hold on to something when you move."
🎓 SCA Checkpoint — Step 1TasksRelating to OthersGlobal Skills
Key phrases that score
"Is the room spinning, or is it more of a lightheaded or unsteady feeling — I'm asking because that changes everything about what might be causing it."
"Any headache when this started? Any double vision, slurred speech, or weakness anywhere?"
"How long does each bout of spinning last — seconds when you move, or is it there all the time?"
"Are you worried this might be a stroke — that's a very natural thing to be concerned about with symptoms like this."
Deductions
  • Not distinguishing true vertigo from presyncope or disequilibrium
  • Not screening for neurological red flags before positional testing
  • Not asking about episode duration — missing the BPPV vs Menière's vs VN distinction
  • Performing Dix-Hallpike before excluding central cause
  • Not asking about DVLA driving implications
🔴 Red
Doesn't distinguish vertigo from dizziness; no neurological red flag screen; Dix-Hallpike before history; doesn't ask episode duration; ICE not explored
🟠 Amber
Vertigo confirmed; neurological screen partially done; episode duration asked; vascular risk factors not linked to central exclusion need; ICE partially explored
🟢 Green
Vertigo confirmed as rotational; neurological red flags fully screened; episode duration, triggers, hearing change, recent viral illness, and vascular risk all covered; stroke concern named and addressed; DVLA mentioned
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Step 2
Triage Engine — Emergency · Urgent · Routine
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🔴 Emergency

999 — Stroke Pathway

Do not delay
  • Any neurological red flag + vertigoHeadache at onset, diplopia, dysarthria, dysphagia, facial numbness, limb weakness/ataxia → 999
  • Central HINTS examNormal head impulse (no catch-up saccade) + direction-changing nystagmus + skew deviation → posterior fossa stroke
  • Sudden hearing loss + severe vertigoAICA stroke territory — 999 immediately; MRI DWI urgently
  • Cerebellar bleed (sudden onset headache + ataxia + vomiting)CT head emergency — neurosurgical referral if cerebellar haematoma
🟠 Urgent

Same-Day / 48-Hour Assessment

Rapid assessment
  • Acute vestibular syndrome — cannot exclude stroke clinicallyIf HINTS equivocal, not reliable, or no trained clinician available → immediate stroke pathway referral; imaging per local protocol (NICE NG127)
  • First attack of Menière's diseaseENT urgent referral for audiogram, tympanometry, and treatment initiation
  • BPPV not resolving with Epley after 3 treatmentsENT referral for vestibular assessment; consider atypical canal or central cause
  • Vertigo with falls causing injuryUrgent vestibular rehabilitation referral; falls assessment; occupational therapy
🟢 Routine

Primary Care Management

GP management
  • BPPV — positive Dix-Hallpike, central features absentEpley manoeuvre in clinic today; Brandt-Daroff exercises at home; review 2 weeks
  • Vestibular neuritis (post-viral, peripheral HINTS)Short-course prochlorperazine (2–3 days) + vestibular rehabilitation; no routine steroids; GP review 1 week
  • Drug-induced vertigoStop or reduce culprit medication; review in 2–4 weeks
  • Recurrent BPPVEpley in clinic; teach Brandt-Daroff for self-management; ENT if more than 3 recurrences/year
🎓 SCA Checkpoint — Step 2Tasks
🔴 Red
Diagnoses BPPV without excluding stroke; no HINTS exam; triage decision not linked to named clinical features
🟠 Amber
Red flags screened; triage broadly correct; HINTS not mentioned; reasoning not shared with patient
🟢 Green
Red flags explicitly excluded; HINTS exam planned before Epley; triage decision linked to named features; reasoning shared with patient including stroke concern addressed
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Step 3
Do I Need This Examination?
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The HINTS examination is the most important bedside test in acute vestibular syndrome — it is more sensitive than MRI in the first 48 hours for posterior fossa stroke. HINTS should be used only in ongoing acute (continuous) vestibular syndrome and only by a clinician trained and experienced in it — it is not a test for brief positional episodes; if not trained, refer immediately via the stroke pathway (NICE NG127). Dix-Hallpike testing must only be performed AFTER central causes have been clinically excluded or the HINTS exam result is consistent with a peripheral lesion. Never perform Dix-Hallpike as the first examination in a patient with acute vertigo without screening for central features first.
ExaminationWhy it mattersFinding + actionMandatory?
HINTS Exam (Head Impulse, Nystagmus type, Test of Skew)The most important bedside test in acute vestibular syndrome. More sensitive than MRI DWI in the first 48 hours for posterior fossa stroke. Peripheral HINTS (reassuring): abnormal head impulse (catch-up saccade present) + direction-fixed nystagmus + no skew. Central HINTS (stroke): normal head impulse + direction-changing nystagmus + skew deviation.Each component independently contributes to the assessment. A normal head impulse test (no catch-up saccade) in a patient with acute vertigo is a red flag for central pathology — the peripheral vestibular system is not damaged if the vestibulo-ocular reflex is intact.Central HINTS = 999. Peripheral HINTS = proceed to Dix-Hallpike. Equivocal HINTS, or no trained clinician = immediate stroke pathway referralYES — first in AVS
Dix-Hallpike testDiagnostic test for posterior canal BPPV — the patient is moved rapidly from sitting to lying with the head rotated 45° toward the affected side. Positive result: geotropic torsional upbeat nystagmus after a latency of a few seconds, lasting under 1 minute, that fatigues on repeat testing.Reported sensitivity 82%, specificity 71% for posterior canal BPPV (AAO-HNS, international — Bhattacharyya 2017). Perform only after central cause has been clinically excluded. If positive, the Epley manoeuvre can be performed immediately in the same clinic visit.Positive (torsional, fatiguing nystagmus) → BPPV confirmed → Epley manoeuvre. Negative (direction-changing, non-fatiguing, or vertical pure nystagmus) → consider central or atypical canal BPPVYES — if peripheral HINTS
Romberg test and gait assessmentRomberg positive (falls with eyes closed) indicates absent proprioceptive or vestibular compensation. Gait in peripheral vertigo: cautious, past-pointing to affected side, able to tandem gait with effort. Central ataxia: wide-based, unable to tandem gait, disproportionate severity of ataxia relative to nystagmus.A patient with "vertigo" who cannot walk at all and has severe truncal ataxia has central vertigo until proven otherwise — the degree of ataxia in peripheral vertigo rarely completely prevents walking.Severe truncal ataxia disproportionate to nystagmus → central pathology → 999 or urgent MRI. Mild to moderate Romberg-positive → peripheral → proceed with vestibular assessmentYES — always in vertigo
BP lying and standingExcludes postural hypotension as a cause of dizziness — particularly relevant in this patient on amlodipine 10 mg (a significant vasodilator at high dose). A systolic drop ≥20 mmHg with symptoms on standing = orthostatic hypotension, which can be mistaken for or contribute to vestibular symptoms.Never perform Dix-Hallpike in a patient who may have orthostatic hypotension without first excluding it — the position change could precipitate a presyncopal episode during the test.Systolic drop ≥20 mmHg → orthostatic hypotension → medication review; lying/standing BP protocol; adjust amlodipine doseYES in this patient
Otoscopy + brief hearing assessmentIdentifies middle ear pathology — otitis media, cholesteatoma, or effusion — as a cause of vestibular symptoms. Whisper test or tuning fork test provides basic hearing assessment — unilateral hearing loss + vertigo requires ENT and MRI IAM exclusion of acoustic neuroma or AICA stroke.In this stem: otoscopy is unlikely to find pathology, but it is a necessary examination to complete the vestibular assessment systematically.Normal → proceed. CSOM or cholesteatoma → ENT. Hearing loss unilateral → MRI IAM or if acute onset + severe vertigo → 999 AICA stroke pathwayYes — if hearing change
🎓 SCA Checkpoint — Step 3Tasks
🔴 Red
Dix-Hallpike performed before HINTS or neurological screen; HINTS exam not mentioned; lying/standing BP not checked in antihypertensive patient
🟠 Amber
HINTS mentioned; Dix-Hallpike planned after; lying/standing BP not mentioned; gait not assessed; findings not linked to central vs peripheral distinction
🟢 Green
HINTS first and result interpreted; Dix-Hallpike only after peripheral confirmed; lying/standing BP in this antihypertensive patient; gait assessed; all findings explicitly linked to peripheral vs central decision
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Step 4
Do I Need This Investigation?
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InvestigationClinical question it answersKey result + action
MRI DWI (diffusion-weighted imaging)The definitive investigation for posterior circulation stroke in vertigo — detects acute ischaemia within hours. However, sensitivity in the first 48 hours is under 80% for posterior fossa — HINTS exam by an experienced clinician is more sensitive. MRI is mandatory if central features are present or HINTS is equivocal. CT is inadequate for posterior fossa lesions (bone artefact).Acute DWI restriction → ischaemic stroke → stroke pathway. Normal → posterior fossa stroke not excluded if within 48 hours — reimage at 48–72 hours if clinical suspicion remains
CT headLess sensitive than MRI for posterior fossa pathology but rapidly available for haemorrhage exclusion. Indicated for sudden onset severe headache + vertigo (subarachnoid or cerebellar bleed) when MRI is not immediately available. Not appropriate for ischaemic posterior circulation stroke exclusion.Cerebellar haematoma → neurosurgery. Normal CT + high suspicion of ischaemia → MRI DWI as next step
Pure-tone audiogramFor recurrent vertigo with associated hearing change — confirms SNHL pattern and severity. Mandatory for Menière's disease workup. Also required for vestibular schwannoma (acoustic neuroma) assessment alongside MRI IAM. Not needed for straightforward first-episode BPPV.Low-frequency SNHL fluctuating → Menière's pattern. Unilateral SNHL → MRI IAM. Normal → peripheral vestibular cause more likely
Blood tests (FBC, glucose, TFTs)Anaemia causes lightheadedness often conflated with vertigo. Hypoglycaemia mimics acute vestibular syndrome. Hypothyroidism causes multisensory dizziness. Not required for straightforward acute BPPV but appropriate for first presentation of sustained vertigo without obvious cause.Anaemia → treat; reassess dizziness. Hypoglycaemia → treat; review medications. Hypothyroidism → treat; dizziness often improves
Lying and standing BP (formal protocol)Formal 3-minute standing protocol excludes orthostatic hypotension as a cause of dizziness — particularly important in patients on antihypertensives (especially amlodipine at 10 mg). Medication-induced orthostatic hypotension can co-exist with BPPV.Systolic drop ≥20 mmHg → orthostatic hypotension → medication review; consider amlodipine dose reduction
🎓 SCA Checkpoint — Step 4Tasks
🔴 Red
Outpatient CT/MRI requested from primary care instead of immediate stroke-pathway referral for central features or equivocal HINTS; "routine bloods" without clinical rationale
🟠 Amber
MRI planned for high-risk patients; audiogram not considered; investigations not explained to patient
🟢 Green
Central features → immediate stroke pathway (imaging per local protocol; an early normal CT does not exclude posterior circulation stroke); audiogram if hearing change; investigations explicitly linked to clinical questions; explains why HINTS is more sensitive than MRI in first 48 hours
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Step 5
Reaching a Diagnosis & DDx — Explained in Plain Language
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🗣️ Explaining Vertigo in Plain Language

"Your balance system uses signals from three places — your inner ear, your eyes, and the pressure sensors in your joints — and your brain combines all three to work out where you are in space. What's happened is that the balance organ in your inner ear has sent an incorrect signal — probably because a tiny crystal has shifted out of place inside the balance organ. When you turn your head, this crystal moves and sends a confusing message to your brain, which interprets it as spinning. The good news is that this is one of the most treatable types of vertigo — there's a specific head movement sequence I can do with you now that repositions the crystal. Most people feel significantly better after just one treatment."

A — Diagnosable + Treatable in Primary Care
GP can diagnose and treat

BPPV — Posterior Canal (most BPPV)

Positional rotational vertigo <1 minute triggered by rolling over, looking up. Dix-Hallpike positive: torsional upbeat nystagmus after a latency of a few seconds, fatigues on repeat. Treatment: Epley manoeuvre — most people improve after one or more manoeuvres. Brandt-Daroff exercises for home use. Review in 2 weeks.

Vestibular Neuritis

Acute onset continuous vertigo for days, post-viral, no hearing loss. HINTS peripheral (catch-up saccade on head impulse). Dix-Hallpike negative. Treatment: prochlorperazine (acute, 2–3 days), vestibular rehabilitation; steroids are not started routinely (evidence mixed) — sudden hearing loss follows the urgent ENT pathway (NICE NG98). Resolves over 1–6 weeks.

B — Refer to ENT / Specialist
Specialist needed

Menière's Disease

Episodic vertigo (20 min–12 hrs) + unilateral SNHL (fluctuating) + tinnitus + aural fullness. Confirmed by ENT with audiogram. Treatment: acute-attack suppressant only; a betahistine trial may be considered (benefit uncertain); dietary trigger trial without excessive salt restriction; diuretics and intratympanic treatment specialist-led. ENT referral for diagnosis and management.

BPPV — Horizontal or Anterior Canal (Atypical)

Dix-Hallpike negative or atypical; horizontal canal BPPV confirmed by supine roll test (geotropic or ageotropic nystagmus). Requires ENT specialist positional manoeuvre (Gufoni or Barbecue roll) or vestibular physiotherapy. Refer if GP Epley fails after 3 attempts.

Vestibular Migraine

Recurrent episodes of vertigo lasting 5 minutes to 72 hours associated with migraine features (headache, photophobia, phonophobia). No clear central HINTS findings. ENT or neurology referral for diagnosis. Treated as migraine prophylaxis plus vestibular rehabilitation.

C — Emergency — Act Now
999 immediately

Posterior Circulation Stroke / TIA

HINTS central features + vascular risk factors. PICA (posterior inferior cerebellar artery) → Wallenberg syndrome. AICA (anterior inferior cerebellar artery) → vertigo + sudden hearing loss + ipsilateral facial numbness. HINTS: normal head impulse, direction-changing nystagmus, skew deviation → 999. MRI DWI urgently.

Cerebellar Haemorrhage

Sudden onset severe vertigo + headache + inability to stand + vomiting in a hypertensive patient. CT head emergency. Neurosurgical referral if cerebellar haematoma with mass effect. 999 immediately. Do not attribute to BPPV without CT exclusion of haemorrhage in a hypertensive patient with severe acute presentation.

📊 HINTS Exam Interpretation Guide
ComponentPeripheral (reassuring)Central (red flag → 999)
Head Impulse TestAbnormal — catch-up saccade visible after rapid head thrust (VOR impaired = peripheral lesion)Normal — smooth conjugate eye movement (VOR intact = central lesion)
Nystagmus TypeDirection-fixed — nystagmus beats the same direction regardless of gaze directionDirection-changing — nystagmus changes direction with different gaze positions; or vertical; or purely horizontal without head impulse abnormality
Test of SkewNo vertical skew deviation on cover-uncover testSkew deviation present — one eye higher than the other on cover-uncover test
Overall interpretationAll 3 peripheral findings = INFARCT unlikely — peripheral cause probableAny 1 central finding = posterior fossa lesion until excluded by MRI DWI
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Step 6
If Referral Is Needed — What the GP Does Before & During
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ConditionUrgencyWhat GP does before referralWhat GP must NOT do
Posterior circulation stroke / central HINTS999 immediatelyHINTS exam result documented. Activate stroke pathway. Note time of symptom onset (thrombolysis window). Record all neurological deficits. Lay patient flat. Do not give aspirin before brain imaging has excluded haemorrhage (NICE NG128).Do not give antiemetics and "wait and see" in a patient with central HINTS or neurological features. Do not allow a high-vascular-risk patient with unexplained sustained vertigo to leave without being seen by a physician today.
BPPV — confirmed, Epley performedRoutine ENT if fails ×3Perform Epley manoeuvre in clinic (see 7C). Provide Brandt-Daroff exercise leaflet. Advise no driving until vertigo resolves. Review 2 weeks — if not resolved, repeat Epley. After 3 failed treatments → ENT referral for vestibular physiotherapy or atypical canal assessment.Do not prescribe long-term prochlorperazine for BPPV — antiemetics impair vestibular compensation and delay recovery. Use only for acute nausea control (maximum 2–3 days).
Vestibular neuritis — confirmed peripheralRoutine vestibular rehabPrescribe prochlorperazine buccal (short course); steroids not started routinely (evidence mixed; sudden hearing loss → urgent ENT pathway, NICE NG98); refer vestibular rehabilitation physiotherapy; advise falls precautions; DVLA advice; review 1 week.Do not prescribe antiemetics for more than 3 days — they suppress vestibular compensation and significantly prolong the recovery period. Vestibular rehabilitation is the cornerstone of treatment, not medication.
Menière's disease (suspected)4-week ENTAudiogram; dietary trial of avoiding individual triggers (evidence for salt thresholds limited); betahistine trial only after ENT assessment (benefit uncertain); prochlorperazine for acute attacks only; DVLA advice (not to drive during attacks). Refer ENT for diagnostic confirmation and specialist management.Do not start betahistine before ENT confirms Menière's diagnosis — the triad must be confirmed on audiometry and clinical assessment before initiating long-term treatment.
7
Step 7
Management — Expectation · Goals · Epley · Drug Cards · Psychosocial · Follow-Up · Safety-Netting
▲collapse
7A — Address the patient's expectation first
🤝
The patient with acute vertigo is frightened and expects either a brain scan or to be told there is nothing wrong. Address both.
1
Validate

Acknowledge how frightening acute vertigo is before any clinical explanation.

"I can see this has been really terrifying — sudden severe spinning and vomiting is one of the most distressing things to experience. I want to reassure you that I'm going to examine you carefully to understand exactly what's happening."
2
Explain

Explain the HINTS exam and why it is actually more informative than a brain scan in the first 48 hours — this is counterintuitive but builds confidence in the examination.

"I'm going to do a specific clinical eye examination now — and actually, for the type of vertigo you have, this bedside test is more sensitive for detecting a brain problem than an MRI scan in the first 48 hours. So this is the most important test we can do right now."
3
Offer

If BPPV is confirmed — offer the Epley manoeuvre today. The immediate treatment offer transforms the consultation from a diagnostic to a therapeutic encounter.

"The good news is that if this is the type I think it is — the crystal type — I can treat it right here today with a specific head movement sequence. Most people feel significantly better immediately afterwards."
7C — The Epley Manoeuvre (BPPV treatment) — teach and perform
🔄
Epley Manoeuvre
Most people improve after one or more manoeuvres — perform in clinic if trained
Mechanism

The Epley manoeuvre repositions otoliths (calcium carbonate crystals) that have drifted from the utricle into the posterior semicircular canal, moving them out of the canal and back into the utricle through a series of sequential head positions that use gravity to guide the crystals. Each position is held until vertigo and nystagmus settle, and for at least about 30 seconds.

Steps

1. Dix-Hallpike to affected side (hold ≥30 sec, until settled). 2. Rotate head 90° to opposite side (hold as above). 3. Roll body toward unaffected side, head now facing floor (hold as above). 4. Sit patient up slowly. 5. Head returns to midline. No routine post-manoeuvre restrictions (sleeping upright, collar) are needed.

Reassess and repeat if appropriate; persisting despite correctly performed treatment → reconsider canal/diagnosis and refer
🏃
Brandt-Daroff Exercises
Home self-management — twice daily until symptoms resolve
Mechanism

Brandt-Daroff exercises habituate the central nervous system to the abnormal vestibular signal by repeated exposure — less specific than Epley but easy to self-administer and effective for residual or recurrent symptoms. Also suitable for patients who cannot tolerate the Epley manoeuvre.

Practical

Sit upright → rapidly lie on affected side (head 45° toward ceiling) → hold 30 sec or until vertigo stops → return to sitting → hold 30 sec → lie on opposite side → hold 30 sec. Repeat 5 times per session, twice daily, for 2 weeks.

Less targeted than Epley; a home Epley may suit confirmed unilateral posterior-canal BPPV
🧘
Vestibular Rehabilitation
Referral to vestibular physiotherapy for vestibular neuritis
Mechanism

Vestibular rehabilitation physiotherapy promotes central compensation for peripheral vestibular deafferentation through structured exercises — gaze stabilisation, habituation exercises, and balance training. The cornerstone of vestibular neuritis management and significantly more effective than antiemetics as a long-term treatment.

Practical

Refer to community vestibular physiotherapy or ENT physiotherapy service. Programme typically 8 sessions over 6 weeks. Exercises must be done consistently — avoiding movement (common in patients with vertigo) dramatically slows recovery and maintains disability.

Vestibular rehabilitation improves persistent imbalance — more effective than medication for vestibular neuritis
🚗
DVLA Advice
No driving while liable to sudden disabling vertigo
Legal requirement

DVLA Group 1 (car): must not drive while experiencing disabling vertigo. Can resume when symptoms are satisfactorily controlled and unlikely to cause sudden disabling recurrence; notify the DVLA if attacks are sudden and disabling (no fixed minimum period in DVLA guidance). Group 2 (HGV/bus): specialist assessment required before return to driving.

Practical

Advise explicitly: "You should not drive until the vertigo has resolved or is well controlled and you are not at risk of sudden disabling attacks." Document this advice in the clinical notes. Notify the patient that they must inform the DVLA if episodes are recurrent and disabling.

DVLA advice is mandatory and must be documented — failure to advise is a medicolegal risk
🏠
Falls Prevention
Acute phase — home safety assessment
Practical

During acute phase of vestibular neuritis or severe BPPV: remove trip hazards; hold handrails at all times; shower seated if possible; do not climb ladders or stairs alone; ensure someone is at home. Occupational therapy falls assessment if recurrent or severely impaired.

Falls prevention during acute vestibular episode reduces serious injury risk in older adults significantly
💊
Antiemetics — Short Course Only
Maximum 2–3 days — longer impairs vestibular compensation
Critical principle

Prochlorperazine provides acute symptom relief for nausea and vertigo — but if continued beyond 2–3 days it suppresses the central compensation mechanism that allows the brain to adapt to vestibular deafferentation. Long-term antiemetics for vertigo significantly prolong disability and are one of the most common management errors in vestibular disorders.

Stop antiemetics within 3 days — vestibular rehabilitation, not medication, is the long-term solution
7F — Drug reference cards: vertigo pharmacological management
Prochlorperazine (Stemetil)
Buccal 3–6 mg BD or oral 5 mg TDS (SPC) — acute symptom control in vestibular neuritis; not a BPPV treatment
Acute vertigomax 2–3 days
✓ Use when
Acute nausea and vomiting preventing oral intake or cooperation with vestibular examination
Short-term symptomatic relief while vestibular compensation begins
Buccal (Buccastem 3 mg) preferred if vomiting prevents oral tablets
✗ Time-limit strictly
Maximum 2–3 days — beyond this duration, suppresses vestibular compensation and prolongs disability
Do NOT prescribe long-term "for dizziness" — this is a very common and harmful prescribing error
Elderly — increased risk of extrapyramidal side effects; consider a lower dose or alternative antiemetic
💬 Counselling

"This tablet will help with the nausea and spinning sensation in the short term. It's very important that you take it for no more than 3 days — after that, the tablet actually slows down your brain's ability to adapt and recover. The recovery really comes from the exercises, not the tablet."

The most examinable prescribing point for prochlorperazine in vestibular disorders: maximum 2–3 days. Prescribing it long-term for chronic dizziness is a very common error in primary care and is specifically tested in the SCA. "I'm giving you this for the acute symptoms, but we'll stop it after 3 days — longer than that actually slows recovery."

Prednisolone — Vestibular Neuritis
Not started routinely in primary care — evidence mixed (Cochrane review, Fishman 2011)
Vestibular neuritisNot routine
✓ Initiate when
Do not start routinely for vestibular neuritis or presumed viral labyrinthitis (evidence mixed)
Sudden hearing loss (≤3 days, within 30 days) → ENT/ED within 24 hours; steroid decisions by the urgent ENT pathway (NICE NG98)
Do not prescribe steroids in primary care as a substitute for urgent ENT assessment
✗ Caution
Diabetes — monitor blood glucose closely during course
If ENT starts steroids for sudden hearing loss, follow their dose and duration
💬 Counselling

"Steroid tablets haven't been shown clearly to help this kind of balance-nerve inflammation, so I won't start them routinely. What speeds recovery is moving and the balance exercises. If your hearing suddenly drops, contact us the same day — that needs an ear specialist within 24 hours."

Steroids for vestibular neuritis are not started routinely in primary care — the evidence is mixed (Cochrane review, Fishman 2011: insufficient evidence from small trials); steroid decisions for sudden hearing loss belong to the urgent ENT pathway (NICE NG98). Pairing this with the explicit statement "maximum 2–3 days of prochlorperazine" demonstrates systematic management thinking.

Betahistine — Menière's Disease
Serc 16 mg TDS — only after ENT confirms Menière's diagnosis
Menière's only24–48 mg daily in divided doses · review 3–6 months
✓ Only when Menière's confirmed
Menière's disease confirmed by ENT with audiogram-supported diagnosis
Time-limited trial — benefit uncertain (Cochrane 2016; BEMED 2016); review at 3–6 months and stop if ineffective
Alongside a trial of avoiding individual dietary triggers — evidence for specific salt thresholds is limited; avoid excessive restriction
✗ Do not use empirically
Do not prescribe betahistine for any acute vertigo without ENT confirming Menière's
Asthma — histamine pathway; caution; peptic ulcer risk
💬 Counselling

"This tablet needs to be taken three times a day every day — it works by reducing the fluid pressure in the inner ear, which is what causes your attacks. You won't feel it working immediately, and it doesn't help everyone — we'll review it at 3 to 6 months and stop it if it isn't helping."

Key SCA point: betahistine is for confirmed Menière's only. "I wouldn't start betahistine today — we need ENT to confirm the diagnosis first with a formal hearing test." This demonstrates safe prescribing and the importance of not jumping to treatment before diagnosis is established.

Aspirin 300 mg (stroke: only after brain imaging; TIA: immediately)
Suspected posterior circulation stroke — do not give in the surgery while awaiting 999; hospital gives it once imaging excludes haemorrhage (NICE NG128)
Stroke / TIA300 mg
✓ Give when
Acute stroke: once brain imaging has excluded intracerebral haemorrhage — as soon as possible, within 24 hours (NICE NG128; hospital team)
Suspected TIA (symptoms fully resolved), not anticoagulated: aspirin 300 mg started immediately (NICE NG128)
✗ Do NOT give if
Already on anticoagulation — do not add aspirin; risk of haemorrhage
Suspected stroke with ongoing deficit before brain imaging — haemorrhage not excluded; withhold until CT result
💬 Counselling

"I'm not giving you aspirin right now — the hospital needs to scan your brain first to make sure there is no bleeding, and they will start the right treatment straight after. The ambulance is on its way."

NICE NG128: in acute stroke, aspirin 300 mg is given only after brain imaging has excluded haemorrhage — so do not give it in the surgery while awaiting 999; for suspected TIA with resolved symptoms, start aspirin 300 mg immediately. Stating this distinction clearly while arranging 999 transfer demonstrates safe emergency management and scores in Tasks.

7G — Psychosocial impact: fear, avoidance, and return to function
🤝
Living with vertigo — acute and chronic impact on daily life
😰
Fear of Recurrence

After a first episode of BPPV or vestibular neuritis, patients develop significant fear of recurrence — particularly when triggers (position change for BPPV, sudden head movements for VN) are unavoidable in daily life. Anticipatory anxiety leads to avoidance of any head movement, which paradoxically delays vestibular compensation and prolongs recovery.

The most important rehabilitation instruction is to move despite the discomfort — habituation exercises work by exposing the vestibular system to the triggering stimulus repeatedly until the central nervous system stops perceiving it as a problem. Avoidance is the enemy of recovery.

"I know it's tempting to keep very still when you're dizzy, but actually the more you move — carefully and safely — the faster your balance system will adapt and recover. Avoiding movement is the one thing that makes recovery slower."
🚗
Driving and Occupation

A teacher with acute disabling vertigo cannot drive to work and cannot stand at the front of a classroom safely — at least in the acute phase. A fit note for the acute phase may be needed. DVLA advice (no driving while liable to sudden disabling vertigo) must be given and documented at this consultation.

The good news for BPPV: the Epley manoeuvre often resolves symptoms within minutes to hours, enabling a rapid return to function. For vestibular neuritis, the acute phase lasts 1–3 weeks — a fit note for this period is appropriate, with review at the end of that period.

"Given how severe this is right now, I'm going to give you a fit note for the next 1–2 weeks — and I'd like you to come back then so we can see how the recovery is going and when it's safe to return."
🧠
Chronic Dizziness and Functional Disorder

Persistent Postural-Perceptual Dizziness (PPPD) is a functional vestibular disorder that develops in a significant proportion of patients following acute vestibular events. The patient continues to experience chronic dizziness long after the acute event has resolved, driven by maladaptive neural compensation and anxiety.

PPPD is managed with vestibular rehabilitation, CBT, and SSRIs in selected cases — not with antiemetics. Recognising this transition from acute organic vertigo to functional dizziness is important; it changes the management from investigation to rehabilitation and psychological support.

"Sometimes after vertigo, the dizziness can linger longer than the original cause would explain — this is quite common and it's something we address with specific exercises and, in some cases, talking therapy. It's very treatable."
🏠
Falls and Home Safety

Acute severe vertigo is a significant falls risk — particularly in older adults or those with pre-existing balance difficulties. The combination of vestibular deafferentation, postural instability, and vascular risk factors (in this patient: hypertension) creates a dangerous context for falls with potentially serious consequences.

Practical acute phase advice: remove trip hazards, use handrails, shower seated, do not climb ladders or stairs alone, have someone at home during the acute phase. Occupational therapy falls assessment if recurrent or severely impaired after the acute phase.

"For the next few days while the vertigo is at its worst, please make sure you're holding on to something whenever you move, and that there's someone with you at home — the risk of a fall is real and I don't want you to get hurt."
7H — Follow-up
1
2 Weeks — BPPV response to Epley

If BPPV: check whether Epley resolved symptoms. If resolved → reassure; Brandt-Daroff as ongoing prevention; DVLA review. If not resolved → repeat Epley (up to 3 times before ENT referral). If vestibular neuritis: assess recovery; stop prochlorperazine if still on it; vestibular rehabilitation under way.

Symptom reviewDVLA check
2
4–6 Weeks — Recovery assessment + return to driving

Review vestibular neuritis recovery; assess readiness to return to driving (once symptoms are controlled and sudden disabling attacks are unlikely); vestibular rehab progress; PHQ-9 if anxiety or chronic dizziness developing; fit note review for return to work.

Driving returnPHQ-9
3
3 Months — If symptoms persist

If dizziness persists beyond 6 weeks after vestibular neuritis → consider PPPD; refer vestibular physiotherapy. If BPPV recurring frequently → ENT referral; explore horizontal canal variant. Audiogram if any hearing change has developed since presentation.

PPPD assessmentAudiogram if hearing change
7J — Safety-netting

⚠ Three scenario-specific phrases

🔴 Emergency — neurological symptoms
"If the vertigo comes back and you notice any headache, double vision, slurred speech, weakness or numbness in your face or limbs, or if you feel that you're going to faint — call 999 immediately. These symptoms alongside dizziness can indicate a stroke and require immediate medical attention."
This safety-net phrase covers the neurological red flags for posterior circulation stroke that can present as or alongside vertigo. Giving these specific symptoms allows the patient to self-triage rapidly. Documenting this advice protects the GP medico-legally in a presentation where a stroke cannot be excluded with absolute certainty in primary care.
💊 Antiemetic time limit
"The prochlorperazine tablets I've prescribed are for the nausea only — please stop them after 3 days even if you still feel dizzy. Taking them for longer actually slows down your brain's ability to recover from the balance problem. The exercises are what will treat the underlying cause."
Long-term prochlorperazine prescription for vertigo is one of the most common and harmful management errors in primary care. Pre-warning the patient to stop after 3 days prevents both the patient and future prescribers from continuing it indefinitely, and reinforces that vestibular rehabilitation is the therapeutic goal.
🚗 DVLA driving restriction
"You should not drive until the vertigo has gone or is well controlled and you're no longer at risk of sudden attacks. If you drive and have an episode of vertigo at the wheel, you could cause a serious accident. Please arrange alternative transport for now, and let's review this at your next appointment."
DVLA guidance: a driver liable to sudden and disabling vertigo must not drive and must notify the DVLA until symptoms are satisfactorily controlled. Documenting this advice explicitly at the first consultation is mandatory for medico-legal protection and is a DVLA statutory requirement.
🎓 SCA Checkpoint — Step 7 (Final)TasksRelating to OthersGlobal Skills
Closing the consultation
"To summarise: I've done the eye examination to check for a brain cause — that looked reassuring. The hearing and balance test suggests this is the crystal type of vertigo, and I'm going to treat it now with the head repositioning sequence."
"The nausea tablet is for 3 days only — please stop it after that even if you're still dizzy, because longer than that slows recovery."
"If you develop any headache, double vision, or weakness alongside the dizziness — call 999 immediately. And please don't drive until the vertigo is gone or well controlled."
"Is there anything you'd like to ask before I do the treatment?"
Deductions — closing
  • Prescribing long-term prochlorperazine without the 3-day limit
  • Performing Epley without first doing HINTS exam
  • Not mentioning DVLA driving restriction
  • Not giving neurological red flag safety-net
  • Not offering Epley manoeuvre in clinic when BPPV is confirmed
  • Diagnosing BPPV without Dix-Hallpike test
Tasks — full criteria
  • Neurological red flags screened before positional testing
  • HINTS exam performed and interpreted correctly
  • Dix-Hallpike only after peripheral HINTS confirmed
  • Epley manoeuvre offered and performed in clinic (BPPV)
  • Prochlorperazine prescribed with explicit 3-day maximum
  • DVLA driving advice (no driving while liable to sudden disabling vertigo) given and documented
Relating to Others — full criteria
  • Acknowledged how frightening acute vertigo is
  • Stroke concern named and addressed directly before examination
  • HINTS exam explained as more sensitive than MRI in first 48 hours
  • Epley offered as treatment today — transformed expectation
  • Falls risk and home safety discussed with genuine concern
  • Closing question asked before the Epley is performed
🔴 Red
Epley before HINTS; long-term prochlorperazine; no DVLA; no neurological safety-net; stroke concern not addressed; no Epley offered for confirmed BPPV
🟠 Amber
HINTS done; Epley performed; prochlorperazine 3-day limit not stated; DVLA mentioned briefly; stroke concern not named before examination
🟢 Green
HINTS first then Dix-Hallpike; Epley in clinic; prochlorperazine 3-day limit explicitly stated; DVLA documented; neurological red flag safety-net given; stroke concern named and addressed empathetically; falls risk addressed
Vertigo — SCA Consultation Scorecard
Based on the official SCA Consultation Tool · RAG self-assessment
0/ 33 pts
🌐
Global Skills
0/7
✓
Tasks
0/15
🤝
Relating to Others
0/11
011172533
Fail
Borderline
Pass
Strong pass
📋
Complete the checklist above to see your score interpretation
"This is terrifying — the room won't stop spinning and I keep being sick. I'm scared something's happened to my brain."
Who you are

Helen Okafor, 57, secondary school teacher. Woke this morning and when she rolled over in bed the room started spinning violently. Vomited twice. Now sitting very still in the GP chair. Describes the spinning as definitely the room moving — not lightheadedness. Hypertension on amlodipine 10 mg + ramipril 5 mg. BMI 32. No headache. No diplopia, slurred speech, or weakness. No hearing change. No recent viral illness.

Hidden concern

You are convinced this is a stroke. Your father died of a stroke at 62, and you know you have high blood pressure. Will not say this directly unless asked — but if the doctor asks "Is there something specific you're worried this might be?" → "My dad died of a stroke — I thought this is what it must feel like."

Symptoms if asked
  • Spinning started when she rolled over in bed — to the right side
  • Worst in the first 30 seconds, then settles a bit
  • Happens every time she moves her head
  • No spinning when sitting completely still
  • No headache, no double vision, no speech difficulty, no weakness
  • Hearing is normal — no change noticed
Responses and challenges
  • If MRI is offered: "Shouldn't I have a brain scan to be safe?" → responds positively if HINTS superiority is explained
  • If Epley is offered: initially reluctant ("Moving my head sounds terrifying") → agrees if it is explained that most people improve after one or more manoeuvres
  • On driving: "I need to drive to school — is there any flexibility?" → accepts restriction when safety framed empathetically
  • On prochlorperazine: "Can't I just take the tablets for longer to get through this?" → accepts 3-day limit if the recovery rationale is explained clearly
"Are you sure this isn't a stroke? Shouldn't I be going to hospital?"

Resolution: Helen accepts the diagnosis and treatment if the candidate: (1) directly names her stroke fear and addresses it before examining; (2) explains HINTS as more sensitive than MRI in first 48 hours; (3) performs or clearly describes the Epley manoeuvre; (4) gives the 3-day prochlorperazine limit with clear rationale; (5) addresses DVLA with empathy for its impact on her work as a teacher. She will ask about an MRI — this is the key expectation management moment of the consultation.

🏥
Clinic Quick Reference
Vertigo — Clinical Decision Framework
NICE NG127 · BPPV Clinical Guidelines (international) · HINTS Examination Protocol
▼expand
🚦 1 — Triage + HINTS Guide
Acute vertigo → neurological screen first → HINTS exam → Dix-Hallpike only if peripheral HINTS
🔴 999 — Central
  • Headache at onset
  • Diplopia/dysarthria/dysphagia
  • Limb weakness or ataxia
  • Central HINTS (normal HIT, direction-changing Nystagmus, Skew deviation)
  • Sudden hearing loss + severe vertigo (AICA)
999 + stroke pathway immediately
🟢 Peripheral → Primary Care
  • Peripheral HINTS (catch-up saccade, direction-fixed Nystagmus, no Skew)
  • BPPV: Dix-Hallpike + Epley in clinic
  • Vestibular neuritis: short antiemetic + rehab
Epley / short antiemetic / vestibular rehab
HINTS Interpretation (3 components)
Peripheral (all 3):
HIT: Catch-up saccade PRESENT ✓
Nystagmus: Direction-FIXED ✓
Skew: NO vertical skew ✓

Central (any 1 = red flag):
HIT: No catch-up saccade ✗
Nystagmus: Direction-CHANGING ✗
Skew: Skew deviation PRESENT ✗
📊 2 — Key Numbers
3 days
Prochlorperazine maximum — longer impairs vestibular compensation
1+
Most BPPV improves after one or more Epley manoeuvres
≥30 sec
Hold each Epley position until vertigo and nystagmus settle
DVLA
No driving while liable to sudden disabling vertigo; resume when controlled
48 hrs
Window when HINTS (expert examiners, Kattah 2009) outperformed early MRI for posterior fossa stroke
82%
Reported Dix-Hallpike sensitivity for posterior canal BPPV (AAO-HNS, international)
⚠ 3 — Safety Netting
🔴 Neurological red flags
"Headache, diplopia, slurred speech, or limb weakness + vertigo → 999 immediately."
💊 Prochlorperazine limit
Stop after 3 days — longer suppresses vestibular compensation and delays recovery.
🚗 DVLA
No driving while liable to sudden disabling vertigo; notify DVLA if attacks are sudden and disabling. Document this advice.
🎓
SCA Quick Reference
SCA Consultation Blueprint
▼expand
🕐 12-Minute Flow
0–2 min
Open + Type of Dizziness
"That sounds really frightening. Can you describe what the dizziness is like — is the room spinning, or is it more of a lightheaded feeling?"
TasksRelating to Others
✗ Not confirming rotational vertigo first · ✗ No empathy for acute distress
2–4 min
Neurological Red Flag Screen
"Any headache when this started? Double vision? Slurred speech? Weakness or numbness anywhere? How long does each bout last?"
Tasks
✗ Performing positional test before this screen · ✗ Missing episode duration question
4–7 min
ICE + HINTS Exam
"Are you worried this might be a stroke? I'm going to do a specific eye test now — this is actually more sensitive for brain problems than an MRI in the first 48 hours."
TasksRelating to Others
✗ Stroke concern not named · ✗ HINTS not performed · ✗ Dix-Hallpike before HINTS
7–10 min
Dix-Hallpike + Epley
"The eye test is reassuring — it doesn't suggest a brain problem. Now I'm going to do the specific position test for the crystal type, and if it's positive, I can treat it straight away."
TasksRelating to Others
✗ No Epley offered after positive Dix-Hallpike · ✗ Diagnosis without Dix-Hallpike
10–12 min
Safety-Net + DVLA + Close
"The prochlorperazine is for 3 days only — stopping it after that actually helps you recover faster. No driving until the vertigo is gone or controlled. If you get a headache or weakness with the dizziness — 999."
TasksRelating to OthersGlobal Skills
✗ Long-term prochlorperazine · ✗ No DVLA · ✗ No neurological red flag safety-net · ✗ No closing question
🚫 8 Danger Zones
✗
Dix-Hallpike before neurological screen or HINTS
→ Neurological red flags → HINTS → Dix-Hallpike. This sequence is non-negotiable.
✗
Prescribing prochlorperazine without the 3-day maximum
→ "This is for 3 days only — longer than that actually slows your recovery by interfering with the brain's ability to adapt"
✗
Not naming the stroke concern before examining
→ "Are you worried this might be a stroke? That's a very understandable thing to be concerned about with these symptoms."
✗
Not offering the Epley manoeuvre in clinic for confirmed BPPV
→ "If the test is positive, I can treat it right here today — there's a specific head movement sequence that works for most people immediately"
✗
Not giving DVLA advice
→ "You should not drive until the vertigo is gone or well controlled — and that advice needs to be documented in your notes today"
✗
Diagnosing BPPV without Dix-Hallpike test
→ The diagnosis of BPPV requires a positive Dix-Hallpike. "I think this might be BPPV — let me do the specific test to confirm it"
✗
Not giving a neurological red flag safety-net
→ "If the vertigo returns and you have any headache, double vision, or weakness — 999 immediately"
✗
Arranging outpatient imaging (or relying on a normal CT) when central features are present
→ Immediate stroke pathway referral — imaging per local protocol; an early normal CT does not exclude posterior circulation stroke
Reviewed: July 2026 · citations verified against the primary sources listed