Respiratory · Full case

Asthma

NICE NG245 BTS-SIGN 2024 CKS 2026 📄 Patient leaflets
AST
Asthma · GP Clinical Reasoning Framework
GP & SCA · NICE/BTS/SIGN NG245 (2024) · GINA 2025
FEV1/FVC <0.7Airflow obstruction on spirometry
≥12% + 200mLReversibility = positive bronchodilator test
SABA >3×/weekPoor control — step up treatment
2+ courses OCS/yrSevere/uncontrolled — refer to specialist
SpO2 <92%Acute severe — target 94–98%
PEFR <50%Acute severe attack threshold
AIR first-lineLow-dose ICS/formoterol PRN from diagnosis — no SABA-only (NG245)
Annual reviewCheck technique, adherence, trigger control
📋 Clinical Stem — Asthma Presentation
A patient with wheeze, breathlessness, and chest tightness — or known asthma for annual review
"Miss Priya Sharma, 28, attends with a 6-week history of episodic wheeze and chest tightness, worse at night and on exercise. She works as a primary school teacher. She has a cat at home that her partner bought recently. Her mother has asthma. She has been using a salbutamol inhaler she borrowed from her flatmate almost daily and is worried about her breathing at work."
Asthma affects 5.4 million people in the UK — 1 in 11 adults. Around three people die from asthma every day in the UK, and the National Review of Asthma Deaths found potentially avoidable factors in most of them. The same pathway applies to new diagnosis, annual review, poor control, and acute exacerbation. The key questions: (1) is this really asthma? (2) is it controlled? (3) what is preventing control?
Scenario A — New diagnosis (SCA)28-year-old teacher. Nocturnal wheeze + exercise dyspnoea. New cat. Daily SABA. Wants to know if it is asthma and whether she needs to get rid of the cat.
Scenario B — Poor control reviewKnown asthma 5 years. Using salbutamol daily. "The brown inhaler makes me feel worse." Does not use preventer. BMI 32. Smoker. No asthma plan.
Scenario C — Acute exacerbationAcute wheeze + breathlessness after URTI. PEFR 55% predicted. SpO2 94%. Tachycardic 108. Increasing SABA. Already had 2 OCS courses this year.
Scenario D — Occupational asthmaBaker presenting with new onset wheeze, 35 years old. Symptoms worse Mon–Fri and on shift, better weekends and holidays. Previous normal lung function.
Scenario E — Difficult/severe asthma3+ OCS courses/year. Using ICS/LABA + LTRA. Eosinophil count 0.45. BMI 34. Possible referral for biologic therapy (mepolizumab).
Key variablesFrequency of SABA use · Nocturnal symptoms · FEV1/PEFR · Trigger identification · Inhaler technique · Adherence · Occupation · Smoking status · Atopy
Your character

Priya Sharma, 28. Primary school teacher. You have had worsening wheeze and chest tightness for 6 weeks, mainly at night and when running. You borrowed your flatmate's blue inhaler and have been using it almost every day — it helps but wears off. Your partner bought a cat 2 months ago. Your mum has asthma. You are worried about your job — you cannot afford to be breathless in class.

ICE — open only if asked
  • Ideas: You think it might be asthma like your mum but hope it is "just a virus." You have heard inhalers are addictive and make lungs lazy.
  • Concerns: You are terrified the doctor will tell you to get rid of the cat. Your partner loves it and it would cause a major argument. Also worried about having to take time off work.
  • Expectations: Find out what is causing the wheeze. Get your own inhaler. Clarify whether you can keep the cat. Know when you need to worry enough to go to hospital.
"I've been wheezing for about 6 weeks now — mainly at night and when I try to go running. I've been borrowing my flatmate's blue inhaler and it does help, but I'm getting through it really quickly. My mum has asthma and I'm worried this might be the same thing. The main thing I wanted to ask about — my partner got a cat a couple of months ago and I'm worried you're going to say I have to get rid of it."
Resist agreeing to remove the cat unless the doctor acknowledges how difficult that would be and offers alternatives first (HEPA filter, bedroom exclusion zone, immunotherapy discussion). Become anxious if told you need hospital today — you have a class to teach. Agree to inhaler treatment only once the "inhalers make lungs lazy" myth has been explicitly addressed. Volunteer the cat is sleeping on your bed only if asked about where the cat spends time.
Steps:
1
Step 1
History Taking — Open Question First · Symptoms · ICE · Psychosocial Context
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Two parallel agendas in asthma history: the clinical (is this asthma? how severe? what is driving poor control?) and the patient's (will I have to give up my cat? are inhalers safe long-term? will this affect my job?). Both must be explored. The open question is the most efficient tool — in one response Priya reveals trigger fear, inhaler myths, and occupational anxiety simultaneously.
🎓 SCA opener — use what you already know
"I can see from your notes that you've been having trouble with your breathing for the past few weeks. Before I go through everything, I'd like to hear from you first — what's been the most worrying part of this for you?"
Acknowledge the presenting problem from the notes. Then open the floor. A single narrative response from Priya will reveal the cat concern, the borrowed inhaler, the occupational anxiety, and the inhaler myth — all ICE elements in one reply.
1A — Open question first, then targeted history
Question to askWhy it matters clinicallyChanges what?
🟢 OPEN QUESTION — always start here"Tell me in your own words what's been happening with your breathing — and what's been worrying you most about it?" Surfaces the hidden agenda immediately. Patients rarely volunteer: trigger fears (cat), inhaler myths ("they make lungs lazy"), occupational anxiety, or hospital fear unless given space. Priya's opening response typically reveals all three ICE elements. Rigid symptom-by-symptom questioning misses these and overruns data gathering time.In SCA: the first minute is scored for open questioning and note use. Both are lost if you dive straight into "Do you have wheeze?" Reveals hidden agendaICE in one response
Symptom characterisation"Tell me about the wheeze and breathlessness — when does it happen, how often, and how does it affect you?" Classic asthma triad: wheeze, breathlessness, chest tightness — episodic, variable, worse at night and with triggers. Nocturnal symptoms are highly predictive of asthma. Quantify: how many nights/week? Can she complete a sentence? PEFR variability >20% supports diagnosis.BTS/SIGN: nocturnal symptoms + response to bronchodilator = strong diagnostic indicators. Diagnosis probabilityPEFR diary needed
Trigger identification"Have you noticed anything that makes it worse — exercise, cold air, dust, animals, stress, infections?" Asthma triggers: allergens (cat/dog dander, house dust mite, pollen), viral URTIs, exercise, cold air, NSAIDs/aspirin, beta-blockers, occupation, strong emotions, menstrual cycle. Identifying the trigger is the single most modifiable management lever. Cat introduced 2 months before symptoms = likely sensitisation.Cat allergen persists on clothing, furniture, and air for months after removal. Bedroom exclusion is the minimum; removal is optimal. Trigger avoidance planOccupational asthma?
Occupation and pattern"Tell me about your work. Does your breathing get better on days off or on holidays?" Occupational asthma: symptoms worse during work, better on weekends/holidays. Teachers exposed to chalk dust, cleaning products. Bakers (flour), hairdressers (bleach), healthcare workers (latex). SWORD scheme — report if suspected. Occupational asthma: once sensitised, further exposure causes progressive deterioration even at low levels.Early diagnosis + removal from exposure = best prognosis. Delayed diagnosis = permanent airways disease. Occupational asthmaSpecialist + SWORD
Current inhaler use"Are you using any inhalers at the moment? Which one and how often? Can you show me how you use it?" SABA use is the single best marker of asthma control. Using >3 times/week = poor control = step up. Daily SABA use = uncontrolled = urgent review. Over-reliance on SABA without preventer = major risk factor for fatal attack. Inhaler technique: a large proportion of patients use their inhaler incorrectly — check it with the device in hand rather than assuming. Wrong technique negates the treatment.BTS/SIGN 2023: SABA over-prescription is independently associated with asthma mortality. Prescribers should consider reducing SABA reliance as a quality indicator. Step on NG245 pathwayTechnique check mandatory
Atopic history and FH"Do you have or have you ever had eczema, hay fever, or food allergies? Does asthma or atopy run in your family?" Atopic triad: asthma + eczema + allergic rhinitis — co-occurrence is common. Personal or family history of atopy significantly increases asthma probability. Mother with asthma in Priya's case raises both genetic risk and the risk that Priya's management knowledge is based on her mother's experience (which may be outdated or wrong).Allergic rhinitis coexists in a large majority of asthma patients — treating it improves asthma control. Ask about nasal symptoms at every asthma review. Atopic asthma confirmedAdd nasal steroid?
Drug history — precipitants"Do you take any aspirin, ibuprofen, or beta-blockers regularly? Any eye drops for glaucoma?" NSAIDs/aspirin: precipitate bronchoconstriction in ~10% of asthma patients (aspirin-exacerbated respiratory disease, AERD). Beta-blockers (including eye drops — timolol): cause bronchoconstriction and can worsen asthma significantly. ACE inhibitors: cause dry cough in 10–15% — often confused with asthma. Identifying these changes management immediately.Timolol eye drops for glaucoma are a classic missed cause of worsening asthma in older patients. Drug-induced wheezeStop precipitant
1B — Red flags: act before continuing history
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Red Flags in Asthma — Features Requiring Immediate Action

Red flagWhy dangerousAction
Cannot complete sentences · SpO2 <92% · Respiratory rate >25/min · HR >110Acute severe asthma — PEFR 33–50% predicted. Hypoxia is a late sign. Silent chest = impending respiratory arrest. Agitation or confusion = type 1 respiratory failure.999 immediately
Silent chest on auscultation in acute attackNo wheeze heard = so little air movement that wheeze has disappeared. Paradoxically worse sign than audible wheeze. Near-fatal attack imminent.999 immediately
Cyanosis · Bradycardia · Exhaustion · Altered consciousnessLife-threatening asthma — PEFR <33% predicted. Paradoxical breathing, hypercapnia, impending arrest. Requires ICU-level care.999 immediately
Haemoptysis · Unilateral wheeze · Unexplained weight loss · Stridor · Age >40 new onsetThese features are NOT typical of asthma. Unilateral wheeze = foreign body or tumour. Haemoptysis = malignancy, TB, PE. Stridor = upper airway obstruction. Must not be labelled as asthma without excluding these.Urgent CXR + 2-week wait
Previous near-fatal attack (ITU/intubation) or hospital admission for asthmaHistory of life-threatening attack is the strongest predictor of a future near-fatal attack. These patients need specialist input, written asthma action plan, and low threshold for hospital admission in any acute episode.Specialist referral + written plan
Pneumothorax in acute wheeze: sudden unilateral pleuritic pain + tracheal deviationSpontaneous pneumothorax can complicate acute asthma. Tracheal deviation to the opposite side = tension pneumothorax = immediate threat to life. Do NOT give pressurised O2 without identifying this first.999 — tension PTX
🛡️

Safeguarding Considerations — Consider in Every Asthma Consultation

Asthma is the most common chronic disease of childhood — and a marker of multiple social vulnerabilities. Poorly controlled asthma in a child or adult can reflect neglect, adverse housing, poverty, or carer capacity issues. Always consider the wider context beyond the inhaler prescription.
🧒 Children — Neglect and Non-Adherence
  • Repeated A&E admissions for asthma in a child = safeguarding concern until proven otherwise
  • Prescriptions never collected, inhalers never used, no school inhaler — is the parent able to manage?
  • Passive smoke exposure in children: major modifiable risk factor. Document and act on it.
  • Mould and damp housing — local authority housing duty; refer to housing officer if needed
  • Child unable to self-manage at school despite good parental understanding — IEP/school health referral
🏠 Housing and Environmental Risk
  • Mould, damp, overcrowding, cockroaches = major asthma triggers — housing is a health determinant
  • Patients who cannot move out of triggering environments (housing insecurity, poverty, domestic entrapment) need social prescribing, not just a prescription
  • Domestic abuse: high stress and sympathetic activation worsens asthma control independently of triggers
  • Partner who controls housing situation may make trigger avoidance impossible (e.g. partner's smoking, keeping allergen-producing pet)
💊 Mental Health and Self-Harm Risk
  • Anxiety and asthma coexist and amplify each other — panic attacks mimic acute asthma and vice versa
  • Oral corticosteroid courses: mood change, agitation, hypomania — document and screen
  • Patients deliberately over-using or under-using inhalers as self-harm behaviour — rare but documented
  • PHQ-9 and GAD-7 at diagnosis and annual review — depression and anxiety independently worsen asthma control and mortality
👩‍💼 Occupational Risk and Coercion
  • Occupational asthma: employer may be reluctant to accommodate workplace adjustments — patient may face job loss
  • Document fitness-for-work advice clearly; patients need written evidence for HR/occupational health
  • Insecure employment may cause patients to deny symptoms or avoid sick leave — impacts safety at work
  • Food industry, healthcare, and cleaning sector workers: highest risk occupational groups; young and migrant workers over-represented
If a safeguarding concern is identified: You do not need certainty — a concern is sufficient to act. Document clearly. In children: consider referral to children's services if parental capacity is in question. In adults: refer to adult safeguarding if abuse or neglect is identified. Address housing issues via social prescribing and local authority routes. Do not let the asthma management agenda override immediate safety.
1C — PMH · FH · Drug history · Social history
🧬 PMH / FH — changes management
FactorWhy it mattersManagement impact
Atopic disease (eczema, hay fever, food allergy)Atopic triad confirms allergic asthma phenotype. Co-existing allergic rhinitis worsens asthma — "one airway, one disease." Treating rhinitis improves asthma control.Add intranasal corticosteroid. Consider allergy testing. SLIT/SCIT immunotherapy if severe allergic asthma.
Previous near-fatal attack / ITU admissionStrongest single predictor of future near-fatal attack. PEFR at baseline may be misleadingly normal between attacks.Specialist referral mandatory. Written asthma action plan. Low admission threshold. Emergency kit at home.
GORD (gastro-oesophageal reflux)Acid reflux triggers vagal-mediated bronchoconstriction and microaspiration. A common, missed cause of difficult-to-control asthma.Trial PPI therapy. Lifestyle modification. Spirometry post-PPI to assess response.
Obesity (BMI >30)Obesity worsens asthma via reduced lung volumes, altered airway mechanics, and pro-inflammatory adipokines. Weight loss of 5–10% can improve symptom control significantly.Weight management programme. SGLT2i if T2DM coexists. Reassess step before stepping up.
PregnancyAsthma control can worsen in pregnancy (especially second trimester). Undertreated asthma carries greater foetal risk than ICS use. Do not step down treatment in pregnancy.Continue ICS — do not stop. Budesonide preferred (most safety data). Monthly review.
Aspirin-exacerbated respiratory disease (AERD)Aspirin, ibuprofen, and NSAIDs precipitate severe bronchoconstriction in ~10% of asthmatic adults. Often missed. Nasal polyps + asthma + aspirin sensitivity = Samter's triad.Avoid all NSAIDs. Paracetamol for analgesia. Add LTRA (montelukast) — particularly effective in AERD.
💊 Drug history · Social history — critical interactions
FactorWhy it mattersManagement impact
Beta-blockers (incl. eye drops: timolol)Block beta-2 receptors in airways → bronchoconstriction. Even topical beta-blocker eye drops for glaucoma can precipitate severe asthma. A commonly missed cause of worsening asthma.STOP oral/topical beta-blockers. Switch to CCB for hypertension. Use prostaglandin analogue eye drops for glaucoma.
NSAIDs / aspirinAERD affects ~10% of asthmatics. Aspirin and NSAIDs inhibit COX-1 → shunts arachidonic acid to LTC4 → bronchoconstriction. Risk is dose-dependent.STOP NSAIDs. Paracetamol or COX-2 inhibitor. Add montelukast. Document allergy clearly in records.
ACE inhibitorsDry cough in 10–15% of patients taking ACEi. Caused by bradykinin accumulation. Often confused with asthma — but is not bronchoconstriction. Can exacerbate underlying asthma.Switch to ARB (no cough). Reassess asthma control after switch to confirm true underlying asthma vs drug-induced cough.
Smoking (active or passive)Reduces ICS efficacy by inducing steroid-resistant inflammation (neutrophilic, not eosinophilic). Smokers with asthma are profoundly underserved by standard ICS therapy. Stepping up ICS dose without addressing smoking is less effective.Smoking cessation is the highest-impact intervention. QUIT referral. Consider high-dose ICS + theophylline. Refer if smoking-related COPD overlap (ACO).
Housing: damp, mould, pets, carpetsHouse dust mite (HDM): major allergen, survives in humid environments and soft furnishings. Mould spores: trigger severe asthma exacerbations. Cat allergen: extremely persistent, airborne for hours.HDM: mattress covers, dehumidifier, frequent washing. Cat: bedroom exclusion, HEPA filter, discuss rehoming sensitively. Mould: housing referral.
Exercise habitsExercise-induced bronchoconstriction (EIB) affects 70–80% of asthma patients. Well-controlled asthma should not significantly limit exercise. Persistent EIB = poor control or cold-air/dry-air exposure during sport.Pre-exercise SABA 15 min before. Warm-up protocol. Nasal breathing. Consider LTRA. If ongoing = step up or investigate alternative diagnoses (EILO).
1D — ICE: Ideas · Concerns · Expectations
💡 Why ICE is critical in asthma

Priya has three overlapping concerns that, if unaddressed, will prevent treatment adherence: (1) the "inhalers make lungs lazy" myth — an almost universal belief that leads to under-use of preventers; (2) the cat — the most emotionally charged trigger conversation in any asthma consultation; (3) occupational impact — a teacher cannot be breathless in front of a class. If you prescribe without addressing these, the patient will not use the inhaler correctly or consistently.

💭 Ideas
"What do you think has been causing this? When you hear the word 'asthma' — what does that bring to mind for you?"
Common misconceptions: inhalers are addictive; ICS causes weight gain; asthma means disability; it will resolve on its own. The "inhalers make lungs lazy" myth is near-universal and must be explicitly corrected before prescribing — otherwise the preventer inhaler will sit unused.
😟 Concerns
"What's been worrying you most? And what do you think we might say about the cat — how would that affect things at home?"
Two layered concerns: (1) the cat — an emotionally charged trigger that directly affects a relationship; ordering its removal without acknowledgement will destroy the therapeutic relationship. (2) Work — a teacher cannot manage a classroom while wheezing. Both must be addressed with specific plans, not generic advice.
🎯 Expectations
"What were you hoping we could sort out today — and what would make this appointment feel useful?"
Priya expects: a diagnosis, her own inhaler, cat guidance, and a clear threshold for seeking emergency help. Address expectation BEFORE the management plan — this is an explicit SCA scoring criterion. Patient who leaves without clarity on the cat will not engage with trigger management.
1E — Psychosocial context: the person living with asthma
🫂 Asthma affects identity, relationships, and how people see themselves as capable adults

For Priya, asthma is not just a respiratory condition — it threatens her professional identity (teacher who cannot speak clearly), her relationship (disagreement over the cat), and her independence (fear of needing emergency help). Addressing only the lung function misses everything that determines whether the management plan will actually be followed.

👩‍🏫 Teaching career

A teacher's voice and breathing are professional tools. Daily wheeze impairs classroom effectiveness and causes anxiety about professional capability. Uncontrolled asthma affects sick days and may trigger occupational health assessment.

"With good asthma control, most people can teach, exercise, and do everything they did before — the goal is to get your breathing to the point where it doesn't hold you back at work."
🐱 The cat

The most sensitive trigger conversation in asthma. Rehoming a pet causes genuine distress and relationship conflict. A graduated approach — bedroom exclusion, HEPA filter, regular vacuuming, allergy testing — allows a shared decision rather than a dictated one. Immediate full rehoming advice often leads to total rejection of the consultation.

"I'm not going to tell you the cat has to go today. Let's look at what we can do step by step — there are ways to reduce your exposure significantly while we figure out how much it's contributing."
💊 Inhaler myths

"Inhalers make the lungs lazy" is believed by the majority of patients starting preventer therapy. This directly causes non-adherence. The myth must be explicitly named and debunked — not ignored. ICS does not reduce the lungs' ability to breathe; it reduces inflammation that is preventing them from doing so.

"The brown inhaler doesn't do the breathing for you — it reduces the inflammation that's been narrowing your airways. Your lungs don't become dependent on it. They become less inflamed."
🧠 Anxiety and asthma

Anxiety and asthma have a bidirectional relationship: anxiety triggers bronchoconstriction and breathlessness triggers panic. Up to 30% of patients with "difficult asthma" have panic disorder or dysfunctional breathing as the primary driver. PHQ-9 and GAD-7 at diagnosis and every review.

"Sometimes anxiety and breathing problems get intertwined and feed each other. How has the breathlessness been affecting your mood and your worry levels?"
🏃 Exercise and identity

Asthma patients frequently reduce activity to avoid breathlessness — this worsens fitness, increases obesity risk, and reinforces the belief that they "can't" exercise. Exercise-induced asthma is very well managed with pre-exercise SABA. Restoring activity is a core goal.

"Exercise-induced symptoms are very common in asthma and very treatable — you should not have to give up running. A puff of the blue inhaler 15 minutes before exercise can make a huge difference."
👨‍👩‍👧 Family influence

Mother with asthma may have given Priya accurate or outdated beliefs about asthma management. Ask what her mother uses and believes. Family modelling affects adherence — if mum never uses a preventer, Priya probably won't either. Explore to correct, not to contradict.

"You mentioned your mum has asthma — what does she use for it? That might tell me what you've grown up thinking is normal for asthma management."
🎓 SCA Checkpoint — Step 1TasksRelating to OthersGlobal Skills
Key phrases that score
"I can see from your notes you've been struggling with your breathing. Before I go through everything, what's been the most worrying part of this for you?"
"You mentioned your partner has a cat — what would it mean for things at home if I said the cat was making this worse?"
"What do you know about the inhalers — have you had any worries about using them long-term?"
"How is this affecting your work as a teacher?"
Deductions
  • Asking "do you have wheeze?" when it is clearly documented in the notes
  • Going straight to prescribing without exploring the cat concern and inhaler myths
  • Telling Priya to get rid of the cat without any acknowledgement of the relationship impact
  • Not asking about occupation — teacher context shapes the entire management plan
  • Missing SABA over-use as the central marker of poor control
🔴 Red
Asks for symptoms already in notes · No open Q · Cat not explored · Inhaler myths not addressed · Goes straight to prescribing
🟠 Amber
Open Q attempted then rigid · Cat mentioned but not explored emotionally · SABA use noted not quantified · Occupation not linked to management
🟢 Green
Notes used first · Open Q yields full ICE · Cat addressed with sensitivity and graduated plan · Inhaler myths explicitly debunked · SABA frequency quantified · Occupation explored · History complete by 6–7 min
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Step 2
Triage Engine — Emergency · Urgent · Routine
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Asthma triage is severity-first, aetiology-second. The first question is never "what step of the BTS ladder?" — it is "is this patient safe right now?" A silent chest in an asthma consultation is a life-threatening emergency. PEFR and SpO2 must be known before any management decision is made in an acute presentation.
🔴 Emergency — 999

Life-Threatening / Near-Fatal

999 now
  • Silent chest or absent breath soundsNo wheeze = no air movement. Near-fatal attack. ICU required.
  • SpO2 <92% / cyanosisHypoxia is a late sign — already significant respiratory failure
  • Cannot complete sentences · exhaustionPEFR <33% predicted — life-threatening attack
  • Bradycardia · hypotension · altered consciousnessRespiratory arrest imminent — immediate intervention required
  • Tension pneumothorax featuresSudden pleuritic pain + tracheal deviation + unilateral absent breath sounds
🟠 Urgent — same day

Acute Severe — Hospital Assessment

Same day
  • PEFR 33–50% predictedAcute severe attack — nebulised salbutamol + oral prednisolone + O2
  • Unable to complete a sentence at restEven if SpO2 acceptable — ongoing severe attack
  • HR >110 or RR >25Tachycardia + tachypnoea in acute attack = severe
  • Attack not responding after 15–30 min SABAIf no improvement with repeated bronchodilators — hospital
  • Previous near-fatal attack with new acute episodeHigh-risk patient — lower admission threshold regardless of PEFR
🟢 Routine — GP management

Stable / Chronic Management

Planned care
  • New diagnosis of asthmaSpirometry + reversibility ± FeNO · Start AIR (low-dose ICS/formoterol PRN, NG245)
  • Annual asthma reviewControl assessment · Inhaler technique · Triggers · Written plan · Step review
  • Poor control (reliever >3×/week)Step up the NG245 pathway · Check technique and adherence before stepping up
  • Mild acute exacerbation (PEFR >50%)Increase SABA · 40–50 mg prednisolone × 5 days · Review in 48h
🎓 SCA Checkpoint — Step 2TasksGlobal Skills
Safety screen — verbalise before management
"Before we go through the plan I want to check a few things — are you breathless right now sitting here? Can you tell me your PEFR — have you measured your peak flow today? Any chest pain or feeling very unwell?"
Deductions
  • Going straight to treatment plan without assessing current severity
  • Missing PEFR or SpO2 in any acute or subacute asthma presentation
  • Not considering silent chest as a warning sign (mistaking absence of wheeze for improvement)
🔴 Red
No PEFR or SpO2 check · Silent chest not recognised · Goes to management without safety screen · Does not admit acute severe attack
🟠 Amber
PEFR checked but not interpreted correctly · Safety screen done but silently · Triage decision delayed · Previous near-fatal history not explored
🟢 Green
PEFR + SpO2 + RR + HR assessed · Silent chest sign known and acted on · Triage category assigned and explained · Previous ITU admissions asked about and acted upon
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Step 3
Do I Need This Examination?
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"Will this examination change management?" Every asthma examination finding must link to a clinical decision. In chronic asthma, the examination establishes baseline and excludes alternative diagnoses. In acute asthma, the examination determines immediate urgency. Explaining each finding to Priya is a domain 3 opportunity.
ExaminationWhy it matters in asthmaWhat finding changes managementChanges management?
PEFR (peak expiratory flow rate)The single most important bedside measurement in asthma. Both diagnostic (variability >20% supports diagnosis) and severity assessment (percentage of predicted).PEFR diary ×2/day for 2–4 weeks: variability >20% = confirmatory for asthma. Essential in occupational asthma diagnosis.<50% = acute severe → hospital. <33% = life-threatening → 999. Variability >20% = confirms asthma.YES — always
Chest auscultation — bilateral wheezeWheeze is expiratory and widespread in asthma. Unilateral wheeze = foreign body or tumour — not asthma. Silent chest in acute attack = near-fatal. Coarse crackles + wheeze = infective exacerbation.Inspiratory stridor ≠ asthma — upper airway obstruction, vocal cord dysfunction, or epiglottitis. A critical diagnostic distinction.Unilateral wheeze → urgent CXR. Silent chest → 999. Stridor → 999.YES — urgency
Respiratory rate, HR, SpO2Tachypnoea (>25) + tachycardia (>110) + SpO2 <92% = acute severe asthma. SpO2 is a late sign — patients can be severely ill with maintained SpO2 until very late. Target 94–98% O2 in acute attack (high-flow oxygen can cause hypercapnia).Pulsus paradoxus (>10 mmHg BP drop on inspiration) = severe asthma — rarely measured in primary care but important to know.SpO2 <92% → 999. RR >25 + HR >110 = acute severe → hospital. Target O2 94–98%.YES — urgency
Nasal examination (nasal polyps, turbinate swelling)Allergic rhinitis in 60–80% of asthma patients — "united airway disease." Nasal polyps: if bilateral + asthma + aspirin sensitivity = Samter's triad. Untreated rhinitis drives persistent airways inflammation and worsens asthma control.Treating co-existing allergic rhinitis reduces asthma exacerbations, SABA use, and hospitalisations — evidence from multiple RCTs.Nasal polyps → ENT referral. Turbinate swelling → add intranasal corticosteroid.YES — add-on therapy
Skin — eczema, urticariaConfirms atopic phenotype. Active eczema = ongoing allergic inflammation = likely driving asthma. Chronic urticaria: may indicate mast cell activation disorder — affects inhaler selection and need for allergy referral.Active atopic eczema → allergen avoidance strategy. Dermatology co-management if severe.YES — phenotype
BMI + general appearanceObesity worsens asthma: reduces FRC, alters airway mechanics, promotes neutrophilic (steroid-resistant) inflammation. Assess for Cushingoid features if on high-dose or frequent OCS courses.Obese asthma: weight loss 5–10% can reduce exacerbations and steroid courses significantly. Consider bariatric referral in severe asthma with obesity.BMI >35 → weight management referral before stepping up. Cushingoid features → review OCS exposure.YES — step decision
🎓 SCA Checkpoint — Step 3Tasks
How to propose examination
"I'd like to measure your peak flow first — that gives us an instant picture of how open your airways are right now. Then I want to listen to your chest, check your oxygen level, and have a look at your nose, because in asthma the nose and lungs are connected and treating both makes a real difference."
Deductions
  • Not measuring PEFR at any asthma presentation — mandatory
  • Not explaining why each examination is being done — missed domain 3 opportunity
  • Not examining the nose in a patient with likely allergic asthma
  • Treating unilateral wheeze as if it were asthma without investigating
🔴 Red
No PEFR · No SpO2 · No chest auscultation · Examination not explained · Unilateral wheeze not investigated
🟠 Amber
PEFR + chest done · SpO2 omitted · Nasal exam not done · Examination not linked to management decision
🟢 Green
PEFR + SpO2 + RR + HR + chest auscultation + nasal exam · Each examination explained with rationale · Finding linked to specific management change · Inhaler technique checked during consultation
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Step 4
Do I Need This Investigation?
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Spirometry with reversibility testing is the gold-standard investigation for asthma diagnosis. Every investigation must answer a specific clinical question. PEFR diaries, FeNO, blood eosinophils, and allergy testing each provide distinct information that changes management in specific phenotypes. Explain what each test is for — it is a domain 3 scoring opportunity.
InvestigationClinical question it answersWhat result changes management?
Spirometry + reversibility (post-bronchodilator)Confirms obstructive pattern and reversibility — the diagnostic cornerstone. FEV1/FVC <0.7 = obstruction. ≥12% improvement + ≥200mL in FEV1 after bronchodilator = positive reversibility = asthma. Must be done when symptomatic for best yield. In over-35s with smoking history: distinguish from COPD or overlap (ACO).Positive reversibility → confirms asthma. No reversibility → consider COPD, VCD, ICS trial. FEV1 <70% predicted at baseline = moderate–severe asthma → specialist input.
PEFR diary (twice daily × 2–4 weeks)Diurnal variability >20% = confirmatory for asthma. Essential for occupational asthma diagnosis (worse on work days, better on days off). Cheaper and more accessible than spirometry. The pattern of variability is as important as the absolute value.BTS/SIGN: PEFR variability = (max–min)/max × 100. >20% supports diagnosis.>20% variability → confirms asthma diagnosis. Occupational pattern (worse Mon–Fri) → occupational asthma pathway + SWORD reporting.
FeNO (fractional exhaled nitric oxide)FeNO is a non-invasive biomarker of eosinophilic airway inflammation, and the FIRST objective test in the NG245 sequence. ≥50 ppb (adults 17+) or ≥35 ppb (ages 12–16) = asthma confirmed — the 40 ppb / 25–39 ppb bands belong to the superseded NG80. A result below the threshold does NOT exclude asthma (smoking lowers FeNO) → go to spirometry with reversibility, then PEF variability. A low FeNO → less likely to respond to ICS alone (consider other diagnoses or non-eosinophilic asthma). Useful when diagnosis is uncertain or to guide ICS stepping.NICE NG245: FeNO recommended for asthma diagnosis alongside spirometry and PEFR.FeNO ≥50 ppb (adults 17+) or ≥35 ppb (ages 12–16) → asthma confirmed (NG245), ICS responsive. Below threshold → does not exclude asthma; next test is spirometry with reversibility, then PEF variability. A persistently low FeNO with poor control → consider VCD, hyperventilation, non-eosinophilic asthma or adherence.
Full blood count — eosinophil countBlood eosinophils ≥0.3 × 10⁹/L = eosinophilic asthma phenotype — predicts ICS response and eligibility for biologic therapies (mepolizumab, benralizumab). High eosinophils with poor control = step up ICS or refer for biologic. Eosinophilia + pulmonary infiltrates = consider EGPA (Churg–Strauss).Eligibility for mepolizumab/benralizumab: blood eosinophils ≥0.3 × 10⁹/L + ≥4 exacerbations/year or OCS-dependent.Eosinophils ≥0.3 → ICS-responsive phenotype. ≥0.3 + poor control on high-dose ICS/LABA → biologic eligibility referral.
IgE (total and specific — RAST/skin prick)Elevated total IgE confirms atopic phenotype. Specific IgE to cat, HDM, grass, mould identifies the causative allergen. Helps focus trigger avoidance — telling a patient to avoid "allergens" is less useful than naming the specific sensitisation. Eligibility for omalizumab (anti-IgE biologic): IgE 30–1500 IU/mL + confirmed allergen sensitisation.Omalizumab eligibility: severe allergic asthma + IgE 30–1500 IU/mL + positive skin prick or specific IgE + ≥4 exacerbations/year.Cat-specific IgE positive → cat is confirmed trigger → graduated rehoming advice. Eligibility for omalizumab if IgE within range + criteria met.
CXR (chest X-ray)Asthma does not cause CXR abnormalities — if it is abnormal, consider an alternative diagnosis. CXR indicated: first presentation to exclude pneumothorax, malignancy, TB; acute severe attack to exclude pneumothorax and infection; if wheeze is unilateral; if response to treatment is poor.CXR in asthma: hyperinflation with peri-bronchial thickening may be seen but is non-specific. A normal CXR does not exclude asthma.Unilateral opacity → tumour, foreign body. Pneumothorax → immediate management. Normal CXR + wheeze → supports asthma (by exclusion of alternatives).
Allergy testing (skin prick or ImmunoCAP)Identifies specific sensitisations to guide avoidance and eligibility for immunotherapy (SLIT/SCIT). Cat dander, HDM, grass, tree pollen, mould. In occupational asthma: workplace-specific antigens (flour, latex, isocyanates). Immunotherapy is disease-modifying for allergic asthma — not just symptomatic.Positive cat allergen → confirmed sensitisation → graduated avoidance plan. Positive HDM → SLIT eligibility. Multiple sensitisations → allergy clinic referral.
🎓 SCA Checkpoint — Step 4TasksRelating to Others
How to explain investigations
"I need to arrange a breathing test — it measures how much air you can blow out and whether a reliever inhaler opens your airways up. That tells me whether asthma is the right diagnosis. I also want to check your peak flow over two to four weeks twice a day — the pattern of variability tells me more than any single measurement."
Deductions
  • Diagnosing asthma without spirometry or PEFR diary — cannot diagnose on symptoms alone
  • Not ordering FeNO when NICE recommends it for uncertain diagnosis
  • Not considering allergy testing when a specific allergen (cat) is the likely trigger
  • Treating on history alone without objective evidence — especially in medico-legal context
🔴 Red
Diagnoses asthma on symptoms alone · No spirometry or PEFR diary · No objective reversibility data · CXR not considered in first presentation
🟠 Amber
Spirometry ordered · FeNO not considered · Allergy testing not linked to cat concern · PEFR diary not offered · Investigations not explained in lay language
🟢 Green
Spirometry + reversibility + PEFR diary explained · FeNO discussed · Allergy testing linked to cat concern and management plan · FBC eosinophils if poor control · Each test explained in plain language with a reason
5
Step 5
Reaching a Diagnosis — Asthma Classification · Lay Language · DDx · Severity
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Asthma diagnosis has two components: confirming objective evidence of variable airflow obstruction (spirometry/PEFR diary/FeNO), and communicating the diagnosis in a way that addresses the patient's specific fears. For Priya, the diagnosis conversation must include: what asthma actually is, what it means for the cat, why inhalers are safe, and what uncontrolled asthma can lead to.
🗣️ Explaining asthma in plain language

"Asthma is a condition where the airways in your lungs become inflamed and sensitive — a bit like having skin that's prone to reacting. When something irritates them — cold air, exercise, an allergen like cat dander — they tighten up and produce extra mucus, which is what causes the wheeze and tightness. The important thing is: asthma is very treatable. With the right inhalers and trigger management, most people with asthma live completely normal lives — including running, teaching, and keeping active. The goal is to get it so well controlled that you barely notice it."

🐱 Addressing the cat — the most important conversation in this consultation

"Does the cat need to go?"
"The honest answer is: sensitisation to cat dander is one of the more significant allergic triggers for asthma, and removing the exposure is the most effective intervention. But I'm not going to tell you the cat has to go today — I understand how significant that is. What I'd like to do is: first, confirm with an allergy test whether cat dander is the specific trigger for you. Second, while we're waiting, there are practical steps that can significantly reduce your exposure — keeping the cat out of your bedroom, a HEPA air purifier, regular hoovering with an allergen filter. If your symptoms improve significantly with treatment alone, we can reassess. If your asthma remains poorly controlled despite treatment and the cat is confirmed as the trigger, we'll need a more direct conversation — but one that I'm committed to having with you, not at you."

Why this matters: An immediate "get rid of the cat" instruction at a first consultation almost universally leads to disengagement. Shared decision-making with a graduated plan has better outcomes. Document the discussion clearly.

5A — BTS/SIGN Control Classification
✓ Well Controlled
Lay: "asthma not affecting daily life"
No daytime symptoms · No nocturnal waking · No exercise limitation · SABA ≤2 days/week · PEFR ≥80% predicted · No exacerbations. Maintain current step. Annual review mandatory.
⚠ Partially Controlled
Lay: "asthma interfering — needs stepping up"
1–2 features of: daytime symptoms >2/week · any nocturnal waking · any activity limitation · SABA >2 days/week. Before stepping up: check technique, adherence, and triggers first. Then step up if confirmed.
🔴 Uncontrolled
Lay: "asthma is not under control — risk of serious attack"
3+ features above · or any exacerbation in the past week · PEFR <60% predicted. Step up treatment. Check for triggers. Consider OCS if severe. Urgent review in 48 hours.
5B — Differential Diagnosis
COPD — Most important DDx in over-35s
Smoking history >20 pack-years · Progressive dyspnoea not episodic · FEV1/FVC <0.7 post-bronchodilator · <15% reversibility. Treat as ACO (asthma-COPD overlap) if features of both. ICS alone insufficient in COPD — add LABA + LAMA.
VCD / EILO — Exercise-Induced Laryngeal Obstruction
Inspiratory stridor NOT wheeze · Symptoms peak at exercise intensity · Rapid resolution post-exercise · Poor response to bronchodilators · Laryngoscopy diagnostic. SLT (speech and language therapy) is treatment, not inhalers. Frequently misdiagnosed as asthma for years.
Cardiac Failure — "Cardiac Asthma"
Orthopnoea · Paroxysmal nocturnal dyspnoea · Bilateral basal crackles · Raised JVP · History of IHD or hypertension. BNP elevated. Treat heart failure — not asthma. ACEi/ARB contraindicated in asthma-focused approach.
Hyperventilation / Dysfunctional Breathing
Normal PEFR and spirometry during symptoms · Tingling in fingers/lips · Anxiety-triggered · Relieved by breathing retraining · Nijmegen questionnaire positive. SLT and CBT more effective than inhalers. Coexists with asthma in ~30% — treat both.
Malignancy / Endobronchial Disease
Unilateral wheeze · Haemoptysis · Progressive dyspnoea · Weight loss · Age >40 new onset · Stridor. Urgent 2WW CXR + chest CT. Do NOT diagnose as asthma without excluding malignancy in this presentation.
EGPA / Allergic Bronchopulmonary Aspergillosis
EGPA (Churg-Strauss): asthma + peripheral eosinophilia + systemic vasculitis features. ABPA: asthma + high IgE + Aspergillus sensitisation + mucous plugging on CT. Both require specialist input and oral corticosteroids / biologic therapy.
🎓 SCA Checkpoint — Step 5TasksRelating to Others
Communicating diagnosis + cat in lay language
"From the breathing test and the pattern of your symptoms, this is consistent with asthma — airways that are inflamed and reacting to triggers. The cat is likely one of those triggers, but I'm not going to tell you it has to go today. Let me explain what we can do in steps."
"The brown inhaler doesn't make your lungs lazy — it reduces the inflammation that's causing this. Most people are surprised how much better they feel once the underlying inflammation is treated."
Deductions
  • Diagnosing asthma without objective evidence
  • Ordering the cat removed without a graduated, empathic discussion
  • Not distinguishing asthma from COPD in an older smoker
  • Jargon without explanation — "FEV1," "reversibility," "ICS" without lay translation
🔴 Red
Diagnoses on symptoms alone · Cat removal instruction without empathy · VCD / cardiac failure not considered · Control level not assessed · Jargon throughout
🟠 Amber
Objective evidence ordered · Inhaler myths not addressed · Cat concern acknowledged but not managed with a graduated plan · Control level not formally assessed
🟢 Green
Objective evidence required and explained · Lay language · Inhaler myths explicitly debunked · Cat addressed with graduated plan and allergy testing · Control level assessed against BTS criteria · Chunk-and-check
6
Step 6
If Referral Is Needed — What the GP Does Before & During
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Most asthma is managed entirely in primary care — referral is targeted, not reflexive. Steps 1–3 and much of Step 4 are delivered by the GP and specialist asthma nurse. Referral is triggered by: near-fatal history, uncontrolled asthma on high-dose ICS/LABA, biologic eligibility, suspected occupational asthma, or diagnostic uncertainty. The GP must not stop treatment while waiting for specialist input.
ScenarioUrgencyWhat GP does before/during referralWhat GP must NOT do
Acute severe asthma (PEFR 33–50%, SpO2 <92%)999 nowHigh-flow O2 to target SpO2 94–98% · Nebulised salbutamol 5mg q15–20min · IV hydrocortisone 200mg or oral prednisolone 40–50mg · Nebulised ipratropium · IV MgSO4 if poor response · Lie patient upright · Continuous monitoringDo NOT give high-flow O2 without monitoring (risk of hypercapnia in COPD overlap). Do NOT sedate. Do NOT give IV aminophylline without senior input. Do NOT discharge if any doubt.
Uncontrolled asthma on Step 4 (high-dose ICS/LABA)Within 4 weeksConfirm adherence and technique before referral. Document FeNO, blood eosinophils, IgE. Refer to difficult asthma / specialist respiratory clinic. Continue current step — do not step down while uncontrolled.Do NOT add further steps without specialist input. Do NOT prescribe biologics without specialist assessment and eligibility confirmation.
Previous near-fatal attack (ITU / intubation)Same weekWritten asthma action plan immediately. Emergency kit prescribed (nebuliser/OCS for home). Refer to specialist respiratory/difficult asthma clinic. Low threshold for hospital admission with any acute episode.Do NOT manage routine follow-up only. Do NOT allow patient to leave without a written action plan. Do NOT delay specialist referral.
Suspected occupational asthmaWithin 4 weeksPEFR diary (at work and off work). Document work exposures. Refer to occupational medicine / specialist respiratory. Consider SWORD reporting. Advise patient not to expose themselves to peak levels while awaiting referral.Do NOT advise patient to leave job without specialist confirmation. Do NOT delay — occupational asthma becomes permanent with continued exposure.
Biologic therapy eligibility (severe eosinophilic or allergic asthma)Routine — via severe asthma clinicDocument: blood eosinophil count, IgE, FeNO, number of OCS courses/year, PEFR, adherence confirmation. Refer to severe asthma/biologic clinic. Continue current treatment. Provide supporting letter with full medication history.Do NOT prescribe mepolizumab, benralizumab, or omalizumab in primary care — specialist-initiated only. Do NOT step down ICS before referral.
Diagnostic uncertainty — asthma vs COPD vs VCDRoutineEnsure full objective assessment: spirometry, FeNO, CXR, PEFR diary, allergy testing. Start empirical ICS trial if asthma suspected. Refer to respiratory medicine with results attached for diagnostic clarification.Do NOT label as asthma without objective evidence — incorrect diagnosis leads to years of inappropriate treatment. Do NOT withhold ICS trial while awaiting referral if symptoms are significantly impairing.
🎓 SCA Checkpoint — Step 6TasksGlobal Skills
How to explain referral
"I want to refer you to a specialist in chest conditions — not because this is serious or scary, but because we want to make sure we have the right diagnosis confirmed, and to check whether there are better treatments available for you. While we wait, I'm going to keep you on the most appropriate treatment so your breathing is as good as it can be."
Deductions
  • Stopping treatment while waiting for specialist appointment
  • Not giving a written asthma action plan to a patient with previous near-fatal attack
  • Prescribing biologics (mepolizumab, omalizumab) in primary care without specialist initiation
  • Not referring suspected occupational asthma — delay = permanent disease
🔴 Red
No written action plan for high-risk patient · Stops treatment pre-referral · Prescribes biologic in primary care · Occupational asthma not referred · Near-fatal history with no low-threshold plan
🟠 Amber
Correct referral decision · Written plan not provided · Reason for referral not explained to patient · Biologic eligibility not assessed before referral
🟢 Green
Correct urgency · Reason explained in plain language · Written action plan for high-risk · Current treatment continued · Pre-referral investigations (eosinophils, IgE, FeNO) documented · "Do NOT" actions identified and avoided
7
Step 7
Management — Expectation · Goals · Non-Medication · Drug Selector · Drug Reference · Psychosocial · Follow-Up · Safety-Netting
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7A — Address the patient's expectation first: validate → explain → negotiate
🤝
Never dismiss the expectation — acknowledge it, share your reasoning, then agree a shared plan
1
Validate — name their expectation

Priya has two specific fears: the cat and the inhalers. Both are rational. Acknowledge them explicitly before any management plan — not after.

"Before I go through the plan — can I come back to the two things you mentioned at the start? The cat, and your worry about the inhalers being addictive. Let me address both of those directly."
2
Explain — share your clinical reasoning

Debunk the inhaler myth explicitly. Offer a graduated approach to the cat — not an ultimatum. Frame treatment as enabling her professional and personal life.

"The brown inhaler doesn't create dependence — it reduces inflammation in your airways. Your airways don't become reliant on it; they become less inflamed. As for the cat, I'm not here to tell you it has to go today — let's do an allergy test first and see what practical steps help while we figure that out."
3
Negotiate — offer something today

Name one specific action agreed today. Give her own inhaler, a written asthma action plan, and a specific follow-up date. Use the occupational context to motivate engagement.

"The goal I want for you is that asthma doesn't affect your teaching, your running, or your home life. With the right plan — and we're starting that today — most people get there. Let's agree what we're doing now and when we're checking back."
7B — Why treatment matters: goals tailored to this patient
Treatment goals
No daytime symptomsNo nocturnal waking No exercise limitationSABA ≤2 days/week Prevent acute attacksAvoid oral corticosteroids Teach without breathlessnessRun again without symptoms
Motivational language — tailored to Priya
"The goal isn't just to stop the wheeze — it's to get to a point where asthma isn't the thing you think about when you wake up, or when you're trying to run, or when you're standing in front of your class."
"Three people die from asthma every day in the UK — and 90% of those deaths are preventable. Not with dramatic interventions — with the right preventer inhaler used correctly, and a written plan for what to do when things get worse."
7C — Non-medication management: mechanism + evidence + tailored advice
Never give generic lifestyle advice. Each intervention below has a specific mechanism, a quantified effect on asthma control, and a measurable outcome. Present each as a real treatment — and tailor it to Priya's specific situation. One change agreed today beats five changes ignored. The cat discussion requires clinical sensitivity — it is not a simple lifestyle instruction.
🐱
Allergen Avoidance — Cat Dander
Target: reduce airborne Fel d 1 protein exposure
Mechanism

Cat allergen (Fel d 1) is produced in sebaceous glands and saliva, deposited on fur, and becomes airborne as tiny particles that remain suspended for hours. Sensitisation causes mast cell degranulation → histamine + leukotriene release → bronchoconstriction + mucous hypersecretion.

Evidence

Cat allergen persists in a home for up to 6 months after removal of the cat. Bedroom exclusion reduces nocturnal exposure significantly. HEPA filter reduces airborne allergen by 40–60%. Complete removal is most effective but even partial measures reduce SABA use.

Tailored — Priya's situation

Graduated approach: (1) cat excluded from bedroom — immediate; (2) HEPA air purifier in bedroom; (3) weekly bathing of cat (reduces Fel d 1 by ~80%); (4) allergy testing to confirm sensitisation; (5) revisit rehoming if uncontrolled despite treatment. Document discussion clearly.

Bedroom exclusion → ↓ nocturnal wheeze significantly
🏠
House Dust Mite Reduction
Target: reduce HDM allergen in mattress + bedding
Mechanism

HDM (Dermatophagoides pteronyssinus) produces Der p 1 allergen in faeces — deposited in mattresses, pillows, carpets, and soft furnishings. Heat and humidity promote HDM multiplication. Sensitisation in 85% of atopic asthma patients.

Evidence

Allergen-impermeable mattress covers reduce HDM exposure by 80–90%. Weekly washing of bedding at 60°C kills HDM. Removing carpets from bedroom reduces allergen load by >90%. Evidence for clinical benefit is modest but cumulative with other measures.

Practical

Mattress and pillow encasings — widely available, cost-effective. Weekly hot wash of bedding. Dehumidifier to keep bedroom humidity <50%. Minimise soft furnishings. Regular hoovering with HEPA-filter vacuum.

Mattress cover + hot wash → ↓ HDM allergen 80–90%
🚭
Smoking Cessation
Target: complete cessation + no passive exposure
Mechanism

Cigarette smoke causes neutrophilic airway inflammation — steroid-resistant. ICS efficacy is significantly reduced in smokers. Smoking activates histone deacetylase (HDAC2) → corticosteroid insensitivity. Passive smoke exposure in non-smoking asthma patients causes similar effects.

Evidence

Smoking cessation in asthma: FEV1 improves, exacerbation rate falls, ICS response improves, symptom scores reduce. Even stopping passive exposure (partner, home, workplace) produces measurable improvement. NICE: smoking cessation is the highest-impact intervention in smokers with asthma.

Practical

QUIT service referral. Varenicline most effective. NRT as alternative. CO breath test at every review — objective feedback motivates change. Consider the impact of passive smoke in the home — partner smoking indoors should be addressed explicitly.

Cessation → ↑ ICS efficacy + ↓ exacerbations significantly
🏃
Exercise — Do Not Avoid
Target: pre-exercise SABA + warm-up → full activity
Mechanism

Exercise-induced bronchoconstriction (EIB): airway cooling + drying → mast cell degranulation → leukotriene-mediated bronchoconstriction occurring 5–15 minutes after exercise. Nasal breathing warms and humidifies air, reducing EIB. Well-controlled asthma should not limit exercise.

Evidence

Pre-exercise SABA (15 min before): reduces EIB by 80%. 10-minute warm-up protocol: reduces EIB by 50%. LTRA (montelukast): effective for EIB, especially in aspirin-sensitive asthma. Regular exercise actually improves asthma control long-term via improved cardiovascular fitness and reduced obesity.

Tailored — Priya

"You should not give up running. Salbutamol 2 puffs 15 minutes before you run will reduce the bronchoconstriction significantly. Start with a 10-minute warm-up. Breathe through your nose on cold days. If it still limits you on treatment, tell me and we reassess."

Pre-exercise SABA → ↓ EIB 80%; exercise ↑ long-term control
😮‍💨
Breathing Retraining (Buteyko / Physiotherapy)
Target: reduce overbreathing + reduce rescue SABA
Mechanism

Dysfunctional breathing patterns (chronic hyperventilation, mouth breathing) lower CO2 → vasoconstriction + increased airway sensitivity. Buteyko technique teaches nasal breathing, reduced breathing volume, and breath control. Reduces the symptom amplification from anxiety + asthma interaction.

Evidence

SIGN 2023: breathing exercises recommended for asthma as adjunct to medication. Buteyko and physiotherapy-led breathing retraining reduce SABA use, improve quality of life scores, and reduce symptom scores. Effect is greatest in those with anxiety co-morbidity or dysfunctional breathing pattern.

Practical

Refer to physiotherapist or respiratory nurse with breathing retraining expertise. Buteyko apps available (evidence weaker). Especially useful when anxiety and asthma coexist — teaches discrimination between panic attack and asthma attack.

↓ SABA use + ↑ QoL — SIGN-recommended adjunct
⚖️
Weight Management
Target: BMI <30; 5–10% weight loss
Mechanism

Obesity reduces functional residual capacity (FRC) → airway closure during tidal breathing. Pro-inflammatory adipokines (leptin, IL-6) promote neutrophilic inflammation → ICS-resistant asthma phenotype. Obesity-related GORD further triggers airway inflammation. Metabolic syndrome worsens overall respiratory physiology.

Evidence

Weight loss 5–10% in obese asthma: FEV1 improves, exacerbation rate falls, OCS courses reduce, quality of life improves. NICE: weight management should be addressed before stepping up ICS in obese patients with apparently uncontrolled asthma.

Practical

Structured weight management programme referral. NHS DESMOND equivalent for respiratory patients. Frame as: "Losing weight is one of the most effective things you can do for your breathing — it changes the type of inflammation and makes your inhalers work better."

↓ OCS courses + ↑ FEV1 with 5–10% weight loss
7D — Prescribing Guide: what to start, in what order, and why
Three decisions, in this order, every time: (1) Does this patient need to step up — or is the apparent failure due to technique, adherence, or an unaddressed trigger? (2) Which step of the NG245 pathway is appropriate? (3) Within that step, which drug fits this patient's phenotype, comorbidities, and preferences? Never skip step 1 — the most common cause of "uncontrolled asthma" is suboptimal technique or non-adherence, not insufficient medication.
1
Before stepping up — always check these three things first
⚠ Check technique
Up to 80% of patients use inhalers incorrectly. Ask the patient to demonstrate their inhaler technique — do not assume. Wrong technique negates even the correct drug at the correct dose. Switch device (MDI + spacer vs DPI) if repeated technique failure.

"Before we change anything — can you show me how you use your inhaler? I'd like to watch you actually do it."
💊 Confirm adherence
FeNO paradoxically low in a patient on high-dose ICS = they are not taking it. Cross-reference prescription collection dates. Non-adherence to ICS is the leading cause of "poorly controlled" asthma. Address the reason — myth, cost, inconvenience — before escalating.

"How often do you find yourself skipping the brown inhaler? What gets in the way?"
🔎 Identify new trigger
Has something changed? New pet, new job, new medication (beta-blocker, NSAID, ACEi), new housing (mould, damp), new relationship stress. A new trigger treated as "worsening asthma" will not respond to a step up — it requires trigger removal.

"Has anything changed recently — at home, at work, or with any other medicines you're taking?"
Only if technique is confirmed correct + adherence confirmed + no new trigger identified → proceed to step up on the NG245 pathway
2
Step-by-step prescribing order — NICE/BTS/SIGN NG245 (2024)
Step 1
New diagnosis / mild
START: AIR — low-dose ICS/formoterol PRN
Combined low-dose ICS/formoterol (e.g. Symbicort 200/6 or Fostair 100/6) 1 puff as needed for symptoms — the "anti-inflammatory reliever" (AIR). NG245: do NOT start salbutamol alone. Demonstrate technique at every prescription.
WHY this first
Formoterol relieves as fast as salbutamol, but every reliever dose now carries ICS — treating the inflammation with each use. AIR roughly halves severe exacerbations vs SABA-only (SYGMA-1, Novel START). SABA-only therapy is no longer recommended at any step.
→ Step up to low-dose MART if using the AIR inhaler ≥3 days/week, any night waking, or any exacerbation
Step 2
Persistent symptoms
MOVE TO: Low-dose MART
Same ICS/formoterol inhaler, now taken regularly: e.g. Symbicort 200/6 one puff BD maintenance PLUS as needed for relief (MART). One inhaler covers prevention and relief. Rinse mouth after maintenance doses. Review at 8–12 weeks.
WHY MART at Step 2
Asthma is an inflammatory disease — regular ICS treats the inflammation, and keeping formoterol as the reliever means every extra puff during a bad patch automatically delivers more ICS. MART reduces exacerbations, admissions and asthma deaths versus fixed-dose regimens with separate SABA.
→ Step up if still uncontrolled after 8–12 weeks on low-dose MART with confirmed technique + adherence
Step 3
Uncontrolled on low-dose MART
INCREASE: Moderate-dose MART
Increase the maintenance dose: e.g. Symbicort 200/6 two puffs BD (or one puff BD of a higher-strength device) + PRN relief (max 8 extra puffs/day). Alternative if MART unsuitable: fixed-dose moderate ICS/LABA + separate reliever. Add-on: Montelukast 10mg nocte if rhinitis, AERD, or EIB co-exist.
WHY moderate-dose MART
Escalating within MART keeps the exacerbation-protection of the ICS-with-every-relief mechanism (~30% fewer severe exacerbations vs fixed ICS/LABA + SABA). ⛔ NEVER prescribe LABA without ICS.
→ If still uncontrolled after 8–12 weeks → check FeNO + blood eosinophils (Step 4)
Step 4
Uncontrolled on moderate MART
CHECK BIOMARKERS → ADD-ON or REFER
NG245 gate: check FeNO and blood eosinophils. If EITHER is raised → refer to a specialist asthma service. If BOTH are normal → 8–12 week trial of a LTRA (montelukast) or a LAMA added to moderate-dose MART; stop if ineffective, then refer.
WHY biomarkers before add-ons
Raised FeNO/eosinophils = ongoing type-2 inflammation despite moderate ICS — these patients need specialist assessment (and may be biologic candidates), not blind dose escalation. Normal biomarkers = bronchodilator-responsive phenotype where a LAMA or LTRA trial is rational. This biomarker gate is new in NG245.
→ Specialist referral if not already made. Biologic eligibility assessment if ≥4 exacerbations/year.
Step 5
Severe asthma
BIOLOGIC ± MAINTENANCE OCS — specialist only
Eosinophilic: Mepolizumab (anti-IL-5) 100mg SC q4w, or Benralizumab 30mg SC q4w (then q8w). Allergic: Omalizumab (anti-IgE) SC q2–4w. Type 2: Dupilumab 300mg SC q2w. Maintenance OCS (prednisolone ≥5mg/day): last resort only — significant systemic toxicity.
WHY biologics at Step 5
Biologics target specific inflammatory pathways — IL-5, IgE, IL-4/13 — rather than broad immunosuppression. Reduces OCS exposure (and its toxicity: diabetes, osteoporosis, adrenal suppression). Eligibility: specialist-confirmed, phenotype-matched. Primary care role: ensure adherence + monitor + annual specialist review. ⛔ Cannot be started in primary care.
→ GP role: refer + document eligibility criteria + continue current step + monitor for OCS complications
3
Within the step — choose the right drug for this patient's phenotype
Which ICS? — device and molecule matter
ScenarioChooseWhy
Standard adult Step 2BDP MDI + spacerCost-effective, spacer maximises lung deposition
Poor MDI techniqueDPI (Turbohaler/Accuhaler)Breath-actuated — eliminates coordination failure
PregnancyBudesonideMost safety data in pregnancy — first choice
Step 3 — MARTSymbicort (budesonide/formoterol)Only formoterol-containing ICS/LABA approved for MART
Oral candidiasis recurrentCiclesonideActivated in lungs only — lowest oral deposition
Add-on choice by phenotype — Step 2–4
Phenotype / featurePreferred add-on
Allergic rhinitis coexistingMontelukast + nasal ICS
AERD (aspirin-sensitive)Montelukast — especially effective
Exercise-induced onlyPre-exercise SABA ± montelukast
Eosinophilic (FeNO ≥50 adult / ≥35 age 12–16, or eos ≥0.3)Increase ICS dose; biologic if Step 5
Allergic — high IgE (30–1500)Omalizumab (specialist)
GORD-triggered asthmaPPI + lifestyle + re-assess ICS need
⛔ Absolute prescribing rules — never broken: (1) LABA must never be prescribed without ICS — LABA monotherapy is independently associated with asthma death. (2) Biologics are specialist-initiated only — primary care role is to refer, not prescribe. (3) Montelukast requires explicit neuropsychiatric counselling at every prescription (MHRA 2020). (4) MART maximum: 8 extra relief puffs/day — exceeding this requires urgent medical review. (5) Do not step up without first confirming technique, adherence, and triggers.
⚙ Interactive Medication Chooser — tick the patient profile, options re-tier live against NICE / BNF
A live, topic-scoped version of the standalone Medication Chooser. The static selector and reference cards below are unchanged.
7E — Medication selection tool — choose patient characteristics for tailored recommendations
Tick the patient's relevant characteristics. The three recommendation boxes update instantly with step recommendations, inhaler choices, and monitoring priorities.
🫁 Control / Severity
🧬 Phenotype / Features
⚕️ Clinical Factors
Treatment recommendation
☑️Select patient characteristics above — recommendations appear instantly
7F — Drug reference cards
SABA — Short-Acting Beta-2 Agonist
Salbutamol (Ventolin) · Terbutaline (Bricanyl)
⚠ Legacy reliever — no longer started alone
Step 1+100mcg 1–2 puffs PRN
✓ Use for
Immediate bronchodilation (onset 3–5 min) — for patients established on fixed-dose ICS/LABA + separate reliever regimens
Pre-exercise: 2 puffs 15 min before exercise to prevent EIB
NG245: never start SABA alone — new diagnoses begin AIR (low-dose ICS/formoterol PRN) instead
⚠ Warning signs of over-reliance
SABA >3×/week = poor control = step up mandatory
SABA >1 canister/month = independently associated with asthma mortality
Prescribing SABA without any ICS is an NG245 quality failure — SABA-only therapy is banned at every step
⚠ Side effects
Tremor, tachycardia, hypokalaemia at high doses (nebuliser)
💬 Counselling phrase

"This is your reliever — it opens your airways quickly. But if you're needing it more than 3 times a week, that's a sign your asthma isn't controlled and we need to do more, not just use more of this."

Reliever frequency is the central control metric. In SCA: always ask how often they use it and interpret it — daily use = uncontrolled = immediate action. Bonus mark: knowing NG245 replaced SABA-first with AIR→MART, and transitioning legacy SABA-only patients at review.

ICS — Inhaled Corticosteroid
Beclometasone · Budesonide · Fluticasone propionate
✓ Preventer — Step 2+
Step 2+BDP 200–400mcg/day (low)
✓ Add ICS when
From diagnosis — under NG245 the ICS arrives with the first reliever (AIR: ICS/formoterol PRN)
Exacerbation in last 2 years requiring OCS
Any risk factor for near-fatal attack
✗ Avoid / caution
High-dose ICS in children — growth monitoring required
Oral candidiasis — rinse mouth after every dose (spacer helps)
Do NOT step down ICS in pregnancy
⚠ Critical teaching point
Must use spacer device for MDI — improves lung deposition 2–4×
Takes 4–6 weeks to achieve full anti-inflammatory effect
Technique check at every consultation
💬 Counselling phrase

"This is the most important inhaler — it treats the inflammation causing your asthma. It doesn't work immediately like the blue one, but after a few weeks you should notice fewer symptoms and needing the blue one less. It won't make your lungs lazy — it will make them less inflamed."

Address the "inhalers make lungs lazy" myth every time ICS is prescribed. Check technique and adherence at every review — non-adherence is the most common cause of "poorly controlled" asthma. Rinse mouth after use to prevent candidiasis.

ICS/LABA Combination
Seretide (fluticasone/salmeterol) · Symbicort (budesonide/formoterol) · Fostair
✓ Step 3–4
Step 3+Symbicort 200/6 1–2 puffs BD
✓ Add LABA when
Uncontrolled on low-dose ICS alone (Step 2 → 3)
MART therapy (Symbicort): single inhaler for both maintenance and relief — reduces exacerbations
Superior to increasing ICS dose alone at Step 3
⚠ Critical rules
NEVER use LABA without ICS — LABA monotherapy associated with asthma death
If adding LABA does not improve control: do not step up further without specialist review
⚠ Side effects
Tremor, palpitations with LABA. Oral candidiasis with ICS component — rinse mouth
💬 Counselling phrase

"This inhaler combines the preventer and an additional bronchodilator that lasts 12 hours. It gives better control than doubling the preventer dose alone. Never use the long-acting part without the preventer — they must always be taken together."

MART (Maintenance And Reliever Therapy) with Symbicort reduces severe exacerbations by ~30% vs fixed-dose ICS/LABA + SABA. Ask about MART adherence — patients often use it only as a reliever.

LTRA — Leukotriene Receptor Antagonist
Montelukast (Singulair) 10mg OD
✓ Step 2–4 add-on
Add-on10mg nocte OD
✓ Especially useful in
Allergic asthma + allergic rhinitis (treats both — oral, once daily)
AERD / aspirin-sensitive asthma — particularly effective
Exercise-induced bronchoconstriction
Step 2 alternative if ICS not tolerated or declined
⚠ Important warning
Neuropsychiatric effects: depression, anxiety, nightmares, suicidal ideation — MHRA 2020 warning. Screen at baseline and after starting. Withdraw if symptoms emerge.
Less effective than ICS as primary preventer — do not use as sole preventer if ICS is indicated
💬 Counselling phrase

"This tablet reduces a different type of airway inflammation to the inhaler — it is particularly good if you also have hay fever. One important thing: a small number of people notice low mood, vivid dreams, or anxiety — if that happens, let me know straight away and we will stop it."

MHRA 2020 black-box warning for neuropsychiatric effects with montelukast. Must counsel at every prescription. Particularly effective in: allergic asthma + rhinitis, AERD, exercise-induced asthma.

MART — Maintenance & Reliever Therapy
Symbicort MART · Fobumix Easyhaler · DuoResp Spiromax
✓ NG245 core pathway
Step 31–2 puffs BD maintenance + PRN
✓ Advantages of MART
Single inhaler for both maintenance and relief — reduces exacerbations ~30% vs fixed-dose regimen
Automatic dose increase during early exacerbation (formoterol has rapid onset like SABA)
Proven to reduce severe exacerbations, hospital admissions, and OCS use
⚠ Limits and rules
Maximum 12 puffs/day total (maintenance + relief combined)
If needing >8 puffs/day relief: seek medical review urgently
Only formoterol-containing ICS/LABA combinations approved for MART (not salmeterol)
💬 Counselling phrase

"With this inhaler you take it morning and evening as normal, but if your breathing gets worse you can also use extra puffs as a reliever — up to 8 extra puffs in a day. If you're needing that many extra puffs, that's a sign to contact us urgently or go to hospital."

MART (low-dose → moderate-dose) is the spine of the NG245 adult pathway. The nuance: patients must understand the daily maximum and the 8-puff urgent review threshold. Common SCA deduction: prescribing MART without explaining the relief-use limit.

Biologic Therapy
Mepolizumab (anti-IL-5) · Benralizumab · Omalizumab (anti-IgE) · Dupilumab
Step 5 — specialist only
Step 5Specialist-initiated SC injection
✓ Consider when (eligibility criteria)
Eosinophilic: mepolizumab / benralizumab — eos ≥0.3 + ≥4 exacerbations/year or OCS-dependent
Allergic: omalizumab — IgE 30–1500 IU/mL + confirmed allergen sensitisation + ≥4 exacerbations/year
Type 2: dupilumab — eos ≥0.15 or FeNO ≥25 + uncontrolled on high-dose ICS/LABA
⚠ Primary care rules
Specialist-initiated only — cannot be started in primary care
GP role: ensure adherence, monitor for side effects, annual review with specialist
💬 Counselling phrase

"This injection works at the root cause — it targets the specific part of your immune system causing the most inflammation in your airways. It is not a cure, but many people find their exacerbations almost disappear. It needs to be given by a specialist team."

In SCA: biologic eligibility should be considered when eosinophil count, FeNO, or multiple OCS courses are present. Referring for assessment is a Tasks domain score — not prescribing biologics yourself.

7G — Psychosocial context: integrate into the plan
💛
Psychosocial Factors — Shape Every Management Decision in Asthma
🐱
The cat — relationship vs health

Rehoming a pet can cause genuine relationship conflict. Graduated approach is more effective than ultimatum. Document the discussion. Allergy testing provides objective evidence that helps shared decision-making without seeming like personal judgment.

"I'm not going to tell you the cat has to go today — let's confirm the allergy, try the practical steps, and revisit this with the evidence."
💊
Inhaler myths — prevent non-adherence

"Inhalers make lungs lazy" must be explicitly debunked at every new prescription. Non-adherence to ICS is the most common cause of poorly controlled asthma. Technique demonstration and return demonstration at every review.

"The preventer inhaler reduces inflammation — it doesn't do the breathing for you. Your lungs won't become dependent on it."
👩‍🏫
Occupational impact

A teacher's breathlessness is professionally visible. Asthma affecting voice or stamina risks classroom authority and self-confidence. Occupational health referral if school exposures are relevant. Fitness-for-work documentation if needed.

"The goal is that asthma doesn't affect your teaching — that's a realistic target once we get your airways controlled."
😰
Anxiety + panic attacks

Anxiety and asthma amplify each other. Panic attack can mimic acute asthma — both cause breathlessness and tachycardia. Breathing retraining helps discriminate. PHQ-9 + GAD-7 at every review. CBT is effective for anxiety component.

"Sometimes when you're anxious your breathing changes in a way that feels like the asthma — would it help to know how to tell the difference?"
🏃
Exercise and identity

Many patients stop exercising to avoid breathlessness — this worsens asthma long-term. Restoring exercise capacity is a core goal. Pre-exercise SABA should be framed as enabling, not as evidence of severity.

"You should be able to run — let's make that happen. The blue inhaler 15 minutes before you set off makes a huge difference."
👨‍👩‍👧
Family modelling

Mother's approach to asthma management shapes Priya's. If mother never used a preventer, Priya probably thinks they are optional. Ask what her mother does — it reveals the patient's default asthma model and where the educational gap is.

"What does your mum use for her asthma? That helps me understand what you've grown up thinking is normal."
7H — Follow-up timeline
Dx
Diagnosis / initiation visit — today

Spirometry + FeNO + PEFR diary started · SABA prescribed (own inhaler) · ICS started if SABA >3×/week · Inhaler technique demonstrated · Written asthma action plan · Trigger avoidance advice (cat — graduated) · Allergy testing requested · Asthma self-management education · Annual review appointment booked

Own SABA todayICS if indicatedAction plan day 1Allergy test requested
4–6w
4–6 weeks — response assessment

PEFR diary review · Symptom control assessment · Inhaler technique recheck · Side effects (candidiasis?) · SABA use frequency · Adherence to preventer · Cat / trigger changes made? · Step up if not controlled

3m
3 months — control confirmation

Formal control assessment (BTS criteria) · Spirometry repeat if initial abnormal · Allergy test result review · Adjust step if needed · Breathing retraining referral if EIB persists · PHQ-9 and GAD-7

1yr
Annual review — mandatory NICE/BTS

Asthma Control Test (ACT) or RCP 3 questions · SABA use in last 3 months · Nocturnal symptoms · Exercise limitation · Inhaler technique · Device concordance · Triggers review · Step up or down · Written action plan updated · Influenza vaccination · Smoking status · Weight · Oral health

Mandatory annuallyTechnique check every visit
Acute
Post-exacerbation review (within 48h and 2–4 weeks)

Every acute exacerbation requiring OCS → review within 48h · Trigger identified? · Step up treatment · Written action plan updated · Consider specialist referral if ≥2 OCS courses/year · Assess for biologic eligibility if ≥4 exacerbations/year

7I — Monitoring targets and thresholds
MeasureTimingAction threshold
SABA use frequencyEvery consultation>3×/week = poor control → step up. >1 canister/month = risk marker → urgent review.
PEFR (personal best)At home twice daily × 2–4 weeks<80% personal best = action point. <50% = acute severe → escalate. Variability >20% = confirms diagnosis.
Asthma Control Test (ACT)Every reviewScore ≤19 = uncontrolled → step up. Score 20–25 = well controlled → maintain. Score <12 = severe → urgent review.
Spirometry (FEV1/FVC)At diagnosis; repeat if clinical changeFEV1 <70% predicted = moderate–severe → specialist consideration. Declining FEV1 trend = step up + review.
FeNOAnnually or when stepping≥50 ppb (adults 17+) / ≥35 ppb (ages 12–16) = raised (NG245 diagnostic threshold; at the step-4 gate a raised FeNO means refer rather than blind-escalate). Persistently low with poor control → consider adherence or an alternative diagnosis.
Blood eosinophilsAnnually in uncontrolled asthma≥0.3 × 10⁹/L = eosinophilic phenotype → biologic eligibility screen. Rising eosinophils on OCS = OCS-dependent → refer.
OCS courses per yearEvery review≥2 courses/year → step up + specialist referral. ≥4 courses/year → biologic eligibility assessment.
Inhaler techniqueEvery reviewSuboptimal technique → retrain before stepping up. Consider device change (DPI vs MDI + spacer). Up to 80% use inhalers incorrectly.

Key targets and thresholds

Well controlled: SABA ≤2/week · No nocturnal symptoms · No exercise limitation · PEFR ≥80% predicted

PEFR targets: Personal best (not population predicted). >80% = well controlled. 50–80% = action. <50% = emergency.

Acute severe: PEFR 33–50% or SpO2 <92% → hospital. Life-threatening: PEFR <33% → 999.

Before stepping up: Always check technique + adherence + triggers first. Stepping up without these = wasted step.

999: Silent chest · SpO2 <92% · Cannot speak · Cyanosis · Bradycardia · Altered consciousness
Safeguarding: Child with repeated A&E · Parent capacity · Passive smoke · Damp housing · Occupational coercion
Urgent: PEFR 33–50% · HR >110 · Unable to complete sentences · Previous near-fatal + any acute episode
Before stepping up: Check technique · Confirm adherence · Identify new trigger · Rule out alternative diagnosis
7J — Safety-netting: exact phrases for every asthma patient

⚠ Three scenario-specific phrases — use these verbatim

🔴 Emergency — when to call 999
"If you cannot complete a sentence, your lips or fingernails look blue, your reliever inhaler is not helping at all, or you feel like you are fighting for every breath — call 999 immediately. Do not drive yourself. Do not wait to see if it gets better on its own. This is the most important thing I will tell you today."
Asthma kills 3 people per day in the UK. 90% of deaths are preventable. Patients and families consistently underestimate severity until it is too late. Naming 999 explicitly, with specific symptoms, saves lives. Silent chest and SpO2 <92% are emergencies even if the patient feels they are coping.
💊 Urgent — when to escalate before it becomes an emergency
"If your blue inhaler is wearing off within 2 hours of each puff, or you are needing it more than every 4 hours, or you wake up 3 nights in a row with breathlessness — contact us the same day or go to an urgent care centre. Do not wait for a routine appointment. Start your prednisolone if you have it prescribed on your action plan."
The window between "bad day" and "acute severe attack" is often only hours. Patients who escalate at the amber stage have better outcomes than those who wait for the emergency. The written asthma action plan makes this decision explicit and removes ambiguity.
🟡 Action — managing poor control at home
"If your symptoms get worse over a few days — more wheeze, waking more at night, using the blue inhaler more than usual — increase your preventer dose as shown on your action plan and contact us for a review within 48 hours. If you have a written plan, follow it. Keep it somewhere visible — on the fridge or in your phone."
The written asthma action plan (3-zone system: green/amber/red) is evidence-based and reduces hospitalisation. BTS/SIGN 2023: every asthma patient should have a written, personalised action plan. It is a Tasks domain score in SCA and a quality indicator in primary care.
4–6 weeksPEFR diary review · Inhaler technique · SABA frequency · Trigger changes
3 monthsFormal control assessment · Spirometry if indicated · Allergy result · Step review
AnnualFull review: ACT + technique + OCS courses + triggers + action plan update + flu vaccine
📋 SCA Consultation Scorecard — self-assess your asthma consultation
Asthma — SCA Consultation Scorecard
Based on the official SCA Consultation Tool · RAG self-assessment · Use after every practice asthma consultation
0/ 33 pts
🌐
Global Skills
Structure, lay language, responsiveness
0/7
📋
Tasks
Data gathering, diagnosis, clinical management
0/15
🤝
Relating to Others
Communication, rapport, shared decision making
0/11
011172533
Not passing <11
Borderline 11–16
Pass 17–24
Strong 25–33
📋
Complete the checklist above to see your score interpretation and personalised feedback.
🏥
Clinic Quick Reference
Asthma — Diagnostic Pathway & Management Summary
NICE/BTS/SIGN NG245 (2024) · GINA 2025 · Numbers · Step ladder · Safety netting · Monitoring
expand
🔬 1 — Diagnostic Pathway & Triage Algorithm
Wheeze · Breathlessness · Chest tightness · Nocturnal symptoms — assess severity first
🚨 Emergency — 999
  • Silent chest / absent breath sounds
  • SpO2 <92% / cyanosis
  • Cannot complete sentences
  • PEFR <33% predicted
  • Bradycardia / exhaustion / confusion
999 immediately
⚠ Urgent — same day
  • PEFR 33–50% / SpO2 92–94%
  • HR >110 / RR >25
  • Not responding to SABA after 30 min
  • Previous near-fatal + any acute episode
  • 2+ OCS courses this year
Hospital / same-day review
✓ Routine — GP management
  • New diagnosis: spirometry + PEFR diary + FeNO
  • Annual review: control + technique + triggers
  • Reliever >3×/week: step up NG245 pathway
  • Mild exacerbation (PEFR >50%): increase SABA + OCS
NG245 pathway
🔢 2 — Key Numbers to Know
PEFR thresholds
>80%
Well controlled
50–80%
Action needed
<50%
Emergency
Reversibility (bronchodilator)≥12% + ≥200mL FEV1
PEFR variability — diagnosis>20% variability
FeNO — eosinophilic inflammation≥50 ppb adult (17+) · ≥35 ppb age 12–16
Blood eosinophils — biologic≥0.3 × 10⁹/L
SABA over-use — mortality risk>1 canister/month
Control criteria and biologic thresholds
Well controlledSABA ≤2/wk, no nocturnal, no limitation
Step up triggerSABA >3×/wk or any nocturnal Sx
Specialist referral≥2 OCS courses/year
Biologic eligibility≥4 exacerbations/year + eos ≥0.3
MART relief max8 extra puffs/day → urgent review
💊 3 — NG245 Pathway (adults & 12+)
Step Treatment Trigger Before stepping up
Step 1AIR — low-dose ICS/formoterol PRNNew diagnosis / infrequent symptoms — SABA-only banned
Step 2Low-dose MART (ICS/formoterol BD + PRN)Reliever ≥3 days/week · night waking · any exacerbationCheck technique · Confirm diagnosis
Step 3Moderate-dose MARTUncontrolled on low-dose MARTAdherence + technique confirmed
Step 4FeNO + eosinophils → raised: REFER · normal: trial LTRA or LAMA 8–12wUncontrolled on moderate-dose MARTBiomarker gate is mandatory (NG245)
Step 5Biologic (mepolizumab / omalizumab / dupilumab) ± OCS≥4 exacerbations/year or OCS-dependent + eligible phenotypeSpecialist-initiated only
⛔ LABA without ICS = asthma death risk · ⛔ SABA >1 canister/month = mortality marker · ⛔ MART max = 8 extra puffs/day · ⛔ Montelukast = counsel neuropsychiatric risk at every prescription
⚠ 4 — Safety Netting & Follow-Up
🔴 Emergency — ALL asthma patients
"Cannot complete a sentence · lips look blue · reliever not helping at all · fighting for every breath → 999 immediately. Do not drive yourself. Do not wait."
🟠 Urgent — escalate before emergency
"Blue inhaler wearing off in <2h · needing it every 4h · waking 3 nights running → contact us same day or urgent care. Start prednisolone if on action plan."
🟡 Action — managing at home
"Symptoms worsening over days → increase preventer as per action plan. Contact us within 48 hours. Keep written plan on fridge or in phone."
Follow-up schedule
Dx
Diagnosis: AIR started (low-dose ICS/formoterol PRN — no SABA-only, NG245) · Inhaler technique demonstrated · Written action plan · PEFR diary · Trigger avoidance · Allergy testing · Annual review booked
4–6w
4–6 weeks: PEFR diary review · Control assessment · Technique recheck · SABA frequency · Trigger changes
3m
3 months: Formal control assessment · Spirometry repeat · Allergy result · Step review · PHQ-9
1yr
Annual: ACT + technique + OCS courses/year + triggers + action plan update + flu vaccine + smoking + weight
📌 Post-exacerbation: review within 48h AND 2–4 weeks after every OCS course. ≥2 OCS/year → refer. ≥4/year → biologic eligibility.
🔬 5 — Monitoring Targets
MeasureTimingAction threshold
SABA use frequencyEvery consultation>3×/week = step up. >1 canister/month = mortality risk marker → urgent review.
PEFR (personal best)2× daily × 2–4 weeks at diagnosis<80% → action. <50% → emergency. >20% variability → confirms diagnosis.
Asthma Control Test (ACT)Every review≤19 = uncontrolled → step up. <12 = severely uncontrolled → urgent review.
Inhaler techniqueEvery reviewSuboptimal → retrain before stepping up. 80% use inhalers incorrectly.
FeNOAt diagnosis; when stepping>40 = high eosinophilic inflammation → ICS increase. <25 = consider adherence/alternative Dx.
Blood eosinophilsAnnually if uncontrolled≥0.3 × 10⁹/L = biologic eligibility screen if ≥4 exacerbations/year.
SpirometryAt diagnosis; repeat if clinical changeFEV1 <70% predicted = moderate–severe → specialist consideration.
OCS courses/yearEvery review≥2 → step up + specialist referral. ≥4 → biologic eligibility assessment.
999: Silent chest · SpO2 <92% · Cannot speak · Cyanosis · Bradycardia · Altered consciousness · Tension PTX
Safeguarding: Child A&E pattern · Passive smoke · Damp housing · Occupational coercion · Medication misuse
🛡️ 6 — Trigger Control & Prevention
Allergen avoidance — cat + HDM
Cat: bedroom exclusion + HEPA + weekly bathing + allergy test. Cat allergen persists 6 months after removal. HDM: mattress covers + 60°C wash + dehumidifier. Confirming sensitisation with allergy test supports shared decision-making.
Rhinitis coexists in 60–80%
Treat allergic rhinitis → improves asthma control. Intranasal corticosteroid + regular antihistamine. SLIT eligible patients: refer allergy clinic. "One airway, one disease" — treat both.
SABA over-reliance — mortality marker
SABA >1 canister/month = mortality risk. Address at every review. Step up ICS. Check technique. MART reduces SABA over-reliance. Written action plan reduces acute attacks.
🎓
SCA Exam Quick Reference
SCA Consultation Blueprint
Tasks · Relating to Others · Global Skills · RAG guide
expand
🕐 12-Minute Consultation Flow
0–2 min
Open & ICE
"I can see from your notes you've been having trouble with your breathing for several weeks and have been borrowing a blue inhaler. Before I go through everything, tell me what's been worrying you most about this."
"You mentioned a cat — what would it mean at home if I said the cat was part of this?"
"What do you know about inhalers — have you had any worries about using them long-term?"
Relating to OthersGlobal Skills
✗ Notes not used · Repeating documented info · No open Q · Going straight to symptoms without surfacing cat concern + inhaler myths
2–4 min
Safety Screen
"Are you breathless right now sitting here? Any chest pain? Have you measured your peak flow?"
Red flags aloud: PEFR + SpO2 · Silent chest sign · Previous near-fatal/ITU · Unilateral wheeze · Haemoptysis
Trigger pattern: cat · HDM · exercise · occupation · NSAID/beta-blocker history
TasksGlobal Skills
✗ No PEFR or SpO2 · Not asking about previous near-fatal attack · Missing NSAID/beta-blocker precipitants · Overrunning data gathering
4–7 min
Context & Risk
SABA use frequency: "How many times a week are you using the blue inhaler? Have you been through a whole canister?" → control assessment
Occupation: teacher — breathless in class · passive chalk dust exposure · occupational asthma pattern?
Family: mother's asthma model · inhaler myth source · PHQ-9 and GAD-7
TasksRelating to Others
✗ No SABA frequency quantified · Generic trigger advice without linking to cat · Occupation not linked to management · Inhaler myths not collected before management
7–10 min
Explain & Address ICE
"The brown inhaler doesn't make your lungs lazy — it reduces inflammation. Your lungs don't become dependent on it; they become less inflamed."
"I'm not going to tell you the cat has to go today. Let's do an allergy test first and try practical steps — bedroom exclusion, HEPA filter — while we figure out how much it's contributing."
"With the right plan, you should be able to teach without breathlessness and run again — that's the goal."
TasksRelating to Others
✗ Inhaler myths not debunked · Cat removal as ultimatum · "ICS causes dependence" not corrected · Jargon · Not linking management to occupational + exercise goals
10–12 min
Plan & Close
"If you cannot complete a sentence, or the blue inhaler isn't helping at all, or you feel like you're fighting for every breath — call 999 immediately."
Own SABA prescribed · ICS started if SABA >3×/week · Inhaler technique demonstrated · Written asthma action plan given · PEFR diary · 4–6 week review named · "Is there anything else?"
TasksRelating to OthersGlobal Skills
✗ No written action plan · No 999 symptoms stated · Vague follow-up · No technique demonstration · No closing question · ICS not started despite daily SABA use
🔴🟠🟢 RAG — All 3 Domains
Tasks
🟢
PEFR + SpO2 checked · Spirometry + PEFR diary ordered · ICS started (SABA >3×/week) · Written action plan given · Technique demonstrated · Allergy test requested · 4–6 week review named · 999 symptoms explicitly stated · Montelukast neuropsychiatric warning counselled if prescribed
🟠
PEFR checked not interpreted · Action plan offered not written · ICS discussed not prescribed · Allergy testing not linked to cat concern · Follow-up vague · 999 symptoms general not specific
🔴
No PEFR · No SpO2 · SABA over-use not identified · No ICS despite daily SABA · No action plan · No technique check · Diagnoses asthma without objective evidence · No safety netting
Relating to Others
🟢
Notes used first · Cat addressed with graduated plan + allergy test (not ultimatum) · Inhaler myths explicitly debunked · Teaching career used as motivational frame · Expectation addressed before plan · Exercise goal enabled · Chunk-and-check · "Anything else?"
🟠
Cat mentioned not explored emotionally · Inhaler myth collected not corrected · Occupation noted not used · Expectation after plan · No chunk-and-check
🔴
Cat removal as instruction · Inhaler myths ignored · Straight to prescribing · Jargon throughout · No shared decision-making · No ICE used in plan
Global Skills
🟢
Notes used first · Open Q yields ICE in first response · Data gathering complete by 6–7 min · Lay language · Control level assessed explicitly · Responsive to cues · Structured · Expectation before management
🟠
Some jargon (FEV1, FeNO not explained) · Overruns data gathering · Misses control level assessment · Expectation addressed after plan
🔴
Notes not used · Jargon throughout · Data gathering incomplete · Cues ignored · No structure · PEFR not measured · Jumps to management without data
💬 Key Phrases
💭 Ideas — inhaler myths
"What do you know about the inhalers — have you heard anything about them that worries you? A lot of people have heard they can make lungs lazy — let me address that directly."
😟 Concerns — the cat
"I want to come back to the cat — because I could hear how important that is. I'm not going to tell you it has to go today. Let's do an allergy test first and try some practical steps while we figure out how much it's contributing."
🎯 Expectation first
"Before I go through the plan — can I address the cat question and the inhaler question right now, because those change how the whole conversation feels."
🗣️ Debunk myth
"The brown inhaler doesn't do the breathing for you — it reduces the inflammation causing your asthma. Your airways don't become reliant on it. They become less inflamed. After 4–6 weeks you should need the blue one much less."
👩‍🏫 Occupational frame
"The goal is that asthma doesn't affect your teaching, your running, or your home life. That's a realistic target once we get your airways controlled with the right treatment."
✅ 999 + close
"If you cannot complete a sentence or the blue inhaler isn't helping at all — call 999 immediately. Here is your written asthma plan. Follow-up in 4–6 weeks. Is there anything else?"
🚫 9 Danger Zones
Telling Priya to rehome the cat immediately
→ Graduated approach: allergy test + bedroom exclusion + HEPA first. Ultimatum = consultation failure.
Not addressing "inhalers make lungs lazy" before prescribing ICS
→ The myth must be explicitly named and debunked. Prescribing without doing this = ICS never used.
Diagnosing asthma without objective evidence
→ Spirometry + reversibility + PEFR diary required. Symptoms alone are insufficient — NICE NG245.
Not measuring PEFR or SpO2 in any acute or subacute presentation
→ PEFR is mandatory. Not measuring = missing severity + missing diagnosis confirmation.
Missing silent chest as near-fatal warning sign
→ No wheeze = no air movement = near-fatal. Absence of wheeze is worse, not better.
Prescribing LABA without ICS
→ LABA monotherapy is independently associated with asthma death. Never alone.
Not providing a written asthma action plan
→ BTS/SIGN mandatory. Most evidence-based safety tool. Omission = Tasks deduction.
SABA >1 canister/month not identified as mortality risk marker
→ This is a BTS/SIGN red flag requiring urgent review and step up. Must be recognised.
Montelukast neuropsychiatric warning not counselled
→ MHRA 2020 black box warning. Must counsel depression/suicidal ideation risk at every prescription.
💊 Drug Quick-Pick
Step 1 — mild intermittent
SABA PRN
Salbutamol. Technique check.
SABA >3×/week or nocturnal
Add ICS
BDP/budesonide. Rinse mouth.
Uncontrolled on ICS alone
ICS/LABA (MART)
Symbicort. Max 8 extra puffs/day.
Allergic rhinitis / AERD / EIB
+ Montelukast
Counsel neuropsychiatric risk.
Step 4 / add-on therapy
High-dose ICS/LABA + LAMA
Specialist input at Step 4+.
≥4 exacerbations/year + eos ≥0.3
Biologic — refer
Specialist-initiated only.
⛔ LABA alone = death risk · ⛔ SABA >1 canister/month = mortality marker · ⛔ Montelukast = counsel neuropsychiatric risk · ⛔ Biologic = specialist only
Reviewed: July 2026 · citations verified against current NICE / UK guidance