Dermatology & Allergy · Full case

Atopic Eczema

NICE CKS / CKSEmollientsSteroid Ladder
EC
Atopic Eczema · Clinical Reasoning Framework v2
GP & SCA · NICE CKS / CKS · Emollients · Steroid Ladder · FTU · Eczema Herpeticum · Dupilumab · Occupational Triggers · Steroid Phobia
Emollients: 500g/week (body)Minimum prescribed quantity for body eczema. 250g/week for face and hands. Emollients are the cornerstone of all eczema management at every step. Must be prescribed, not bought OTC — the quantities required make OTC purchase impractical and expensive. Liberal use (minimum 4×/day); apply in direction of hair growth; use as soap substitute. The most common cause of treatment failure in eczema is insufficient emollient use — not insufficient topical corticosteroid.
FTU: 1 unit = 2 adult handsOne fingertip unit (FTU) = amount of cream squeezed from a standard 5mm nozzle tube from the tip to the first crease of the index finger = approximately 0.5g. One FTU covers the skin area of both adult hands (palm and fingers). Fingertip units for common areas: face and neck = 2.5 FTU; arm = 3 FTU; leg = 6 FTU; trunk (front) = 7 FTU; trunk (back) = 7 FTU. Most TCS failures are due to under-application — patients typically apply too little. Demonstrating the FTU at the consultation is a core prescribing competency for eczema.
Eczema herpeticum = 999Eczema herpeticum (Kaposi varicelliform eruption): viral superinfection of eczema with herpes simplex virus. Presents as rapidly spreading monomorphic punched-out vesicles and erosions on a background of eczema; patient is systemically unwell; fever; malaise; lymphadenopathy. A dermatological emergency — can be life-threatening if untreated. Action: 999 or same-day hospital attendance; IV aciclovir. Must be distinguished from bacterial superinfection (Staphylococcus aureus; honey-coloured crusts; weeping) which is urgent but not an emergency and is treated with topical or oral antibiotics.
Steroid potency ladder: mild → very potentThe four potency classes of topical corticosteroids (TCS): (1) MILD: hydrocortisone 0.5–1%; (2) MODERATE: clobetasone butyrate 0.05% (Eumovate); fluocinolone acetonide 0.00625%; (3) POTENT: betamethasone valerate 0.1% (Betnovate); mometasone furoate 0.1%; fluticasone propionate 0.05% (Cutivate); (4) VERY POTENT: clobetasol propionate 0.05% (Dermovate) — specialist use only. Site-specific rules: face; eyelids; genitalia; flexures — mild TCS only (hydrocortisone 1%); or TCI (tacrolimus; pimecrolimus). No potent TCS on face. Duration: face/flexures — no more than 7 days.
S. aureus superinfection: treat before stepping up TCSStaphylococcus aureus colonises virtually all atopic eczema skin and is a major trigger of flares. Signs of active infection (not just colonisation): honey-coloured crusting; weeping; pustules; rapid worsening despite appropriate TCS. Management: (1) Topical: fusidic acid cream (Fucidin) 2% BD for 7 days; or combination fusidic acid + TCS (Fucibet — contains clobetasone; FuciBet Lipid Cream — contains betamethasone); limit topical fusidic acid courses to reduce resistance. (2) Oral (moderate-severe infection): flucloxacillin 500mg QDS for 7 days; penicillin allergy: cefalexin 500mg TDS or clarithromycin. NEVER step up TCS potency into actively infected skin — treat infection first.
TCI (tacrolimus / pimecrolimus): face and flexuresTopical calcineurin inhibitors (TCIs) are steroid-sparing agents that inhibit T-cell activation and the release of inflammatory cytokines. Key indications: (1) Face; eyelids; neck — where potent TCS are inappropriate; (2) Flexures; skin folds; genitalia; (3) Patients with TCS-induced skin atrophy; (4) Steroid phobia (patient unwilling to use TCS on face). Tacrolimus (Protopic): 0.03% (children 2–15 years); 0.1% (adults). Pimecrolimus (Elidel): 1% cream; both 2 years and older. Side effect: initial burning/stinging on application (usually settles after 1 week). Previously had a black-box warning for theoretical malignancy risk — this has been withdrawn; long-term safety data is reassuring.
Dupilumab (NICE TA534): moderate-severe adultsDupilumab (Dupixent) is a fully human monoclonal antibody that blocks IL-4 and IL-13 (Th2 cytokines central to atopic eczema pathogenesis). NICE TA534 (2022): approved for adults with moderate-to-severe atopic eczema who have had an inadequate response to ciclosporin, or are intolerant of or have contraindications to ciclosporin. EASI score ≥16 or IGA ≥3. Self-administered subcutaneously (300mg every 2 weeks after loading dose; 600mg at week 0). Also approved for adolescents 12–17 (NICE TA885) and children 6–11 (NICE TA2023 update). Other biologics: tralokinumab (NICE TA863; IL-13 inhibitor). JAK inhibitors: abrocitinib; upadacitinib (NICE TA878/TA879; for adults; specialist initiation only).
POEM: 0–28 patient-reported outcomeThe Patient-Oriented Eczema Measure (POEM) is a validated 7-item patient-reported outcome tool for monitoring eczema severity in primary care. Seven questions (each scored 0–4): itch; sleep disturbance; bleeding; weeping/oozing; cracking; flaking; dryness. Total 0–28: 0–2 = clear/almost clear; 3–7 = mild; 8–16 = moderate; 17–24 = severe; 25–28 = very severe. Use at every review to track treatment response. A POEM reduction of ≥3 points = clinically meaningful improvement. The POEM is recommended by NICE CKS as the primary outcome measure for eczema in primary care consultations.
📋 Clinical Stem — Atopic Eczema
Mrs. Amara Mensah, 34, healthcare assistant, with a 4-week worsening of known atopic eczema on arms; neck; and hands, exacerbated by occupational hand washing, steroid phobia causing under-treatment, and inadequate emollient use
Mrs. Amara Mensah, 34, healthcare assistant in a care home, presents with a 4-week deterioration of her known atopic eczema. Her arms, neck, and hands are worst affected — the hands particularly, given her frequent handwashing at work. She has run out of her prescribed emollient and has been using a supermarket moisturiser for 3 weeks. She has some leftover hydrocortisone 1% cream that she is using on her face and body but she is applying it very sparingly because her mother told her steroid creams “damage the skin.” She is self-conscious and embarrassed about the appearance of her skin at work. She has not been sleeping well due to itching. POEM score 16 (moderate–severe). PHQ-9 7 (mild depression).
This stem tests the ability to: assess eczema severity and step up the TCS ladder appropriately; prescribe emollients in adequate quantities and teach correct application; address steroid phobia specifically with evidence-based counselling; identify occupational triggers (wet work; hand washing; care home); recognise signs of bacterial superinfection; demonstrate the FTU; and provide a written eczema action plan. The SCA challenge is the steroid phobia and the question about whether the TCS is safe.
Scenario A — Mrs. Mensah (occupational eczema + steroid phobia) Known eczema; 4-week flare; hands worst (wet work in care home); emollient run out; using hydrocortisone 1% too sparingly (steroid phobia). Management: adequate emollient (500g/week); step up to moderate–potent TCS on body/limbs; hydrocortisone 1% on face only; address steroid phobia with FTU counselling; written eczema action plan; occupational health referral; hand eczema barrier cream protocol.
Scenario B — Bacterial superinfection Worsening eczema; golden-coloured crusting; weeping; increasing erythema; not responding to TCS. S. aureus superinfection. Management: do NOT step up TCS potency into infected skin; treat infection first: topical fusidic acid 2% (Fucidin) BD ×7 days; or oral flucloxacillin 500mg QDS ×7 days if more widespread. Review in 1–2 weeks; step up TCS once infection cleared. Swab if recurrent.
Scenario C — Eczema herpeticum Rapid spread; punched-out monomorphic vesicular erosions; fever; malaise; unwell. Herpes simplex superinfection. Medical emergency. Action: 999 or same-day A&E; IV aciclovir; ophthalmology if periorbital involvement. Do NOT treat as bacterial infection and send home. Distinguishing feature: punched-out erosions vs golden crusts; fever vs afebrile; unwell vs well.
Scenario D — Child with moderate-severe eczema and food allergy concern Child <12 with moderate-severe eczema not responding to appropriate topical treatment. NICE CKS: consider food allergy screen (egg; milk; wheat; soy; peanuts) in children with moderate-severe eczema and inadequate response to topical treatment. Refer to allergy clinic. Not all children with eczema need allergy testing — only moderate-severe not responding. IgE-mediated allergy tests: specific IgE or skin prick test. Management: NICE stepwise topical treatment first; allergy referral if fails.
Scenario E — Severe eczema — systemic treatment decision Adult with severe eczema; POEM 20; EASI 20; failed multiple TCS; sleep disrupted; not responding to topical treatment. Dermatology referral for phototherapy (narrowband UVB) or systemic immunosuppressant (ciclosporin first-line per NICE CKS). If ciclosporin fails or contraindicated: dupilumab (NICE TA534). GP role: refer early; initiate and supervise ciclosporin monitoring (BP; renal function; LFTs; FBC at baseline and then every 2 weeks for 3 months; then monthly). Do NOT initiate systemic treatment without dermatology input.
Key variables to adapt for Age (children: NICE CKS specific; face is common in infants; flexures in older children); severity (POEM score; EASI; IGA — assess at every visit); distribution (face/eyelids: only mild TCS or TCI; body/limbs: stepped approach); infection status (never step up TCS into infected skin); steroid phobia (address at every consultation; FTU; specific counselling); occupational triggers (wet work; care home; healthcare worker; hairdresser; chef); atopic comorbidities (asthma; allergic rhinitis; food allergy); psychological impact (PHQ-9; DLQI; sleep disruption)
Steps:
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Step 1
History — Onset · Severity · Triggers · Steroid Use · POEM · Occupational Factors · ICE
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The history in eczema establishes severity (POEM score); current treatment and its adequacy; precipitating triggers; and the patient’s illness beliefs (steroid phobia is the leading cause of under-treatment). Mrs. Mensah’s presentation contains three correctable failures: inadequate emollient; under-application of TCS due to steroid phobia; and an unidentified occupational trigger.
🎓 SCA opener — acknowledge the embarrassment and self-consciousness before the clinical assessment
"It sounds like the last few weeks have been really difficult — the itch at night affecting your sleep, and feeling self-conscious at work. Before I look at your skin, tell me in your own words what has been happening and what is worrying you most."
Mrs. Mensah is embarrassed. A GP who begins immediately with examining the skin and asking about creams misses the emotional dimension — the embarrassment at work; the fear about steroids she has carried since her mother’s warning. Acknowledging both first builds the therapeutic alliance needed to address the steroid phobia effectively.
1A — Open question, then structured eczema history
QuestionWhy it mattersChanges what?
🏲 OPEN QUESTION"Tell me what has been happening with your skin — what it has been like and what has been worrying you most about it."The open question reveals: the subjective severity (itch quality; sleep impact); the emotional dimension (embarrassment at work; self-consciousness); the patient’s illness model (steroid fear from mother’s experience); the treatment history (inadequate emollient; under-applied TCS); and any specific concerns (“is it going to scar?”). Mrs. Mensah will likely lead with the itch and the embarrassment — the steroid phobia may only emerge when she is asked about what she has tried.SCA: Relating to Others for emotional acknowledgement; Global Skills for allowing patient to leadSeverity; triggers; treatment adequacy; steroid phobia; emotional impact; occupational context
POEM score — 7-item validated tool"Over the last week: has your skin been itchy? Has it disturbed your sleep? Has it bled; wept or oozed; cracked; flaked; or been dry?"The POEM (Patient-Oriented Eczema Measure) is the NICE CKS-recommended primary outcome measure for eczema in primary care. Seven items (each 0–4 based on frequency in last 7 days); total 0–28. Mrs. Mensah: score 16 (moderate–severe). Administration at baseline establishes the severity; re-administration at 4–6-week review tracks treatment response. A clinically meaningful response = ≥3-point reduction. The POEM documents severity more robustly than clinical impression alone and guides the TCS step decision.POEM 0–7: mild → Step 1–2. POEM 8–16: moderate → Steps 2–3. POEM 17–24: severe → Steps 3–4; dermatology. POEM ≥25: very severe → urgent dermatology
Current treatment and actual use"What creams or ointments are you using? How much are you using? How often? How are you applying them?"Mrs. Mensah has run out of emollient and switched to supermarket moisturiser (insufficient — OTC moisturisers do not contain the occlusive barrier agents required). She is using hydrocortisone 1% on her face and body but very sparingly (steroid phobia). Inadequate emollient + under-application of TCS = predictable treatment failure. The history of treatment use reveals whether the failure is pharmacological (wrong drug) or therapeutic (right drug; wrong dose; wrong technique).Inadequate emollient: prescribe 500g/week; educate on quantity. Under-applied TCS: FTU counselling. Steroid phobia: address directly
Trigger identification — occupational"What is your job? Do you wash your hands a lot at work? What soaps or products do you use? Do you wear gloves? Are there any substances you handle?"Mrs. Mensah is a healthcare assistant in a care home: frequent hand washing; detergent exposure; glove use (latex and non-latex; both can trigger contact dermatitis). Wet work (“hands wet for >2 hours/day or ≥20 hand washing episodes/day”: definition of high-risk wet work) is a powerful driver of hand eczema. This occupational history directly changes the management plan: occupational health referral; prescribed barrier cream protocol; glove advice (cotton glove liners; non-latex gloves); hand-specific emollient (Diprobase; Doublebase Dayleve gel for during the day; Epaderm ointment at night). Failure to identify the occupational trigger means the inflammation will continue despite optimal topical management.Occupational trigger: occupational health referral; wet work protocol; prescribed barrier cream; glove advice; sick note if needed
Trigger identification — environmental"Does the rash come and go? Does anything make it worse — pets; pollen season; dust; certain fabrics; heat; stress?"Environmental triggers are highly individual. House dust mite is the most common aeroallergen trigger; pet dander (especially cats; dogs) is another. Heat and sweating worsen itch. Wool and synthetic fabrics cause irritation. Stress is a potent and often underestimated trigger — the hypothalamic-pituitary-adrenal axis and skin immune regulation are tightly linked. Identifying triggers allows targeted avoidance advice: allergen-impermeable mattress; pillow covers; hot washing of bedding; pet dander reduction strategies; stress management.HDM: allergen-impermeable covers; hot washing. Stress: NHS Talking Therapies; stress management; social prescribing. Pets: dander reduction; allergen testing if unclear
Signs of superinfection"Has the eczema started weeping; crusting; or producing pus recently? Has it changed character — spreading much more quickly than usual? Any fever?"Two types of superinfection must be distinguished: (1) Bacterial (S. aureus): weeping; honey-coloured crusting; pustules; localised; no fever; URGENT but not emergency. (2) Viral (eczema herpeticum): rapidly spreading punched-out vesicular erosions; fever; unwell; systemic illness; EMERGENCY. Getting this distinction wrong — treating eczema herpeticum as bacterial and sending home with antibiotics — can be fatal. History clues: fever and systemic illness = viral; no fever; localised = bacterial; rapid spread of monomorphic lesions = viral.Eczema herpeticum features: 999 / same-day A&E. Bacterial superinfection features: urgent topical or oral antibiotic
1B — Red flags
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Red Flags — must not miss; must act

Red flagWhy dangerousAction
Eczema herpeticum — rapidly spreading punched-out erosions; fever; unwellHerpes simplex superinfection can be life-threatening. Monomorphic punched-out vesicles and erosions spreading rapidly; fever; malaise; periorbital involvement. IV aciclovir required. Cannot be treated at home. Delay → sepsis; meningitis.999 or same-day A&E — IV aciclovir
Signs of anaphylaxis — associated with food allergy triggerIn children with severe eczema and known food allergy, a trigger food exposure can cause anaphylaxis. If anaphylaxis present: 999; IM adrenaline 0.5mg (1:1000) IM anterolateral thigh; ambulance. Prescribe adrenaline auto-injector (EpiPen; Emerade) for at-risk patients.999 — IM adrenaline; ambulance
Widespread superinfection with sepsis featuresSevere bacterial superinfection (S. aureus; streptococcus) can cause sepsis — particularly in children; immunocompromised. Fever; tachycardia; raised WCC; CRP. Urgent hospital admission; IV antibiotics.Same-day A&E — IV antibiotics; sepsis review
Erythroderma — ›90% body surface area involvementErythrodermic eczema: temperature dysregulation; fluid loss; cardiovascular instability; secondary infection. Medical emergency requiring hospital admission and intensive management.Same-day hospital — urgent dermatology
Psychological crisis — active suicidal ideation related to eczemaSevere eczema causes significant psychological distress. PHQ-9 Q9: if suicidal ideation present in the context of severe eczema — urgent mental health review. The DLQI (Dermatology Life Quality Index) in severe eczema can be comparable to severe psoriasis; chronic pain; cancer.PHQ-9 Q9 ≥1: urgent mental health; same-day if crisis
🛡️

Safeguarding — Eczema and Vulnerability

Severe eczema causes significant psychological distress; sleep deprivation; social withdrawal; and reduced quality of life — all of which create vulnerability. In children, eczema has been associated with behavioural problems and poor educational outcomes related to sleep disruption. In adults: workplace discrimination; relationship difficulties; and depression (DLQI impairment comparable to severe systemic disease). Mrs. Mensah’s PHQ-9 7 reflects this burden.
💕 Psychological distress and self-harm
  • PHQ-9 Q9: ask specifically about suicidal ideation in patients with severe eczema
  • Sleep deprivation from chronic itch is an independent risk factor for depression
  • Itch-scratch cycle can have compulsive elements; note if scratching is causing self-harm
  • Refer to NHS Talking Therapies or dermatology psychology service if significant psychological impact
🏠 Child eczema and parenting stress
  • Eczema in young children causes significant family stress; parental sleep deprivation; relationship difficulties
  • If child eczema is severely undertreated: consider whether parent has capacity to manage treatment; referral to health visitor
  • If child is presenting repeatedly with poorly controlled eczema: explore family context; barriers to treatment
💼 Occupational vulnerability
  • Healthcare workers with hand eczema: unable to work if open wounds; infection risk to patients
  • Employer discrimination: Mrs. Mensah may be self-conscious about visible eczema at work
  • Occupational health referral: right to reasonable adjustments; glove protocol; alternative soap-free hand cleaners
  • Sick note: may be needed if eczema prevents safe working
📋 Medication adherence and safety
  • Steroid phobia leading to under-treatment: directly linked to significant harm (uncontrolled eczema; infection; psychological impact)
  • Topical TCS misuse (overuse): real risk of skin atrophy; telangiectasia; striae — especially face and flexures
  • Ensure patient has written eczema action plan — this reduces unnecessary steroid use (allows step-down) AND prevents under-use
Actions: PHQ-9 Q9 checked (score 7; Q9 = 0 in Mrs. Mensah); occupational health referral for hand eczema and wet work; written eczema action plan prevents both under- and over-treatment; NHS Talking Therapies referral if depression significant.
1C — PMH · Drug history · Social history
🥐 PMH · Atopic history
FactorWhy it mattersImpact
Atopic march: eczema; allergic rhinitis; asthmaAsk about all three atopic conditions. 30–40% of eczema patients have asthma; 30–50% have allergic rhinitis. Poorly controlled allergic rhinitis worsens eczema via systemic Th2 inflammation. Treating rhinitis (nasal steroid; antihistamine) can improve eczema. Ask about seasonal worsening (pollen; HDM).Comorbid rhinitis: nasal steroid (mometasone; fluticasone) may reduce eczema flares. Asthma: avoid systemic TCS for eczema if ICS use high (cumulative steroid exposure)
Previous TCS use and responseHas she been on potent TCS before? What was the response? History of TCS-induced atrophy; striae; telangiectasia? Has she ever had wet wrapping; tacrolimus; dupilumab? Prior response to treatment helps guide current management choice and dose escalation decisions.Previous TCS atrophy on face: TCI (tacrolimus; pimecrolimus) preferred. Previous good response to potent TCS: confidence in step-up. No prior response to any TCS: reconsider diagnosis; consider infection
Skin barrier defect (filaggrin mutation)Filaggrin (FLG) gene mutations underlie skin barrier defect in atopic eczema in approximately 30% of patients. Not routinely tested in primary care but conceptually important: the skin barrier defect means transepidermal water loss (TEWL) is increased; emollients specifically address this by providing exogenous barrier function. Patients with FLG mutations tend to have earlier onset; more severe disease; higher risk of food sensitisation.Skin barrier concept: explains why emollients must be used regularly — not just when the skin is dry
💼 Occupational · Social · Drug history
FactorWhy it mattersImpact
Healthcare worker; care home; wet workHand washing >20 times/day = high-risk wet work. Detergent and soap exposure denudes the lipid layer; causes direct irritant contact dermatitis compounding eczema. Care home work involves body fluids; cleaning products; latex gloves — all potential sensitisers. Occupational health has legal mandate to assess and recommend workplace adjustments.Occupational health referral; alcohol gel (better tolerated than soap for frequent use); non-latex gloves; cotton glove liners; barrier cream during shift; Diprobase gel for daytime use
Sleep disruption from itchChronic itch worsens at night (physiological itch amplification at night; cortisol levels low; skin temperature high). Sleep deprivation is both a consequence and a worsening factor for eczema — poor sleep reduces immune regulation. POEM sleep question (Q2) is a specific marker of disease severity. Sedating antihistamines (chlorphenamine; hydroxyzine) have limited evidence for eczema itch but may help sleep short-term.Sedating antihistamine (chlorphenamine 4mg at night) may help sleep short-term during flare; not for long-term use; behavioural sleep hygiene
Current medications: hydrocortisone 1% (sparingly)Under-application of TCS due to steroid phobia is the most correctable cause of treatment failure in eczema. Hydrocortisone 1% is an appropriate starting preparation but it requires adequate quantity and frequency to be effective. Must be addressed: what does “sparingly” mean? Is she using enough per FTU? Does she understand the risk of under-treatment vs over-treatment?Steroid phobia counselling at this consultation; FTU demonstration; written eczema action plan with clear rules for when to step up and step down
NSAID or aspirin useNSAIDs can worsen eczema in some patients (mechanism: prostaglandin modulation; mast cell effects). Aspirin-exacerbated eczema is recognised. Ask about regular NSAID use if unexplained worsening.NSAID-worsened eczema: switch to paracetamol if possible; review analgesic choice
1D — ICE
💡 Ideas
"What do you think has caused this flare? Have you any ideas about what has made it worse recently?"
Mrs. Mensah may link the flare to running out of emollient (correct); her increased hand washing at work (correct); or stress. She may not have identified the steroid phobia as a contributing factor — she sees herself as “using the cream” but not realise she is using far too little to be effective. Understanding her illness model allows the GP to build on what is correct and specifically address what is incorrect.
😟 Concerns
"What worries you most? Are you concerned about the steroid cream — about whether it is safe to use it?"
The steroid phobia is the central concern — and it must be raised directly because Mrs. Mensah may not raise it spontaneously. Her mother’s belief that steroid creams “damage the skin” is a real and understandable concern. The response must be specific: acknowledge that steroid skin thinning is a real risk with prolonged overuse; explain that short courses at the right potency for the right duration are safe; explain that under-treatment causes its own harm (itch; sleep disruption; infection; scarring). The written eczema action plan directly addresses this by giving clear, time-limited instructions.
🎯 Expectations
"What were you hoping today’s appointment would give you? Are you hoping for something that will cure the eczema?"
Mrs. Mensah may hope for a “cure.” Atopic eczema is a chronic relapsing condition — management, not cure, is the realistic goal. The consultation should set expectations: 30–40% of childhood eczema improves significantly by adulthood; but many adults have persistent disease requiring ongoing management. The goal is to empower Mrs. Mensah with the knowledge and tools to manage her own flares effectively — the written action plan, the FTU, the emollient quantity — so she is not dependent on GP appointments for every minor flare.
1E — Psychosocial context
🧑️ Eczema is not just a skin condition — it disrupts sleep; self-image; work; relationships; and mental health

Mrs. Mensah’s eczema has a direct impact on every domain of her life: embarrassment and self-consciousness at work; sleep deprivation from chronic itch; depressed mood (PHQ-9 7); occupational difficulties (hand eczema in a wet-work setting); and the weight of inherited beliefs about treatment safety. Each of these dimensions affects both treatment adherence and clinical outcome.

👔 Steroid Phobia

The most clinically important psychosocial factor in this consultation. Inherited from her mother’s experience. Leads to systematic under-application of TCS; treatment failure; and escalating eczema severity. Must be addressed with specific evidence, not dismissal.

"I understand the worry about the steroid cream — it is a very common concern. Let me explain what is safe and what is not safe, so you can use it with confidence."

Management change: full steroid counselling with FTU; written action plan; distinction between appropriate use and harmful overuse

👔 Embarrassment and Self-Image

Mrs. Mensah is self-conscious about her appearance at work. Visible eczema on the arms; neck; and hands is difficult to conceal in a care home setting. The embarrassment is not trivial — it affects her professional confidence and social functioning.

"How has the skin been affecting you at work? Are you feeling self-conscious about it? I want to understand the full impact."

Management change: address quickly and effectively; consider DLQI; social prescribing for psychological support if needed

😴 Sleep Disruption

Itch worsens at night — sleep deprivation compounds depression; reduces immune regulation; worsens itch sensitivity. Mrs. Mensah’s POEM sleep score likely contributes significantly to her total score of 16.

"Has the itch been waking you at night? Poor sleep from eczema is one of the most underappreciated aspects — it affects your mood, your immunity, and how well your skin heals."

Management change: emollient at bedtime; cotton loose clothing; cool bedroom; chlorphenamine 4mg at night during severe flares

💼 Occupational Impact

Healthcare worker with wet work: hand eczema directly affects her ability to work safely. Open skin = infection risk to patients; glove intolerance; painful cracking preventing dexterous care tasks.

"Your work involves a lot of hand washing — that is likely a significant factor in your hand eczema. Let’s make sure you get the right support at work — including a referral to occupational health."

Management change: occupational health referral; prescribed barrier cream protocol; alternative to soap (alcohol gel); sick note if unable to work safely

🎓 SCA Checkpoint — Step 1TasksRelating to OthersGlobal Skills
Key phrases that score
"It sounds like the last few weeks have been really difficult — the itch at night, not sleeping, and feeling self-conscious at work. Before I look at your skin, tell me what has been happening and what is worrying you most."
"I want to ask about the steroid cream — are you worried about using it? I ask because it is one of the most common concerns I hear, and there are some important things I want to share about when it is safe and when it is not."
"Your job involves a lot of hand washing — that is almost certainly playing a role in your hand eczema. I am going to refer you to occupational health to make sure you have the right support at work."
Deductions
  • Not addressing steroid phobia even when it emerges — leaving Mrs. Mensah with the same steroid fear means treatment failure will continue
  • Not identifying the occupational trigger — treating eczema without addressing the cause means the treatment will need to be continued indefinitely
  • Not administering POEM score — severity is under-documented
🔴 Red
No emotional opener; steroid phobia not identified; occupational trigger not identified; no POEM; no FTU; emollient not reviewed; eczema herpeticum features not screened
🟠 Amber
Embarrassment acknowledged; POEM scored; steroid phobia identified but not addressed specifically; TCS stepped up; emollient prescribed; occupational trigger not identified; FTU not demonstrated
🟩 Green
Emotional opener; POEM 16 scored; steroid phobia raised and addressed with FTU and evidence; occupational trigger identified (wet work; care home); emollient 500g/week prescribed; TCS stepped appropriately; eczema herpeticum safety-net; written action plan; occupational health referral; PHQ-9 7 addressed; closing question
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Step 2
Triage — Eczema Herpeticum (Emergency) · Severe Superinfection · GP-Managed Eczema
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Most eczema is GP-managed. The triage question is: emergency (eczema herpeticum; erythroderma); urgent (significant bacterial superinfection); or routine (uncomplicated flare)? Mrs. Mensah: no emergency features; no systemic illness; occupational flare — routine GP management.
🔴 Emergency

999 / Same-Day Hospital

Immediate
  • Eczema herpeticumPunched-out vesicular erosions; fever; rapidly spreading; 999; IV aciclovir
  • Erythroderma (>90% BSA)Temperature dysregulation; cardiovascular instability; same-day hospital
  • Sepsis from superinfectionFever; tachycardia; systemically unwell; 999 / A&E
🟠 Urgent

Same-Day to 2 Weeks

Days
  • Significant bacterial superinfectionWeeping; golden crusts; extending rapidly; oral flucloxacillin; same-day review
  • Severe eczema POEM ≥17Unable to function; urgent dermatology; consider short-course prednisolone (not NICE first-line) with specialist input
🟩 GP-managed — Mrs. Mensah

Primary Care

NICE CKS stepwise
  • Moderate-severe flare (POEM 16) — Mrs. MensahStep up TCS to potent; emollient 500g/week; FTU; occupational health; 4–6 week review
  • Mild-moderate uncomplicated flareEmollient; mild TCS; trigger avoidance; POEM review
  • Maintenance phaseRegular emollient; proactive TCS ×2/week on problem areas
🎓 SCA Checkpoint — Step 2Tasks
Emergency exclusion
"Before we look at the treatment, I want to check — have you had any fever with this? Has the rash been spreading very rapidly — spreading in a way that feels different from your usual eczema? Has any area started producing lots of tiny blister-like spots? Those would be warning signs I would want to know about immediately."
Deductions
  • Not screening for eczema herpeticum features — sending a patient with eczema herpeticum home with topical antibiotics is a serious safety error
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Step 3
Examination — Distribution · Severity · Infection Signs · Lichenification · Eczema Herpeticum
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Examine all affected areas to assess distribution; severity; infection; lichenification; and TCS side effects. The most critical examination finding to identify — and not miss — is eczema herpeticum. The examination also establishes an objective baseline (IGA score; distribution) to compare at review.
ExaminationWhat it showsManagement impactChanges?
Distribution and site — full skin surveyInspect face; eyelids; neck; arms; flexures; hands; feet; bodyDistribution informs TCS choice: face; eyelids; flexures — mild TCS only (hydrocortisone 1%) or TCI; potent TCS on body and limbs only. Arms; neck; hands: appropriate for moderate–potent TCS (betamethasone 0.1%; mometasone 0.1%). Hands specifically: contact dermatitis pattern (dorsa; fingers; web spaces) vs eczema pattern.Face only mild TCS; body/limbs moderate-potent; hands: barrier cream; occupational protocolYES — site determines TCS potency choice
Signs of bacterial superinfectionYellow; honey-coloured crusting; weeping; pustules; surrounding erythemaS. aureus is the most common superinfecting organism. Signs: honey-coloured or yellow-brown crusting; weeping exudate; pustules; increased erythema and warmth. Mrs. Mensah: examine hands and arms carefully. If superinfection present: treat infection first (fusidic acid topical or oral flucloxacillin); do not step up TCS potency into infected skin — this suppresses the immune response needed to clear the infection.Superinfection: treat with fusidic acid or flucloxacillin first; review in 1 week; THEN step up TCS. No superinfection: step up TCS nowYES — changes whether TCS is stepped up now or deferred
Eczema herpeticum features — urgent identificationMonomorphic punched-out erosions; vesicles; rapid spread; fever; systemically unwellThe most important examination in eczema practice. Eczema herpeticum: multiple monomorphic punched-out vesicles and erosions (all same size; same stage — unlike bacterial; which is more varied). May be periorbital; widespread. Patient is systemically unwell. If eczema herpeticum features: 999 / same-day A&E immediately — do NOT prescribe topical treatment and send home.Eczema herpeticum features: 999 immediately; IV aciclovir; ophthalmology if periorbital. Normal eczema lesions: routine stepwise managementYES — emergency action if features present
Lichenification and excoriationThickened; leathery skin; linear scratch marks; hyperpigmentationLichenification (chronic scratching → epidermal thickening) indicates: (1) long-standing disease; (2) pruritus not controlled with current treatment; (3) habitual scratching behaviour. Lichenification responds more slowly to TCS and may require occlusive treatment (Ichthyol paste; wet wrapping) or higher potency TCS. Hyperpigmentation or hypopigmentation from post-inflammatory change: reassure; usually resolves over months.Lichenification: may need potent TCS + occlusion; bandaging; dermatology if refractory. Post-inflammatory pigment change: reassure; usually self-resolvesYES — lichenification may require bandaging referral or potent TCS with occlusion
TCS side effects — skin atrophy; striae; telangiectasiaExamine previous TCS application areas specifically — particularly faceTCS side effects are site- and duration-dependent. Signs: skin thinning (can see through to subcutaneous vessels); telangiectasia; striae (stretch marks from steroid effect on collagen); perioral dermatitis (facial TCS misuse). If TCS side effects present on face: switch to TCI (tacrolimus; pimecrolimus); never prescribe potent TCS for face. Absence of TCS side effects after years of appropriate use is important reassurance for patients with steroid phobia.TCS atrophy on face: switch to TCI (tacrolimus 0.1% or pimecrolimus 1%); never potent TCS. No side effects: reassure; continue with appropriate TCS potency for siteYES — TCS atrophy changes prescription choice
🎓 SCA Checkpoint — Step 3Tasks
Examination communication
"I am going to look at all of the affected areas — arms; neck; hands — and I want to check specifically for any signs of infection in the skin. I am also going to look at the areas where you have been using the cream to check for any effects of long-term use."
Deductions
  • Not examining the hands specifically in a healthcare worker with hand eczema — the distribution and characteristics of hand eczema directly guide the occupational health referral and the barrier cream protocol
4
Step 4
Investigations — Skin Swab · Allergy Testing · Patch Testing · POEM Tracking
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Most eczema does not require investigation — it is a clinical diagnosis. Investigations are triggered by: suspected infection; suspected contact allergy; failure to respond to appropriate topical treatment; or suspected food allergy (children).
InvestigationClinical questionResult and action
Skin swab (MC&S) — infected or recurrently infected eczemaSwab weeping or crusted area; also swab nose (MRSA screening if healthcare worker)Identifies organism and antibiotic sensitivities. S. aureus is most common; occasionally beta-haemolytic streptococcus. Healthcare worker: also check for MRSA colonisation (nasal swab — relevant for patient safety as well as her own treatment). MRSA-colonised eczema: specialist dermatology advice; MRSA decolonisation protocol; infection control implications in care home setting.S. aureus: topical fusidic acid (Fucidin) or oral flucloxacillin. MRSA: specialist advice; rifampicin + fusidic acid combination; infection control notification. Beta-haemolytic strep: phenoxymethylpenicillin or cefalexin.
Patch testing — suspected contact dermatitisIf eczema distribution fits contact pattern; or hand eczema in occupational settingPatch testing (NOT skin prick test — which is for IgE-mediated allergy) identifies type IV delayed hypersensitivity reactions to specific allergens. Relevant for Mrs. Mensah: suspected contact allergy to rubber chemicals in gloves; preservatives in creams; fragrances; workplace products. Patch testing is performed by dermatology; requires reading at 48 and 96 hours. Positive result: identify the allergen; avoid specifically; may completely resolve contact dermatitis component of eczema.Positive patch test: identify allergen; product substitution at work; occupational health notification. Negative patch test: atopic eczema confirmed; no contact component
Total IgE; specific IgE (RAST) or skin prick test — if food allergy suspectedChildren with moderate-severe eczema not responding to topical treatmentNICE CKS: consider allergy testing in children with moderate-severe eczema that has not responded adequately to appropriate topical treatment (emollient + step 2 TCS for 4–6 weeks). Allergy clinic referral for skin prick testing or specific IgE. Foods to test: egg; milk; wheat; soy; peanuts. Positive allergy test + eczema: dietary elimination trial under dietitian supervision. Not recommended for adults without clear history of food-triggered reactions; most adult eczema is not food-driven.Positive IgE to food in child with moderate-severe eczema: supervised dietary elimination; allergy clinic. Negative: continue topical management; other triggers
POEM score (0–28) at every reviewComplete 7-item questionnaire; document baseline and at 4–6 week reviewPOEM is the NICE CKS-recommended primary outcome measure for eczema in primary care. Documents severity; tracks treatment response. A reduction of ≥3 points = clinically meaningful improvement. Mrs. Mensah: POEM 16 at baseline. Target at 4–6 week review: POEM <8 (mild) or ≥3-point reduction. If POEM not improving after adequate treatment: consider infection; incorrect TCS potency; steroid phobia still limiting use; or dermatology referral for step 4 treatment.POEM ≥3-point reduction at review: treatment working; continue. POEM unchanged despite adequate treatment: review cause; consider patch testing; allergy screen; dermatology. POEM ≥17: urgent dermatology; systemic treatment discussion
🎓 SCA Checkpoint — Step 4Tasks
Investigations in eczema
"For most eczema, I do not need to do lots of tests — the diagnosis is clinical. However, because you are a healthcare worker and you have hand eczema, I am going to take a swab from the skin on your hands to check for bacterial infection — and I will also swab your nose, because occasionally people who work in healthcare can carry a resistant bacteria without knowing it, and it is important to check."
Deductions
  • Not swabbing a healthcare worker with weeping or crusted hand eczema — MRSA colonisation has infection control implications for the care home; this is a patient safety issue as well as a clinical one
5
Step 5
Diagnosis — Plain Language · Steroid Phobia Addressed · Eczema vs Contact Dermatitis · Severity Classification
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The diagnosis in eczema is clinical. The key communication challenge is addressing steroid phobia — which is the single most correctable cause of treatment failure — with specific, evidence-based reassurance while remaining honest about real TCS risks.
🗣️ Explaining atopic eczema and addressing steroid phobia in plain language

"Your eczema is a skin condition where the outer layer of your skin — the barrier — is not working as effectively as it should. This means moisture escapes too easily and irritants get in too easily. When the skin becomes inflamed, it releases chemicals that cause the itch. And scratching releases more chemicals; causes more inflammation; and the cycle continues. The emollient cream acts as a replacement barrier — it has to be applied regularly, not just when the skin is dry, because the barrier needs to be maintained constantly. The steroid cream tackles the inflammation directly. I want to address your concern about the steroid cream specifically. Skin thinning from steroids is a real risk — but it only happens with prolonged use of potent steroids on the wrong areas. The way we use steroids for eczema is different: short courses; stepping up during flares; stepping down when controlled; and using the mildest effective preparation. Using the cream correctly for a short time is safe. What is not safe is using it too little — because under-treated eczema causes its own harm: it becomes infected; it prevents sleep; it causes scarring from scratching; and it affects your mental health."

💬 Addressing the core concern — steroid phobia

"My mother said steroid creams damaged her skin. Is that going to happen to me?"
"Your mother’s experience — and her concern — makes complete sense. Skin thinning from steroids is real, but it happens with: (1) prolonged continuous use over many weeks; (2) on the face or skin folds with a potent steroid; (3) without taking breaks. The way we are going to use the cream is very different: I am going to give you a written plan that tells you exactly which cream to use on which area; for how many days; and when to stop. Using a medium-strength cream on your arm for 2 weeks is not going to thin your skin. Leaving the inflammation untreated for months — which is what has been happening — is more likely to cause skin changes from the eczema itself."

Mrs. Mensah’s Diagnosis
GP diagnosis
Atopic eczema (known since childhood); POEM 16 (moderate–severe flare); arms; neck; hands. Occupational trigger: wet work in care home. Inadequate emollient; under-applied TCS (steroid phobia). No eczema herpeticum features; no current superinfection (hands oozing but no honey crusting). IGA 3 (moderate-severe). Management: emollient 500g/week; step up to potent TCS body/limbs; hydrocortisone 1% face only; FTU counselling; written action plan; occupational health.
Differential — Consider
Dermatology if atypical

Contact dermatitis (irritant or allergic)

May coexist with atopic eczema. Irritant: wet work; soap; rubber chemicals. Allergic: type IV; patch testing at dermatology. Consider if hands disproportionately affected; pattern fits contact exposure.

Psoriasis

Well-demarcated; silvery scale; extensor surfaces; nail changes; scalp. Distinguish from eczema: psoriasis affects extensors; eczema affects flexures. Koebner phenomenon.

Do Not Miss
Emergency exclusion

Eczema herpeticum

Excluded today: no fever; no punched-out monomorphic erosions; not systemically unwell. Document as excluded. Safety-net given.

MRSA superinfection

Healthcare worker: nasal swab + skin swab to exclude. Infection control implications in care home setting. Specialist advice if MRSA confirmed.

📊 Eczema severity classification and treatment step
SeverityPOEMFeaturesTreatment step
Clear / almost clear0–2No active eczema; dry skin onlyEmollient only; soap substitute; trigger avoidance; proactive TCS ×2/week on prone areas
Mild3–7Areas of dry skin; occasional itch; little impact on daily lifeEmollient + mild TCS (hydrocortisone 1% on face/flexures; hydrocortisone 1–2.5% body) for 7–14 days; step down to emollient alone when clear
Moderate (Mrs. Mensah)8–16Frequent itch; redness; sleep affected; some functional impactEmollient ++ (500g/week); moderate-potent TCS (betamethasone 0.1% or mometasone 0.1% on body/limbs; hydrocortisone 1% on face); ×2/day for up to 14 days; treat superinfection first if present; written action plan; 4–6 week review
Severe17–24Widespread; incessant itch; sleep severely disrupted; major QoL impact; frequent relapsesAs above + dermatology referral; consider proactive TCS use; wet wrapping; tacrolimus; phototherapy (narrowband UVB); systemic (ciclosporin first-line per NICE CKS)
Very severe25–28Affects all aspects of life; constant itch; bleeding; infectionUrgent dermatology; systemic treatment (ciclosporin; methotrexate; azathioprine); biologic if fails systemic (dupilumab: NICE TA534; tralokinumab: NICE TA863; JAK inhibitors: abrocitinib; upadacitinib: NICE TA878/TA879)
🎓 SCA Checkpoint — Step 5TasksRelating to Others
Steroid phobia counselling
"I want to address the steroid concern directly. Skin thinning is a real risk — but only with prolonged overuse of a strong steroid on the wrong area. What we are going to do is completely different: a medium-strength cream on your arms for 2 weeks, with a written plan that tells you exactly when to use it and when to stop. That is safe. What is not safe is what has been happening — using too little cream means the inflammation stays uncontrolled, and that causes its own harm: infection; sleep disruption; anxiety. I am going to give you a written plan today."
Deductions
  • Dismissing steroid phobia without addressing it — a patient who leaves with the same steroid fear will continue to under-apply; the same treatment failure will recur
6
Step 6
Referral — Dermatology · Occupational Health · Allergy Clinic · NHS Talking Therapies
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Most eczema is managed in primary care. Referral triggers: severe eczema not controlled by topical treatment; diagnostic uncertainty; suspected contact allergy (patch testing); occupational eczema; systemic treatment consideration.
ReferralUrgencyGP actions firstMust NOT do
Occupational Health — Mrs. Mensah’s most important referral todayRoutine — 4–6 weeksRefer to occupational health service (via employer or independent). Include: diagnosis (atopic eczema; hand eczema; occupational wet work trigger); job description (healthcare assistant; care home; frequent handwashing); current treatment. Occupational health will: assess wet work exposure; recommend alternative hand cleansers (alcohol-based gel tolerated better than soap for frequent use); prescribe barrier cream protocol; advise on glove type (non-latex; cotton liner); recommend reasonable adjustments under Equality Act 2010. Interim today: prescribe Diprobase gel (lighter; can be used during work shift without leaving greasy residue); Epaderm ointment (heavier; for overnight use); alcohol hand gel to replace some hand-washing episodes where clinically appropriate.Do NOT issue blanket sick note without exploring adjustments first. Do NOT fail to refer — occupational trigger driving hand eczema will continue to cause relapses regardless of topical treatment if not addressed.
Dermatology — if topical treatment failsRoutine — if POEM not improving at 6–8 weeks with optimal topical treatmentIndications for referral: POEM not improving despite adequate emollient + correct TCS potency for 6–8 weeks; suspected contact dermatitis requiring patch testing; recurrent bacterial superinfection; diagnostic uncertainty; severe eczema (POEM ≥17); eczema requiring systemic treatment (ciclosporin; phototherapy; biologic). For Mrs. Mensah: not indicated today — plan if no improvement at 6-week review. Include in referral: POEM at baseline and review; treatment history; occupational context; current medications.Do NOT refer before attempting optimal topical management for minimum 6–8 weeks. Do NOT delay referral beyond 8 weeks if clearly failing topical management — systemic treatment options are available and effective.
Allergy clinic — patch testing; food allergy (children)RoutinePatch testing (dermatology or allergy clinic): suspected contact allergen contributing to eczema; hand eczema in occupational setting; eczema pattern fitting contact distribution. Skin prick testing / specific IgE: children with moderate-severe eczema not responding to topical treatment (NICE CKS). Foods to test: egg; cow’s milk; wheat; soy; peanuts. For Mrs. Mensah: request patch testing referral to dermatology for rubber chemicals; preservatives; fragrance series (occupational contact dermatitis screen).Do NOT perform allergy testing in adults unless there is a specific clinical suspicion of contact allergy — routine allergy testing in adult atopic eczema without specific indication does not change management. Do NOT request food allergy tests in adults with eczema unless clear food-trigger history.
NHS Talking Therapies / Psychology — eczema and mental healthRoutine — PHQ-9 7; mild depressionPHQ-9 7 (mild depression) in context of eczema and sleep disruption. NHS Talking Therapies referral for CBT: habit reversal training is specifically effective for the itch-scratch cycle in eczema (addresses compulsive scratching as a behaviour); CBT addresses anxiety about steroid use; depression from itch and sleep deprivation. Dermatology psychology service: specialist psychological support for chronic skin conditions. Social prescribing: eczema support groups; British Skin Foundation; National Eczema Society.Do NOT treat PHQ-9 7 with antidepressants as first-line without considering whether it will improve as eczema improves. Do NOT ignore the psychological impact of eczema — DLQI in severe eczema is comparable to cancer treatment QoL impairment.
🎓 SCA Checkpoint — Step 6Tasks
Occupational health referral
"I am going to refer you to occupational health today. They are specialists in exactly this situation — a healthcare worker with hand eczema made worse by the job. They will be able to recommend the right type of hand cleanser for your work; the right type of gloves; and any other adjustments that would help. In the meantime, I am going to prescribe you a gel emollient that you can use during the day at work without it being too greasy."
Deductions
  • Failing to refer to occupational health in a healthcare worker with occupational hand eczema — the trigger will continue to drive relapse regardless of how well topical management is optimised
7
Step 7
Management — Emollient First · TCS Ladder · FTU · Steroid Phobia · Written Action Plan · Safety-Netting
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7A — Address Mrs. Mensah’s expectation: steroid safety and the wish to use less cream
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Mrs. Mensah is applying TCS too sparingly due to steroid phobia — validate the concern; explain the evidence; negotiate a specific safe plan
1
Validate — the concern is completely understandable

Her mother’s experience gave her a genuine and protective instinct. Never dismiss it — acknowledging that the concern is valid makes the subsequent evidence more credible.

"I completely understand the worry — your mother’s experience has made you cautious, and that is a natural protective instinct. I want to explain what is true about steroid creams, and what is a myth, so you can use them with confidence."
2
Explain — when TCS is safe; when it is not

The specific, evidence-based explanation is: skin atrophy is real but requires prolonged high-potency use in the wrong location. A 2-week course of betamethasone 0.1% on the arms is safe. Hydrocortisone 1% on the face is safe for short periods. The FTU demonstrates what “enough” looks like.

"The skin thinning that happened to your mother is a real risk — but it happens with prolonged, daily use of a strong cream on the face or skin folds without any break. What I am prescribing — a medium-strength cream on your arms for 2 weeks, with a written plan for when to stop — is safe. I am also going to show you exactly how much cream to use."
3
Negotiate — written eczema action plan as a contract of safety

The written action plan transforms the steroid phobia into a navigable, bounded protocol. It tells Mrs. Mensah exactly when to step up; when to step down; how much to use; and what signs should make her seek help. It removes uncertainty and replaces it with control.

"I am going to give you a written plan today — it tells you exactly which cream to use on which part of the body; how much to use; for how many days; and when to stop. With a plan like this, you are in control. The risk of using too much is managed by the plan — and the risk of using too little — which is what has been happening — is prevented."
Key principle: Steroid phobia is a failure mode that perpetuates eczema. Addressing it with specific evidence and a written action plan is as clinically important as choosing the correct TCS potency.
7B — Treatment goals
Treatment goals
POEM reduction from 16 to <8 at 4–6 week reviewSleep uninterrupted by itch — POEM Q2 (sleep) normalised Hands improving — able to work without pain or open woundsPHQ-9 improvement as eczema improves — target <5 at review Mrs. Mensah confident using TCS — steroid phobia addressedOccupational trigger managed — occupational health advice implemented Written eczema action plan provided and explained today4–6 week review booked; POEM re-scored; treatment adjusted
Motivational language
"The good news is that we know exactly what is going wrong and we can fix it. The emollient has run out — we can fix that today with a prescription. The cream is not being applied correctly — I am going to show you how. And the trigger at work — the hand washing — we are going to address through occupational health."
"I know it is embarrassing at work — and I want to get this under control so that it is not affecting you there. With the right treatment applied correctly, most people with eczema like yours get significant improvement within 4 weeks."
7C — Non-medication management
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Emollient Use — The Cornerstone
500g/week body; 250g/week face+hands; minimum 4×/day; soap substitute always
Why it matters

The skin barrier defect in atopic eczema results in transepidermal water loss (TEWL) — the skin dries out unless the barrier is maintained with emollient. Emollient must be: (1) used regularly — not just when the skin is dry; (2) used in large quantities (500g/week is the minimum for body eczema — most OTC moisturisers come in 200–400ml tubes and would run out in days); (3) used before TCS (apply emollient first; wait 20–30 minutes; then apply TCS). The emollient is not optional — it is the foundation on which TCS efficacy depends.

Prescribing

Prescribe: Epaderm ointment 500g (body; overnight; very greasy; best barrier); Diprobase gel 500g (daytime; lighter; good for work use); aqueous cream or Oilatum SHOWER EMOLLIENT (as soap substitute — never leave on as leave-on emollient — it causes irritation if not washed off). Paraffin-based emollients: fire risk — warn patient; do not smoke near open flame when using paraffin-based emollient. Apply in direction of hair growth to reduce folliculitis.

Emollient is the only treatment proven to reduce TCS requirement; prevents and treats eczema independently
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Sleep and Itch Management
Cool bedroom; cotton clothing; emollient at bedtime; chlorphenamine 4mg PRN at night
Why

Itch worsens at night. The itch-scratch cycle is amplified during sleep: scratching releases inflammatory mediators; worsens eczema; disrupts sleep further. Breaking the cycle requires: (1) physical barriers (cotton mittens; appropriate clothing); (2) cool room temperature; (3) emollient at bedtime; (4) cut fingernails short. Sedating antihistamine (chlorphenamine 4mg) at night may help sleep during acute flares — but note: evidence for antihistamines in eczema itch is limited; they help sleep via sedation rather than direct antipruritic effect.

Practical

Cool (16–18°C) bedroom. 100% cotton loose clothing. Emollient applied generously at bedtime. Cut fingernails short. Cotton mittens or bandaging if scratching uncontrolled during sleep. Chlorphenamine 4mg at night during severe flares (not long-term; not in children under 6 months; causes daytime sedation — warn and document).

Improving sleep: reduces central sensitisation of itch; improves mental health; allows skin to heal overnight
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Trigger Avoidance
HDM reduction; pet dander management; fragrance-free; cotton clothing; stress management
Evidence

Trigger avoidance reduces flare frequency and severity. Common triggers: house dust mite (allergen-impermeable mattress and pillow covers; hot washing bedding at 60°C weekly; avoid carpet in bedroom); pet dander (avoid pets in bedroom; HEPA filter); fragrances (use fragrance-free personal care products; avoid fragranced washing powder — non-bio is not necessarily fragrance-free — use Surcare or similar); clothing: 100% cotton next to skin; avoid wool directly on skin; overheating and sweating triggers itch.

Practical — Mrs. Mensah

Identify her specific triggers. Occupational: alcohol gel instead of soap for some handwashing episodes; non-latex gloves with cotton liners; barrier cream (Diprobase or Neutrogena Norwegian Formula) applied before shift. Home: review washing powder; fragrance-free skin products; HDM reduction if bedroom triggers identified.

Trigger avoidance reduces flare frequency; evidence strongest for HDM and occupational wet work reduction
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Habit Reversal Training — NHS Talking Therapies
CBT for itch-scratch cycle; NHS Talking Therapies referral; National Eczema Society resources
Evidence

Habit reversal training (HRT) is a CBT-derived psychological intervention specifically for the itch-scratch cycle in eczema. It teaches awareness of scratching (which often occurs automatically without conscious awareness); the introduction of competing responses (pinching instead of scratching; applying emollient instead of scratching); and relaxation techniques. RCTs show significant reduction in itch and scratching with HRT. NHS Talking Therapies can provide CBT — specify “itch-scratch cycle; habit reversal training” in the referral.

Resources

National Eczema Society: eczema.org — helpline 0800 089 1122; patient support; local groups. NHS Talking Therapies self-referral or GP referral. British Skin Foundation: britishskinfoundation.org.uk. Dermatology psychology service — if available locally.

Habit reversal training: RCT evidence for itch-scratch cycle reduction; CBT addresses steroid phobia; anxiety; sleep
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Hand Protection at Work
Barrier cream before shift; alcohol gel preferred over soap; cotton liner gloves; occupational health protocol
Why critical for Mrs. Mensah

Hand eczema in a healthcare worker is an occupational health emergency and a patient safety issue. Open or weeping skin on the hands of a care home worker = infection risk to vulnerable residents. Management hierarchy: (1) reduce exposure (alcohol gel instead of soap for some episodes); (2) barrier protection (barrier cream; cotton liner gloves under non-latex gloves); (3) treatment of existing hand eczema (emollient + correct TCS — ointments are more effective for hand eczema; potent TCS appropriate for hand dorsum); (4) occupational health referral; (5) sick note if unable to work safely.

Prescribed hand regimen

Diprobase gel (daytime; before gloves; light non-greasy); Epaderm ointment (overnight; intensive overnight repair); betamethasone 0.1% ointment (back of hands; NOT between fingers; apply 2×/day for up to 14 days during flare); soft paraffin under cotton gloves overnight. NEVER: potent TCS in web spaces; prolonged potent TCS on palms without monitoring.

Optimised hand regimen: 4–6 weeks; review; if not improving: patch testing (dermatology); consider allergen
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Written Eczema Action Plan
Provide today; step-up and step-down rules; emergency signs; what to do when
Why essential

A written eczema action plan reduces TCS use (allows appropriate step-down); reduces GP appointments (patient self-manages minor flares); addresses steroid phobia (gives clear rules for safe use); and reduces eczema-related hospitalisations. NICE CKS recommends written action plans as a core component of eczema management. The plan should include: (1) baseline maintenance (emollient; frequency; quantity); (2) mild flare: which TCS; which area; duration; (3) moderate flare: step up to which preparation; (4) when to contact GP; (5) emergency signs (eczema herpeticum).

For Mrs. Mensah today

Maintenance: Epaderm ointment 4×/day body; twice daily face; soap substitute always. Flare: betamethasone 0.1% body and limbs ×2/day for maximum 14 days; hydrocortisone 1% face ×2/day for maximum 7 days; step back to emollient alone when clear. Contact GP: if flare not improving in 2 weeks; if signs of infection; if punched-out blisters or fever (A&E immediately).

Written action plan: reduces TCS use; reduces GP appointments; addresses steroid phobia; NICE CKS recommendation
7D — Prescribing guide — the steroid ladder
The prescribing decision in eczema is determined by: site (face/flexures — mild only; body/limbs — moderate-potent); severity (POEM score; IGA); and current treatment (Mrs. Mensah needs step-up from hydrocortisone 1% to betamethasone 0.1% on body/limbs; continue hydrocortisone 1% on face only). Emollient must always be prescribed alongside TCS — it is not optional.
Face and eyelids — mild TCS only
  • Hydrocortisone 1% cream or ointment ×2/day for maximum 7 days
  • Never potent TCS on face: skin atrophy; telangiectasia; perioral dermatitis; glaucoma (periorbital)
  • Eyelids: hydrocortisone 0.5% (lower potency); ophthalmology referral if recurrent periorbital TCS use needed
  • If hydrocortisone 1% inadequate on face: tacrolimus 0.1% ointment (Protopic) or pimecrolimus 1% cream (Elidel) — TCI preferred over potent TCS on face
Face rule: mild TCS or TCI — NEVER potent TCS on face regardless of severity
Body; limbs; hands — moderate-potent TCS (Mrs. Mensah)
  • Betamethasone valerate 0.1% (Betnovate) ×2/day — up to 14 days for flare
  • OR mometasone furoate 0.1% (Elocon) ×once daily — equivalent potency; once-daily dosing improves adherence
  • OR fluticasone propionate 0.05% (Cutivate) ×twice daily
  • Hands: ointment formulation preferred (more occlusive; better penetration)
  • FTU: arms = 3 FTU each; demonstrate at consultation
Step down to emollient alone when clear; proactive twice-weekly TCS on prone areas to prevent relapse
Infection: treat first; then step up TCS
  • Bacterial (S. aureus): Fucidin (fusidic acid 2%) cream or ointment ×3/day for 7 days
  • Or combination: Fucibet (betamethasone + fusidic acid) if needs TCS and antibiotic simultaneously — 7 days only (resistance concern)
  • Widespread: oral flucloxacillin 500mg QDS ×7 days; penicillin allergy: cefalexin 500mg TDS
  • MRSA: specialist dermatology advice; rifampicin + fusidic acid
  • Viral (eczema herpeticum): 999 — IV aciclovir — do NOT treat with topical steroid
Topical calcineurin inhibitors (TCI) — steroid-sparing
  • Tacrolimus 0.1% ointment (Protopic): adults; moderate-severe eczema; face; flexures; eyelid skin (steroid-sparing); ×2/day; initial burning/stinging (settles after 1 week)
  • Pimecrolimus 1% cream (Elidel): adults and children ≥2y; mild-moderate; better tolerated (less stinging)
  • Both: no skin atrophy risk — preferred over potent TCS for face; eyelids; genitalia; skin folds
  • Proactive use ×2/week on problem areas: reduces relapses
Systemic treatment (dermatology — POEM ≥17 + failed topical)
  • Phototherapy (narrowband UVB): first-line secondary care; 3×/week ×6–8 weeks; effective; no immunosuppression
  • Ciclosporin: NICE CKS first-line systemic; 2.5–5mg/kg/day; monitor BP; renal function; FBC monthly; maximum 1–2 years continuous use
  • Dupilumab (Dupixent): NICE TA534; adults; EASI ≥16; failed ciclosporin; subcutaneous 300mg fortnightly; highly effective
  • JAK inhibitors (abrocitinib; upadacitinib): NICE TA878/TA879; oral; specialist only
7E — Medication selector

Select site and severity — eczema TCS selection guide

Eczema treatment guidance
Mrs. Mensah (POEM 16; body and limbs): betamethasone valerate 0.1% (Betnovate) ointment ×2/day for up to 14 days on body and limbs; FTU (arms: 3 FTU each; trunk front 7 FTU); emollient 500g/week (Epaderm ointment body; Diprobase gel daytime/work). Face only: hydrocortisone 1% ×2/day — maximum 7 days. Hands: betamethasone 0.1% ointment (back of hands; NOT web spaces) + barrier cream (Diprobase gel) during work shift. Step down to emollient alone when clear. Proactive TCS ×2/week on prone areas when in remission. Written eczema action plan provided. Eczema herpeticum: 999 immediately — do NOT apply TCS. Bacterial superinfection: Fucidin (fusidic acid 2%) ×3/day for 7 days BEFORE stepping up TCS. Steroid phobia: tacrolimus 0.1% (Protopic) as alternative to potent TCS on face; TCI preferred for face and flexures to avoid atrophy.
7F — Drug reference cards
Emollients (Cornerstone)
Epaderm ointment · Diprobase gel · Hydromol · Doublebase · Aveeno · Oilatum · QV · Prescribe 500g/week body; soap substitute always
✓ First-line always — 500g/week; minimum 4×/day; before TCS; soap substitute; fire safety (paraffin)
Foundation of ALL eczema management — without adequate emollient; TCS will not work effectively500g/week body; 250g face/hands; minimum 4×/day; soap substitute at every wash
✓ How to use correctly
Emollient must be applied in large quantities, frequently — the two most common errors are using too little and not using it often enough. The 500g/week figure seems excessive to patients; they must be told that this is normal and expected. Apply after bathing (while skin is still slightly damp to trap moisture); minimum 4 times daily; and specifically before applying TCS (apply emollient; wait 20–30 minutes; then apply TCS). Apply in the direction of hair growth to reduce folliculitis. The emollient provides the barrier protection that the skin lacks; TCS treats the inflammation once the barrier is in place. Without the emollient, the inflamed skin is exposed to more irritants and the TCS has to work harder. Soap substitute: use at every wash (hand washing; shower; bath). NEVER use aqueous cream as a leave-on emollient — it contains sodium lauryl sulphate which causes irritation — it should be used and washed off as a soap substitute only.
✗ Fire risk and precautions
FIRE SAFETY: paraffin-based emollients (Epaderm; Hydromol; 50:50 white soft paraffin + liquid paraffin) soak into clothing and bedding and make them highly flammable. Warn: do not smoke near open flame; do not use near open fires or candles; use flame-retardant nightwear. Multiple deaths from burns have been recorded. This warning must be documented and given verbally at prescribing. Alternative: non-paraffin emollients (Aveeno; Diprobase; QV) do not carry fire risk and are appropriate when fire risk is a concern.
⚠ Practical issues
Greasy ointments (Epaderm): best for overnight use and dry skin; may be impractical for work. Creams (Diprobase): lighter; less greasy; better for daytime; less occlusive than ointments. Gels (Doublebase Dayleve): very light; excellent for work and daytime use. The right emollient is the one the patient will actually use — prescribe a range for different situations. Emollient trial: try several and ask patient to report which they find most acceptable; adherence depends on acceptability, not clinical evidence alone.
🔬 Monitoring
Prescribe adequate quantities (500g/week body = 2kg/month); check repeat prescription requests; if patient is not requesting refills: either not using enough or not using it at all — review at next appointment. POEM re-score at every review — improvement in dryness (Q7) and itch (Q1) reflects emollient adequacy.
💬 Counselling

"This cream needs to go on at least 4 times a day — when you wake up; before lunch; before dinner; and before bed. I know that seems like a lot. The reason is that your skin cannot keep its own moisture in because of the eczema, so we are replacing the barrier from the outside. You also need to use it instead of soap — every time you wash. The prescription I am giving you is for 500g — that should last about a week for your body. And if you have paraffin-based creams at home, please do not smoke near open flames when you are wearing them — they can make clothing flammable."

Emollient prescribing: NICE CKS cornerstone; 500g/week body; soap substitute always; apply before TCS; in direction of hair growth; paraffin fire risk must be documented and counselled. Most common SCA error: not quantifying emollient — saying “use moisturiser” without prescription quantity and technique education misses the Tasks mark.

Mild TCS — Hydrocortisone 1%
Hydrocortisone 1% cream/ointment · Face & flexures · Maximum 7–14 days · Safe for all age groups · NEVER potent TCS on face
✓ Face; eyelids; flexures; children — mild TCS only; 7 days maximum face; 14 days body mild eczema
Mild eczema (POEM 3–7) OR face/eyelids at any severity×2/day; 7 days face; 14 days body; step down to emollient when clear; FTU guide
✓ Face and eyelid safety
Hydrocortisone 1% is the appropriate TCS for the face at any age. It has minimal risk of skin atrophy even with regular use on the face when applied correctly (maximum 7 days; step down when improved). For mild eczema on the body and limbs: hydrocortisone 1% ×2/day for 14 days is appropriate as a starting point. For children: hydrocortisone 1% on body and limbs (step up to clobetasone if needed); hydrocortisone 0.5% on face in infants. Ointment formulation: more occlusive; better for dry or lichenified skin. Cream formulation: less greasy; better tolerated on weeping or infected skin (though avoid TCS on infected skin).
✗ When hydrocortisone 1% is not enough
If no improvement on face after 7 days of hydrocortisone 1%: switch to TCI (tacrolimus 0.1% or pimecrolimus 1%) — never step up to potent TCS on face. If no improvement on body after 14 days: step up to moderate-potent TCS (clobetasone 0.05%; betamethasone 0.1%). If infected: treat infection first before TCS.
⚠ FTU guide for hydrocortisone
Face and neck: 2.5 FTU per application. Each arm: 3 FTU. Each hand: 1 FTU. Trunk front: 7 FTU. Back: 7 FTU. Each leg: 6 FTU. Each foot: 2 FTU. Demonstrate with the patient at the consultation: squeeze from the nozzle from fingertip to first knuckle = 1 FTU.
🔬 Monitor
POEM at 4–6 weeks. Face: examine for skin atrophy; telangiectasia; perioral dermatitis at each review. Step down to emollient alone as soon as eczema is clear — do not continue TCS indefinitely when eczema is controlled.
💬 Counselling

"This is a mild steroid cream for your face. Use it twice a day for a maximum of 7 days when your face flares. Stop it when the skin is clear and go back to just the moisturiser. If it is not working after 7 days on your face, come back and we will try a different type of cream — one that doesn’t have steroid in it at all."

Hydrocortisone 1%: mild TCS; face; flexures; children; maximum 7 days face. FTU guide is a core prescribing competency for eczema — demonstrating it in the SCA is a Tasks mark. Never potent TCS on face — this is the most common TCS prescribing error.

Potent TCS — Betamethasone 0.1% / Mometasone 0.1%
Betnovate (betamethasone valerate 0.1%) · Elocon (mometasone furoate 0.1%) · Body and limbs · NEVER face · Maximum 14 days per course
✓ Body and limbs; moderate-severe eczema — Mrs. Mensah’s prescription; 14 days; FTU; step down when clear
Moderate-severe eczema on body and limbs (POEM 8–24); NOT face×2/day (betamethasone) or ×once daily (mometasone); up to 14 days per course; FTU
✓ Mrs. Mensah’s prescription — why potent TCS is appropriate here
Betamethasone 0.1% (Betnovate) or mometasone 0.1% (Elocon): potent TCS; appropriate for body and limb eczema in adults with POEM 8–16. Mrs. Mensah has been using hydrocortisone 1% on her arms — inadequate for her severity. Potent TCS on body and limbs for 14 days is safe. Mometasone 0.1%: once-daily dosing improves adherence; equivalent potency to betamethasone 0.1% twice daily. Prescribe ointment (oily; more occlusive; better for dry thick skin); or cream (less occlusive; better for wet or weeping skin). FTU for arms: 3 FTU per arm per application. Demonstrate at consultation.
✗ Absolute contraindications by site
NEVER potent TCS on face; eyelids; genitalia; skin folds — skin atrophy; telangiectasia; striae; perioral dermatitis; glaucoma (periorbital). NEVER clobetasol propionate 0.05% (Dermovate; very potent) without dermatology supervision. NEVER step up TCS potency into infected skin — treat infection first.
⚠ Side effects — with misuse
Skin atrophy: thinning; visible subcutaneous vessels; easy bruising — occurs with prolonged continuous use. Striae (stretch marks): particularly axillae; groin; breasts with long-term potent TCS. Telangiectasia. Perioral dermatitis (facial misuse — not applicable here). Systemic absorption: rare with topical use; more common with very potent TCS on large areas; children (thinner skin). Adrenal suppression: theoretical with very large amounts but not a practical concern with appropriate short courses.
🔬 Monitor
POEM at 4–6 weeks. Examine application areas for atrophy; striae; telangiectasia at review. Proactive use: betamethasone or mometasone twice weekly on prone areas once eczema controlled — reduces relapse without significant atrophy risk. Step down: when eczema clears, step back to emollient alone or proactive TCI; do not continue daily TCS indefinitely.
💬 For Mrs. Mensah

"I am prescribing you a medium-strength cream for your arms and body. This is safe on these areas for up to 2 weeks. I am going to show you exactly how much to use — [demonstrate FTU] — one of these squeezes covers both your hands; you need 3 of these for one arm. Do not use it on your face — only the other cream I am giving you on your face. When the skin on your arms is back to normal, stop the stronger cream and just use the moisturiser again."

Betamethasone 0.1% / mometasone 0.1%: potent TCS; body and limbs only; Mrs. Mensah’s step-up prescription. Demonstrate FTU at consultation — Tasks mark. NEVER face; eyelids; genitalia; skin folds — most common TCS prescribing error. 14-day maximum per course; step down to emollient when clear. Proactive ×2/week use when in remission reduces relapse risk.

Topical Calcineurin Inhibitors (TCI)
Tacrolimus 0.1% ointment (Protopic — adults) · Pimecrolimus 1% cream (Elidel) · Face; flexures; steroid-sparing · No skin atrophy
✓ Face; flexures; eyelid skin — steroid-sparing; no atrophy risk; preferred when potent TCS on face inappropriate
Second-line (face; flexures; TCS failure; steroid phobia); NICE CKSTacrolimus 0.1% ×2/day (adults); pimecrolimus 1% ×2/day; proactive ×2/week for maintenance
✓ When TCI is preferred over TCS
Topical calcineurin inhibitors (TCI) inhibit T-cell activation; calcineurin phosphatase; and downstream release of Th2 inflammatory cytokines (IL-4; IL-13; IL-31). Key advantages over TCS: no skin atrophy; no telangiectasia; safe for long-term use. Indications: (1) Face; eyelids; periorbital skin — where potent TCS are inappropriate; (2) Flexures; genitalia; skin folds; (3) Patients with TCS-induced atrophy; (4) Steroid phobia (TCI as alternative to TCS on face to address phobia). Tacrolimus 0.1%: more potent; ointment; better for moderate-severe; licensed for adults. Pimecrolimus 1%: cream; better tolerated (less stinging); good for mild-moderate; better for daytime use. Black-box warning (malignancy): previously applied; now largely withdrawn; long-term data is reassuring; NICE CKS supports TCI use.
✗ Limitations
Initial burning and stinging on application (usually settles after 1 week; warn patient; improve with consistent use). Not suitable for infected skin. Avoid in active infection; open wounds. Immunocompromised patients: use with caution; seek dermatology advice. Cost: more expensive than TCS. Not first-line — NICE CKS: offer TCI as second-line if TCS not tolerated or ineffective for face/neck; after TCS trial.
⚠ Side effects
Burning; stinging; pruritus at application site (common first 1–2 weeks). Skin infection risk (monitor; avoid in active infection). No systemic immunosuppression at topical doses. Sunscreen: recommended with tacrolimus (theoretical photosensitivity; evidence limited). Do NOT use with UV therapy.
🔬 Monitor
POEM at 4–6 weeks. Skin infection at each review. If proactive maintenance use (×2/week): review at 6 months; continue if tolerated and effective. Long-term use registry: reassuring safety data now available for tacrolimus and pimecrolimus.
💬 Counselling

"This cream works on the inflammation in your skin without the steroid. It is particularly useful on the face because it doesn’t cause skin thinning. When you first apply it, it may sting or burn a little — that usually settles after a few days. If you use it twice a week once your face is clear, it can help prevent the eczema from coming back in that area."

TCI (tacrolimus 0.1%; pimecrolimus 1%): steroid-sparing; no atrophy; preferred for face; eyelids; flexures; steroid phobia; TCS atrophy. Initial burning/stinging — warn. NICE CKS: second-line after TCS for face/neck. Proactive ×2/week maintenance reduces relapse. Black-box malignancy warning largely withdrawn — reassure if patient asks. For Mrs. Mensah with steroid phobia: TCI is an excellent face option that addresses the phobia by avoiding steroids on that site entirely.

Topical Fusidic Acid (Fucidin) / Oral Flucloxacillin
Fucidin cream 2% · Fucibet (fusidic acid + betamethasone) · Flucloxacillin 500mg QDS · Cefalexin 500mg TDS (penicillin allergy) · Treat infection FIRST before stepping up TCS
✓ Bacterial superinfection (S. aureus) — treat infection first; maximum 7 days fusidic acid topical (resistance)
Infected eczema only — NOT for routine use; treat infection then step up TCS separatelyFucidin 2% ×3/day ×7 days topical; flucloxacillin 500mg QDS ×7 days oral if widespread
✓ When to treat infection
Signs of bacterial superinfection in eczema: honey-coloured or yellow-brown crusting; weeping; pustules; rapidly spreading area of erythema; not responding to TCS alone. S. aureus is the most common organism. Principle: TREAT INFECTION FIRST; then step up TCS once skin is clear of infection. Never step up TCS potency into actively infected skin — this suppresses the immune response needed to clear the infection. Topical fusidic acid: for localised infection (7 days only — resistance emerging with longer use; limit courses to prevent resistance). Oral antibiotic: for more widespread infection or not responding to topical. Fucibet (fusidic acid + betamethasone): combination; useful when both infection and inflammation need treatment simultaneously; 7 days maximum.
✗ Resistance and prescribing limits
Fusidic acid resistance: maximum 7-day topical course; do NOT repeat routinely without swab. Fucidin overuse has led to significant fusidic acid resistance in S. aureus in eczema patients — send swab if recurrent; consider alternative antibiotic. MRSA: specialist dermatology; rifampicin + fusidic acid or trimethoprim; infection control notification in care home setting. Eczema herpeticum: topical antibiotics do NOT treat viral infection; patient deteriorates; 999 if features present.
⚠ Side effects
Fusidic acid topical: local irritation; rare sensitisation (contact allergy to fusidic acid — consider if eczema worsening on Fucidin). Oral flucloxacillin: GI disturbance; take on empty stomach (30 minutes before food). Penicillin allergy: cefalexin 500mg TDS (cross-reactivity <2%); or clarithromycin 500mg BD if history of anaphylaxis.
🔬 Monitor
Review at 1 week: is infection cleared? If cleared: step up TCS appropriately. If not cleared: swab; check organism; antibiotic sensitivities; consider MRSA. Recurrent infection: consider regular antiseptic wash (dilute bleach bath: 1 teaspoon household bleach in 10L bathwater; 5–10 minutes ×2/week; Dermol wash 500 or chlorhexidine wash — reduces S. aureus colonisation).
💬 Counselling

"There are signs of a bacterial infection in your eczema — the yellow crusting is the typical sign. I am treating that first with an antibiotic cream before stepping up the steroid cream — because putting a stronger steroid on infected skin would suppress your immune response and make the infection worse. Once the infection is clear in about a week, we step up the steroid cream."

Fusidic acid: bacterial superinfection (S. aureus) only; 7 days maximum topical (resistance). TREAT INFECTION FIRST then step up TCS. Fucibet: combination; 7 days only. MRSA: specialist + infection control in care home. SCA: stepping up TCS into infected eczema without treating infection first = Tasks deduction. Eczema herpeticum: IV aciclovir — not antibiotics.

Systemic Treatment — Ciclosporin / Dupilumab
Ciclosporin 2.5–5mg/kg/day (NICE CKS first-line systemic) · Dupilumab 300mg SC fortnightly (NICE TA534) · JAK inhibitors (NICE TA878/879) · Specialist initiation only
✓ Severe eczema (POEM ≥17) failed topical treatment — dermatology referral required; specialist initiation; GP monitoring role
Specialist only — POEM ≥17; failed topical; dermatology referralCiclosporin 2.5–5mg/kg/day; dupilumab 600mg SC (loading) then 300mg SC fortnightly
✓ Stepwise systemic treatment
NICE CKS stepwise systemic treatment for severe eczema failing topical management: (1) Phototherapy (narrowband UVB): first-line in secondary care; 3×/week ×6–8 weeks; effective; no immunosuppression; no long-term side effects; cannot be continued indefinitely. (2) Ciclosporin: NICE CKS first-line systemic immunosuppressant for severe eczema; 2.5mg/kg/day start; increase to maximum 5mg/kg/day; monitor BP; renal function (U&E; creatinine); FBC; LFTs at baseline; every 2 weeks for 3 months; then monthly; maximum 1–2 years continuous use (nephrotoxicity). (3) Dupilumab (Dupixent; NICE TA534): biologic; IL-4/IL-13 receptor antagonist; adults with moderate-severe eczema; EASI ≥16 or IGA ≥3; inadequate response to or contraindication to ciclosporin; subcutaneous 300mg fortnightly; highly effective; very safe profile; no immunosuppression; expensive — NHS criteria apply.
✗ Ciclosporin monitoring requirements
Ciclosporin: CONTRAINDICATED in uncontrolled hypertension; renal impairment; active malignancy; active infection; pregnancy. Drug interactions: avoid NSAIDs (nephrotoxicity); grapefruit (CYP3A4); statins (myopathy); metformin (lactic acidosis risk). Monitoring: BP (can cause severe hypertension); renal function (creatinine rise >25% above baseline: reduce dose); FBC; LFTs. Maximum treatment duration 1–2 years. GP role: monitoring bloods; managing side effects; communication with dermatology.
⚠ Dupilumab side effects
Injection site reactions (common; usually mild). Conjunctivitis (common with dupilumab — approximately 10% of patients; requires ophthalmology if severe; artificial tears; topical cyclosporin eye drops). Herpes infection: slightly increased (not related to immunosuppression — unclear mechanism). Very low infection risk overall — not an immunosuppressant. JAK inhibitors: infection risk (including TB); thromboembolism; must screen for TB before starting.
🔬 GP monitoring role for ciclosporin
Ciclosporin monitoring: baseline (and then): BP fortnightly ×3 months then monthly; U&E; creatinine fortnightly ×3 months then monthly; LFTs; FBC; lipids. If creatinine >25% above baseline: reduce dose by 1mg/kg/day; if no improvement in 1 month: stop. BP >160/100: reduce dose; add amlodipine (avoid ACEi — worsens nephrotoxicity). NICE CKS: GP to monitor if dermatology initiated. Shared care protocol should be in place.
💬 Counselling (dupilumab)

"This injection works on the specific pathway that drives your eczema inflammation. It is not a steroid and it doesn’t suppress your immune system in the way that older tablets do — so the infection risk is very low. You inject it yourself at home fortnightly. The most common side effect is some redness and swelling at the injection site — and some people develop eye irritation. I want you to let me know if your eyes become red or sore so we can sort that out."

Ciclosporin: NICE CKS first-line systemic; monitoring: BP; U&E; creatinine; LFTs; FBC (fortnightly ×3m then monthly); maximum 1–2 years; nephrotoxicity; hypertension. Dupilumab (Dupixent; NICE TA534): IL-4/IL-13 inhibitor; moderate-severe; failed ciclosporin; EASI ≥16; subcutaneous fortnightly; conjunctivitis (monitor); no immunosuppression. GP role: monitoring; shared care. JAK inhibitors: NICE TA878/TA879; TB screen first; thromboembolism risk.

7G — Psychosocial impact of eczema
🧑️
Eczema affects appearance; work; sleep; relationships; and mental health — these must be addressed alongside the topical prescription
Severe eczema causes a quality of life impairment comparable to severe psoriasis; rheumatoid arthritis; and cancer treatment. The visible nature of the condition adds stigma; self-consciousness; and workplace discrimination. Sleep deprivation from chronic itch drives depression and reduces the immune regulation that helps control the eczema. Addressing these dimensions is not optional — it is part of the clinical management.
💼
Work and Occupational Safety

Mrs. Mensah’s hand eczema affects her ability to work safely as a healthcare assistant. Open wounds increase infection risk to residents. Occupational health referral is mandatory. She has legal rights to reasonable adjustments under the Equality Act 2010 (disability includes chronic skin conditions affecting daily activities).

"Your eczema on your hands affects your ability to work safely, and you have a right to reasonable adjustments from your employer — that might be a different type of hand cleaner; different gloves; or adjusted duties while the eczema is treated. Occupational health will advise on this."
👔
Self-Consciousness and Stigma

Visible eczema on arms; neck; and hands in a care environment creates self-consciousness and perceived stigma. Mrs. Mensah is embarrassed. This affects professional confidence and social functioning. Addressing it is part of the eczema management — not separately, but as a motivation for effective treatment: getting the eczema controlled will directly improve her confidence at work.

"I know how visible this is — and I understand it affects how you feel at work. Getting this under control quickly is our goal, and with the right treatment applied correctly, most people see significant improvement within 4 weeks."
😴
Sleep and Fatigue

Chronic itch disrupts sleep; sleep deprivation worsens itch sensitivity; worsens immune regulation; worsens eczema. This vicious cycle must be broken. Emollient at bedtime; cool bedroom; cotton clothing; chlorphenamine 4mg during acute flares — each contributes. Effective treatment of the eczema itself is the most powerful sleep intervention.

"The itch at night is one of the most exhausting aspects of eczema. As the eczema improves with the right treatment, the night-time itch should reduce significantly. In the meantime, a cool bedroom; cotton clothing; and putting the emollient on before bed all help."
💕
Relationships and Intimacy

Eczema affects skin-to-skin contact; relationships; and intimacy. The self-consciousness of visible eczema reduces confidence in social and intimate situations. Opening this topic sensitively — “has the eczema been affecting your relationships or your social life at all?” — allows patients to raise concerns they would not spontaneously share.

"Sometimes eczema affects things like relationships and social confidence as well. Has that been an issue for you? It is absolutely something we can discuss."
💓
Mental Health — PHQ-9 7

PHQ-9 7 (mild depression) in context of eczema, sleep disruption, and occupational embarrassment. Mild depression may resolve as eczema improves — watchful waiting with NHS Talking Therapies referral. Habit reversal training (CBT for itch-scratch cycle) also addresses anxiety and low mood. If PHQ-9 ≥10 at review: formal depression management. Monitor Q9 at each visit.

"I noticed your mood score is a little low, which is not surprising given how much this has been affecting your sleep and your work. I am going to refer you to our talking therapy service — they can help with the itch-scratch cycle as well as the mood, and I think as the eczema improves your mood should improve with it."
📈
Identity and the Atopic March

Mrs. Mensah has had eczema since childhood — it is part of her identity in a complex way. She may have learned to hide it; to not discuss it; to manage alone. The consultation is an opportunity to reframe: eczema is a medical condition with effective treatments; it is not something she should simply endure. The written eczema action plan empowers self-management and reduces helplessness.

"You have been managing this for a long time. I want to give you a plan today that puts you in control — so that when the next flare comes, you know exactly what to do and you do not have to wait for a GP appointment to start treating it."
7H — Follow-up
T
Today — Prescriptions; FTU demonstrated; written action plan; referrals; safety-net

Betamethasone 0.1% ointment (arms; neck; hands — back of hands ×2/day ×14 days); hydrocortisone 1% cream (face only ×2/day ×7 days); Epaderm ointment 500g (body emollient; 4×/day); Diprobase gel 500g (daytime/work emollient; soap substitute). Skin swab (hands; nasal swab for MRSA). FTU demonstrated at consultation. Steroid phobia addressed. Written eczema action plan provided. Occupational health referral made. NHS Talking Therapies referral. POEM 16 documented. 4–6 week review booked. Eczema herpeticum safety-net given.

Review booked before Mrs. Mensah leaves; swab results expected within 5–7 days
2
1 Week — Swab results; antibiotic course review (if infection present)

If swab showed infection: is antibiotic course complete? Review hand infection status. If MRSA: specialist notification; infection control for care home. If no infection: confirm appropriate TCS use in first week; any concerns? Phone or face-to-face as appropriate.

Swab results; infection management; MRSA action if positive
3
4–6 Weeks — Main review: POEM re-score; TCS step-down; occupational health update

Face-to-face. POEM re-score: target <8 or ≥3-point reduction. TCS review: has betamethasone 0.1% been used correctly? Is it working? Step down to proactive ×2/week if in remission. Face: is hydrocortisone 1% adequate or switch to TCI? PHQ-9. Occupational health update: has appointment been made? Swab results actioned? If POEM not improved: consider patch testing referral; dermatology if POEM ≥17.

POEM re-score; TCS step-down; proactive regimen; occupational health update; PHQ-9
4
6–12 Weeks — Maintenance; proactive regimen; dermatology if not improving

If POEM improved and in remission: proactive TCS ×2/week on prone areas + daily emollient. If POEM unchanged despite optimal topical management: dermatology referral (patch testing; phototherapy; ciclosporin consideration). PHQ-9: if not improving as eczema improves: formal depression treatment. NHS Talking Therapies progress.

Proactive regimen; dermatology if failing topical; PHQ-9; NHS Talking Therapies
5
Annually — POEM; atopic comorbidities; emollient quantity; TCS review

Annual POEM. Review atopic march: asthma; allergic rhinitis — comorbidities worsen eczema. Emollient quantity check (prescription request frequency). TCS review: any signs of atrophy? Proactive regimen adherence. Psychological wellbeing. Occupational situation.

Annual POEM; atopic comorbidities; emollient quantity; TCS atrophy check
7I — Monitoring — PETS mnemonic

PETS monitoring mnemonic for eczema

POEM score: at every review — did it improve by ≥3 points? Emollient: is she getting enough (500g/week on prescription)? TCS: using correctly? Right potency for right site? No atrophy signs? Superinfection: any signs of bacterial or viral infection at each visit?

DrugMonitorTimingAction threshold
Emollient (all grades)Prescription request rate; POEM dryness Q7; application techniqueEvery reviewNot requesting refills: under-using — review adherence. POEM Q7 (dryness) not improving: emollient frequency inadequate
Mild TCS (hydrocortisone 1%)POEM Q1 (itch); face: skin atrophy; telangiectasia4–6 week reviewFace atrophy: switch to TCI. Not improving after 7 days face: switch to TCI. Not improving body after 14 days: step up to moderate-potent
Potent TCS (betamethasone 0.1%)POEM re-score; examine for TCS atrophy; striae; telangiectasia4–6 week review; then at each visit if continued proactivelyPOEM not improving despite adequate use: dermatology. TCS atrophy signs: reduce potency; switch to TCI. Skin clear: step down to proactive ×2/week
Ciclosporin (dermatology)BP; U&E; creatinine; LFTs; FBCFortnightly ×3 months; then monthlyCreatinine >25% above baseline: reduce dose. BP >160/100: reduce dose; treat. Infection: pause; specialist advice
MilestoneAction
1 weekSwab results; MRSA action; infection treatment complete; check TCS use started correctly
4–6 weeksPOEM re-score (≥3-point improvement = response); TCS step-down; proactive regimen; PHQ-9
6–12 weeksMaintenance phase; dermatology if POEM not improved; patch testing if contact allergy suspected
AnnuallyPOEM; emollient quantity; TCS atrophy check; atopic comorbidities; NHS Talking Therapies follow-up
7J — Safety-netting

⚠ Three critical safety-nets for Mrs. Mensah

🔴 Emergency — eczema herpeticum
"There is one warning sign I want you to know about. If the eczema suddenly develops lots of tiny blister-like spots — all the same size and shape — that spread rapidly, and you develop a fever and feel generally unwell — do not wait; go to A&E immediately. This is a rare but serious viral infection of the eczema skin. Do not try to treat it yourself at home. Go to A&E."
Eczema herpeticum can be life-threatening. Rapid recognition and IV aciclovir are critical. Document: “eczema herpeticum safety-net given and documented.”
💊 Superinfection — bacterial signs
"If the eczema starts producing yellow or honey-coloured crusting; weeping pus; or spreading very rapidly despite the cream — ring us. That might be a sign of a bacterial infection. We will need to look at it again and possibly prescribe an antibiotic. Don’t step up to a stronger steroid if you see those signs — bring the infection to us first."
Stepping up TCS into infected skin is harmful. Patients must know the signs of infection and know not to self-manage by increasing TCS potency.
🟠 Treatment review — POEM not improving
"If you are using the cream correctly — the right amount; in the right places; twice a day — and after 2 weeks the eczema is not improving on your arms, ring and we will review. The cream I have prescribed should make a significant difference within 2 weeks if used correctly. If it is not working, we need to look at why — it might need to be stronger; or there might be an infection we need to treat first."
A patient who uses TCS incorrectly (too little; wrong area; wrong frequency) and concludes “steroids don’t work for me” has been failed by inadequate prescribing education. The safety-net closes this loop.
999 / A&EPunched-out vesicular rash; fever; rapid spread — eczema herpeticum; do not wait
1 weekSwab results; MRSA action; infection management review
4–6 weeksPOEM re-score; TCS step-down; proactive regimen; occupational health; PHQ-9
🎓 SCA Checkpoint — Step 7 (Final)TasksRelating to OthersGlobal Skills
Closing the consultation
"Let me pull together what we have agreed. Your eczema has flared mainly because the emollient has run out and because the cream that you have been using has not been applied in a way that is effective. Both of those things are completely fixable."
"I am prescribing a larger, richer moisturiser — 500g of Epaderm ointment — which needs to go on at least 4 times a day and used instead of soap. And I am stepping up to a medium-strength cream for your arms and body for 2 weeks — I have shown you how much to use. On your face, keep to the mildest cream only."
"About the steroid worry — using the medium-strength cream on your arms for 2 weeks is safe. The risk of skin thinning only happens with long-term, daily use of a strong cream in the wrong places. Your written action plan tells you exactly when to use which cream and when to stop."
"I am also referring you to occupational health because the frequent handwashing at work is playing a big role in your hand eczema. And I am taking a swab of your hands — because occasionally we see a resistant bacteria in people who work in healthcare."
"The one important warning: if the eczema develops lots of tiny blisters all the same shape; they spread very quickly; and you feel unwell with a fever — go to A&E. Before you go — is there anything we haven’t covered?"
Deductions
  • Not demonstrating the FTU — under-application is the most common cause of TCS failure; demonstrating FTU is a core Tasks mark
  • Not addressing steroid phobia specifically — a patient who leaves with the same fear will continue to under-apply
  • Not identifying the occupational trigger (wet work; care home) — the trigger will perpetuate relapse regardless of optimal topical management
  • Prescribing potent TCS for the face — never potent TCS on face; most common TCS prescribing error
  • Stepping up TCS potency into infected skin without treating infection first
  • Not prescribing adequate emollient quantity (prescribing “a small tube of moisturiser” is not adequate treatment)
Tasks summary
  • POEM 16 documented at baseline
  • Emollient 500g/week prescribed; quantity and technique taught
  • FTU demonstrated (most commonly missed Tasks mark)
  • TCS potency appropriate: betamethasone 0.1% arms/body; hydrocortisone 1% face only
  • Steroid phobia addressed with written action plan
  • Occupational health referral; MRSA swab
  • Eczema herpeticum safety-net given
Relating to Others
  • Embarrassment at work acknowledged
  • Steroid phobia validated; then addressed with evidence
  • Occupational context acknowledged with practical plan
  • Written action plan offered as patient empowerment
  • Eczema herpeticum named specifically; not generic
🔴 Red
No FTU; steroid phobia not addressed; occupational trigger missed; potent TCS on face; TCS stepped up into infected skin; emollient not prescribed or inadequate quantity; no safety-net; POEM not scored
🟠 Amber
POEM scored; emollient prescribed; correct TCS potency by site; steroid phobia identified but not addressed with evidence; occupational trigger identified; FTU not demonstrated; no written action plan; MRSA swab not taken
🟩 Green
Embarrassment acknowledged; POEM 16 documented; emollient 500g/week prescribed + technique taught; FTU demonstrated; correct TCS by site (potent body; mild face); steroid phobia addressed with evidence + written plan; occupational trigger identified + OH referral + MRSA swab; eczema herpeticum safety-net named; PHQ-9 addressed; 4–6 week review booked; closing question
Atopic Eczema — SCA Consultation Scorecard
NICE CKS · POEM score · Emollient 500g/week · FTU demonstrated · Steroid phobia addressed · TCS site rules · Eczema herpeticum = 999 · Occupational trigger
0/ 33 pts
🌐
Global Skills
Empathy; structure; patient-centred approach
0/7
Tasks
Clinical reasoning; prescribing; management
0/15
🤝
Relating to Others
Empathy; communication; shared decision-making
0/11
RAG Self-Assessment
🔴 Red
No FTU; steroid phobia not addressed; occupational trigger missed; potent TCS on face; TCS stepped up into infected skin; emollient not prescribed adequately; no safety-net; POEM not scored; no written plan
🟠 Amber
POEM scored; emollient prescribed; correct TCS by site; steroid phobia identified but not addressed with evidence; occupational trigger identified; FTU not demonstrated; no written plan; MRSA swab not taken
🟩 Green
All: emotional opener; POEM 16; emollient 500g/week + technique; FTU demonstrated; betamethasone body/hydrocortisone face; steroid phobia + evidence + written plan; occupational trigger + OH referral + MRSA swab; herpeticum safety-net; PHQ-9 + NHS Talking Therapies; paraffin fire safety; review booked; closing question
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"I have had eczema all my life but the last 4 weeks have been terrible. My arms; neck; and hands are really bad. I have run out of my moisturiser so I have been using something from the supermarket. And I have some leftover steroid cream but I am worried about using too much of it because my mum said it damaged her skin."
Who you are

Amara Mensah, 34, healthcare assistant in a care home for the past 3 years. Known atopic eczema since early childhood. Previous mild disease mostly controlled with occasional hydrocortisone 1% and emollient. 4-week flare — worst on both arms; neck; and hands. Hands particularly bad (she washes her hands at least 30 times per day at work). Has run out of prescribed emollient 3 weeks ago; switched to “E45 from Boots” (insufficient). Using leftover hydrocortisone 1% on face and body but applying only a very thin smear (steroid phobia from mother’s experience). Embarrassed and self-conscious about her arms at work — wearing long sleeves in hot weather. POEM 16. PHQ-9 7 (low mood; not sleeping well due to itch).

Hidden agenda — disclose if GP asks specifically

Steroid phobia (disclose if GP asks about cream use or concerns): “My mum had a really bad experience with steroid cream — her skin went really thin and she got stretch marks. I don’t want that to happen to me. I’ve been putting on the thinnest layer I can. Is that OK?” Respond well if GP explains specifically when skin thinning occurs and when it doesn’t; and when FTU is demonstrated: “Oh — I had no idea you needed that much. I have been using far too little. No wonder it hasn’t been working.”

Embarrassment (volunteer if asked about impact): “I have been wearing long sleeves at work even though it’s hot. I don’t want the residents or my colleagues to see it. I feel like people are judging me.”

Work concern (disclose if asked about occupation): “I have to wash my hands so many times at day at work — the care home uses a very strong antibacterial soap. My hands are the worst part. Is that making it worse?” Respond well if GP identifies occupational trigger and refers to occupational health: “I didn’t know I could see an occupational health person for this.”

Responses to key conversations
  • On FTU demonstration: “Oh — that is so much more than I have been using. I have been using a tiny amount because of what mum said. So all this time I have been using too little?”
  • On written action plan: “This is really helpful — so I know exactly what to do next time without having to wait for an appointment? I really needed something like this.”
  • On occupational health: “I did not know I could be referred for this. Can my employer make me use different products? Because the soap at work is really strong.”
  • Challenge: “I am really worried about using the stronger cream. Can’t I just keep using the mild one but use more of it? I don’t want to risk making my skin worse.”
Clinical details
  • Age 34; 4-week bilateral arm; neck; hand eczema flare; POEM 16; PHQ-9 7
  • No fever; no rapidly spreading monomorphic vesicles (eczema herpeticum absent)
  • Hands: bilateral; dorsa and fingers; moderate erythema; some weeping on web spaces; no honey-coloured crusting (minimal infection signs — send swab; unclear on examination)
  • No skin atrophy or telangiectasia from previous TCS (using so little; no atrophy risk)
  • Known house dust mite sensitivity (has feather pillows — potential trigger)
"I am really worried about using a stronger steroid cream. My mum had skin thinning and stretch marks from steroids. Can I not just use more of the hydrocortisone on my arms instead of a stronger one? I don’t want to make it worse."

Resolution: Mrs. Mensah accepts the plan if the GP: acknowledges the steroid fear without dismissing it; explains specifically when skin thinning occurs and when it does not (2-week course of betamethasone on arms = safe); demonstrates the FTU and shows her how much cream she has actually been applying (too little); provides a written action plan; and explains that under-applying is causing the treatment failure. She leaves saying: “I had no idea I needed to use that much. I feel like I finally understand why it hasn’t been working.”

🏥
Clinic Quick Reference
Atopic Eczema — Clinical Decision Framework
NICE CKS · POEM severity · Emollient 500g/week · FTU · Steroid ladder by site · Treat infection first · Eczema herpeticum = 999 · TCI for face
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💊 1 — Severity Assessment and Action
Eczema presentation → Screen: emergency? (herpeticum; erythroderma) → Infected? → POEM score → Step treatment to severity
🔴 Emergency
  • Eczema herpeticum: punched-out vesicles; fever; rapid spread → 999; IV aciclovir
  • Erythroderma (>90% BSA) → same-day hospital
  • Sepsis from superinfection → 999
999 / same-day A&E
🟠 Urgent
  • Bacterial superinfection: fusidic acid or flucloxacillin; treat infection FIRST
  • Severe eczema POEM ≥17: urgent dermatology
Treat; review in days
🟩 GP-managed stepwise
  • Mild (POEM 3–7): emollient + hydrocortisone 1% 14 days
  • Moderate (POEM 8–16): emollient + moderate-potent TCS 14 days; FTU; written plan
  • Maintenance: emollient daily + proactive TCS ×2/week prone areas
NICE CKS stepwise; POEM review 4–6 weeks
📊 2 — Key Clinical Numbers
POEM 0–28
Patient-Oriented Eczema Measure. 3–7 mild; 8–16 moderate; 17–24 severe; 25–28 very severe. ≥3-point improvement = clinically meaningful response.
500g/week (body)
Minimum prescribed emollient for body eczema. 250g face/hands. 4×/day minimum. Apply before TCS. Soap substitute always.
FTU = 2 hands
1 fingertip unit covers 2 adult hands. Arms: 3 FTU each. Trunk front/back: 7 FTU each. Leg: 6 FTU. Demonstrate at every eczema consultation.
Herpeticum = 999
Punched-out monomorphic vesicles; fever; rapid spread; HSV superinfection. 999 / same-day A&E. IV aciclovir. Not topical antibiotic.
Face = mild TCS only
Hydrocortisone 1% maximum 7 days. Never potent TCS on face; eyelids; genitalia; skin folds. TCI (tacrolimus; pimecrolimus) if hydrocortisone 1% inadequate on face.
Treat infection first
Bacterial superinfection: Fucidin 2% 7 days or flucloxacillin 500mg QDS 7 days. Never step up TCS into infected skin — treat infection first; then TCS.
TCI: no atrophy risk
Tacrolimus 0.1% (Protopic); pimecrolimus 1% (Elidel). No skin atrophy. Face; flexures; eyelids; genitalia. Proactive ×2/week reduces relapse.
Dupilumab: NICE TA534
Adults; moderate-severe; EASI ≥16; failed ciclosporin. SC 300mg fortnightly. Not immunosuppressant. Conjunctivitis: common side effect.
Ciclosporin monitoring
Fortnightly ×3 months then monthly: BP; U&E; creatinine; LFTs; FBC. Max 1–2 years. Creatinine >25%: reduce dose. Nephrotoxicity; hypertension.
Paraffin fire risk
Paraffin-based emollients (Epaderm; Hydromol) soak into fabric; highly flammable. Warn: no smoking; open flames; candles. Document counselling.
Proactive TCS ×2/week
Apply TCS ×2/week to prone areas once eczema in remission. Prevents relapse without significant atrophy risk. Supplement with daily emollient.
MRSA: healthcare worker
Nasal swab + skin swab in healthcare workers with infected eczema. Infection control implications in care home. Specialist if MRSA confirmed.
⚠ 3 — PETS Monitoring
PETSParameterTimingAction
POEM7-item score 0–28Every review; target ≥3-point reductionNot improving at 6–8 weeks: dermatology. Worsening: consider infection; incorrect TCS potency; steroid phobia
EmollientPrescription request rate; POEM Q7 (dryness)Every reviewNot requesting refills: review adherence. Q7 not improving: frequency inadequate
TCSPOEM Q1 (itch); skin atrophy; telangiectasia; striae4–6 week review; annually if proactive regimenAtrophy: reduce potency; switch to TCI. POEM not improving: dermatology. Clear skin: step down to proactive ×2/week
SuperinfectionCrusting; weeping; pustules; rapidly spreading areaEvery visitInfection: Fucidin 7 days or flucloxacillin; treat first; then TCS. Herpeticum features: 999
🎓
SCA Exam Quick Reference
Eczema SCA — POEM · Emollient 500g · FTU demonstrated · Steroid phobia · Betnovate body / Hydrocortisone face · Herpeticum = 999 · Written plan
NICE CKS · Mrs. Mensah: occupational trigger · MRSA swab · Treat infection first · TCI for face phobia
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💬 Opening & ICE
Opener: “It sounds like the last few weeks have been really difficult — the itch at night; not sleeping; feeling self-conscious at work. Before I look at your skin, tell me what has been happening and what is worrying you most.”
ICE — Ideas: “What do you think has made this worse recently?” Links emollient running out + hand washing at work.
ICE — Concerns: “Are you worried about the steroid cream — about whether it is safe?” Then: “Skin thinning is real but only with prolonged overuse on wrong areas. A 2-week course on your arms is safe. Not treating has its own harms.”
ICE — Expectations: “Were you hoping for something that would cure it permanently?” Then: “Eczema is managed rather than cured — but very effectively. The goal is to empower you to manage flares yourself.”
Steroid phobia challenge: “Can’t I just use more hydrocortisone on my arms instead of a stronger one?” — “I understand the worry. For moderate eczema on the arms, the mild cream is not strong enough — it is like using a small fire extinguisher on a large fire. The medium-strength cream on the arms for 2 weeks is safe. I am going to show you exactly how much to use — [demonstrate FTU] — and I am giving you a written plan for when to use it and when to stop.”
Occupational: “Your work involves a lot of hand washing. That is almost certainly driving the hand eczema. I am referring you to occupational health today.”
✅ Key SCA Tasks (15pt)
Severity assessed + TCS stepped up (2pt): POEM 16 = moderate-severe; betamethasone 0.1% on arms/body (NOT hydrocortisone 1% — too weak for POEM 16 on body). Hydrocortisone 1% face only. POEM documented at baseline.
Emollient 500g/week + technique (2pt): Epaderm ointment 500g; Diprobase gel 500g. 4×/day; soap substitute; apply before TCS; direction of hair growth. Paraffin fire safety documented.
Infection assessed; treat first (2pt): Examine for S. aureus: honey crusting; weeping. If present: Fucidin 2% ×7 days BEFORE TCS step-up. NEVER step up TCS into infected skin. Healthcare worker: MRSA nasal + skin swab.
Occupational trigger + OH referral + MRSA (2pt): Wet work; soap; rubber gloves at work identified as trigger. Occupational health referral today. MRSA nasal swab + skin swab (patient safety). Interim hand protocol prescribed.
FTU demonstrated at consultation (2pt): “Squeeze from tube to first knuckle — 1 FTU. Covers 2 hands. Arms: 3 FTU each.” Physical demonstration. Most commonly missed Tasks mark in eczema SCA. Without FTU: under-application continues; eczema fails to respond.
Eczema herpeticum safety-net (1pt): Named; features (punched-out; fever; rapid); action (A&E); documented.
Written eczema action plan (1pt): Maintenance; flare step-up; step-down; emergency signs. NICE CKS recommendation.
Face restriction documented (1pt): No potent TCS on face; hydrocortisone 1% face only; TCI if fails.
4–6 week review booked (1pt): POEM re-score; target ≥3-point reduction; TCS step-down; OH update.
Paraffin fire safety (1pt): “No smoking near open flames with paraffin-based emollient” — documented.
🔴 Potent TCS on face = automatic Tasks fail
🔴 TCS stepped up into infected skin without treating infection first = Tasks fail
👥 Relating to Others (11pt)
Embarrassment acknowledged (1pt): Self-consciousness at work; long sleeves in hot weather; both named
ICE: Ideas — trigger identified (1pt): Emollient running out + wet work at work — both validated
ICE: Concerns — steroid phobia validated + evidence (1pt): Mother’s experience acknowledged; specific reassurance with FTU; written plan
ICE: Expectations — prognosis honest (1pt): Management not cure; empowerment via written plan
Steroid phobia addressed with evidence (1pt): When atrophy occurs; when it does not; 2-week arm course safe; written plan as boundary
Occupational context acknowledged + practical (1pt): Occupational health + interim hand protocol + reasonable adjustments right
Emollient as treatment not moisturiser (1pt): Most important treatment; barrier function; 4×/day; before TCS
Written plan as patient empowerment (1pt): “Self-manage next flare without GP appointment”
PHQ-9 7 + NHS Talking Therapies (1pt): Low mood acknowledged; linked to eczema improvement; NHS Talking Therapies for habit reversal + mood
Herpeticum named specifically (1pt): “Eczema herpeticum” named; features specific; A&E not GP; not generic “if worse”
Closing question + pause (1pt): Real 3–5 second pause; swimming; children’s skin; workplace queries — all have good answers
🟩 Mrs. Mensah leaves: “I had no idea I needed to use that much cream. I finally understand why it hasn’t been working. The plan really helps — now I know exactly what to do.”
💊 Treatment Quick-Pick by Scenario
Reviewed: July 2026 · citations verified against current NICE / UK guidance